BAX is required for neuronal death after trophic factor deprivation and during development
about
Mutually exclusive subsets of BH3-only proteins are activated by the p53 and c-Jun N-terminal kinase/c-Jun signaling pathways during cortical neuron apoptosis induced by arseniteNeurotrophins: roles in neuronal development and functionMovement of Bax from the cytosol to mitochondria during apoptosisImmortalization of mouse myogenic cells can occur without loss of p16INK4a, p19ARF, or p53 and is accelerated by inactivation of BaxSusceptibility to neurodegeneration in a glaucoma is modified by Bax gene dosageMature neurons: equipped for survivalAnti-apoptotic BCL-2 family proteins in acute neural injuryBAX unleashed: the biochemical transformation of an inactive cytosolic monomer into a toxic mitochondrial poreMicroarray Analysis Reveals Increased Transcriptional Repression and Reduced Metabolic Activity but Not Major Changes in the Core Apoptotic Machinery during Maturation of Sympathetic Neurons.Mesencephalic Astrocyte-derived Neurotrophic Factor (MANF) Has a Unique Mechanism to Rescue Apoptotic NeuronsIntrinsic Axonal Growth and the Drive for RegenerationMolecular control of the neural crest and peripheral nervous system developmentSynergistic anti-apoptotic activity between Bcl-2 and SMN implicated in spinal muscular atrophyIntrinsic and extrinsic pathway signaling during neuronal apoptosis: lessons from the analysis of mutant miceGustatory innervation and bax-dependent caspase-2: participants in the life and death pathways of mouse taste receptor cellsAtm and Bax cooperate in ionizing radiation-induced apoptosis in the central nervous systemInteraction of Brn3a and HIPK2 mediates transcriptional repression of sensory neuron survivalCritical function of endogenous XIAP in regulating caspase activation during sympathetic neuronal apoptosisEthanol-induced neuronal apoptosis in vivo requires BAX in the developing mouse brainConcomitant loss of proapoptotic BH3-only Bcl-2 antagonists Bik and Bim arrests spermatogenesisp53 and Bax: putative death factors in taste cell turnoverGamma protocadherins are required for synaptic development in the spinal cordThe combined functions of proapoptotic Bcl-2 family members bak and bax are essential for normal development of multiple tissuesMsx2 and p21(CIP1/WAF1) mediate the proapoptotic effects of bone morphogenetic protein-4 on ventricular zone progenitor cellsTumor-suppressor p53 is expressed in proliferating and newly formed neurons of the embryonic and postnatal rat brain: comparison with expression of the cell cycle regulators p21Waf1/Cip1, p27Kip1, p57Kip2, p16Ink4a, cyclin G1, and the proto-oncogeneBax and Bcl-2 interaction in a transgenic mouse model of familial amyotrophic lateral sclerosisDifferences in bcl-2- and bax-independent function in regulating apoptosis in sensory neuron populationsFrizzled3 is required for neurogenesis and target innervation during sympathetic nervous system developmentCoronin-1 is a neurotrophin endosomal effector that is required for developmental competition for survival.Deletion of Bax eliminates sex differences in the mouse forebrainThe role of Bax in glutamate-induced nerve cell deathBcl-2 and Bax function independently to regulate cell deathA differential developmental pattern of spinal interneuron apoptosis during synaptogenesis: insights from genetic analyses of the protocadherin-gamma gene clusterBax-dependent spermatogonia apoptosis is required for testicular development and spermatogenesisBax ablation prevents dopaminergic neurodegeneration in the 1-methyl- 4-phenyl-1,2,3,6-tetrahydropyridine mouse model of Parkinson's diseaseBid induces the oligomerization and insertion of Bax into the outer mitochondrial membraneSexual Differences in Cell Loss during the Post-Hatch Development of Song Control Nuclei in the Bengalese FinchBrn-3b enhances the pro-apoptotic effects of p53 but not its induction of cell cycle arrest by cooperating in trans-activation of bax expressionDeath Receptor 6 Promotes Wallerian Degeneration in Peripheral Axons.NeuroD regulates neuronal migration.
P2860
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P2860
BAX is required for neuronal death after trophic factor deprivation and during development
description
1996 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1996 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 1996
@ast
im September 1996 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 1996/09/01)
@sk
vědecký článek publikovaný v roce 1996
@cs
wetenschappelijk artikel (gepubliceerd op 1996/09/01)
@nl
наукова стаття, опублікована у вересні 1996
@uk
مقالة علمية (نشرت في سبتمبر 1996)
@ar
name
BAX is required for neuronal d ...... ivation and during development
@ast
BAX is required for neuronal d ...... ivation and during development
@en
BAX is required for neuronal d ...... ivation and during development
@nl
type
label
BAX is required for neuronal d ...... ivation and during development
@ast
BAX is required for neuronal d ...... ivation and during development
@en
BAX is required for neuronal d ...... ivation and during development
@nl
prefLabel
BAX is required for neuronal d ...... ivation and during development
@ast
BAX is required for neuronal d ...... ivation and during development
@en
BAX is required for neuronal d ...... ivation and during development
@nl
P2093
P3181
P1433
P1476
BAX is required for neuronal d ...... ivation and during development
@en
P2093
C. M. Knudson
E. M. Johnson
J. L. Elliott
S. J. Korsmeyer
T. L. Deckwerth
W. D. Snider
P304
P3181
P356
10.1016/S0896-6273(00)80173-7
P407
P577
1996-09-01T00:00:00Z