Characterization of junD: a new member of the jun proto-oncogene family
about
Selective interaction of JNK protein kinase isoforms with transcription factorsCloning of Nrf1, an NF-E2-related transcription factor, by genetic selection in yeastExpression of c-jun, jun B and jun D proto-oncogenes in human peripheral-blood granulocytesRedox activation of Fos-Jun DNA binding activity is mediated by a DNA repair enzymeInteractions among LRF-1, JunB, c-Jun, and c-Fos define a regulatory program in the G1 phase of liver regenerationIsolation of cDNAs for DNA-binding proteins which specifically bind to a tax-responsive enhancer element in the long terminal repeat of human T-cell leukemia virus type ISmall Maf proteins heterodimerize with Fos and may act as competitive repressors of the NF-E2 transcription factorNeoplastic transformation of rat thyroid cells requires the junB and fra-1 gene induction which is dependent on the HMGI-C gene productInduced ATF-2 represses CDK4 transcription through dimerization with JunD inhibiting intestinal epithelial cell growth after polyamine depletionAdapted Boolean network models for extracellular matrix formation.Ligand-specific sequential regulation of transcription factors for differentiation of MCF-7 cellsTissue restricted expression and chromosomal localization of the YB-1 gene encoding a 42 kD nuclear CCAAT binding protein.The human papillomavirus type 16 E7 gene product interacts with and trans-activates the AP1 family of transcription factorsThe immediate-early growth response in regenerating liver and insulin-stimulated H-35 cells: comparison with serum-stimulated 3T3 cells and identification of 41 novel immediate-early genes.The basic region of Fos mediates specific DNA binding.A network of mitogen-activated protein kinases links G protein-coupled receptors to the c-jun promoter: a role for c-Jun NH2-terminal kinase, p38s, and extracellular signal-regulated kinase 5.AP1 regulation of proliferation and initiation of apoptosis in erythropoietin-dependent erythroid cellsFra-1 induces morphological transformation and increases in vitro invasiveness and motility of epithelioid adenocarcinoma cells.Molecular determinants of NOTCH4 transcription in vascular endothelium.mXBP/CRE-BP2 and c-Jun form a complex which binds to the cyclic AMP, but not to the 12-O-tetradecanoylphorbol-13-acetate, response elementNegative transcriptional regulation of human interleukin 2 (IL-2) gene by glucocorticoids through interference with nuclear transcription factors AP-1 and NF-AT.An AP-1 binding site is the predominant cis-acting regulatory element in the 1.2-kilobase early RNA promoter of human cytomegalovirus.Bcl-2 and Bcl-X(L) block thapsigargin-induced nitric oxide generation, c-Jun NH(2)-terminal kinase activity, and apoptosisAdenovirus E1A negatively and positively modulates transcription of AP-1 dependent genes by dimer-specific regulation of the DNA binding and transactivation activities of Jun.Mouse models of endocrine tumours.The vascular smooth muscle alpha-actin gene is reactivated during cardiac hypertrophy provoked by load.Interleukin-12: an update on its immunological activities, signaling and regulation of gene expressionAntitumor promotion by phenolic antioxidants: inhibition of AP-1 activity through induction of Fra expression.Transcriptional and epigenetic substrates of methamphetamine addiction and withdrawal: evidence from a long-access self-administration model in the rat.Transcriptional activation of the human rod cGMP-phosphodiesterase beta-subunit gene is mediated by an upstream AP-1 element.Determinants of half-site spacing preferences that distinguish AP-1 and ATF/CREB bZIP domainsStructure and mapping of the fosB gene. FosB downregulates the activity of the fosB promoterThe functional consequences of variation in transcription factor binding.Induction of junB expression, but not c-jun, by granulocyte colony-stimulating factor or macrophage colony-stimulating factor in the proliferative response of human myeloid leukemia cells.Global expression analysis identified a preferentially nerve growth factor-induced transcriptional program regulated by sustained mitogen-activated protein kinase/extracellular signal-regulated kinase (ERK) and AP-1 protein activation during PC12 ceCoupled and uncoupled induction of fos and jun transcription by different second messengers in cells of hematopoietic origin.A relational database of transcription factors.Promoter-level expression clustering identifies time development of transcriptional regulatory cascades initiated by ErbB receptors in breast cancer cells.JunD protects against chronic kidney disease by regulating paracrine mitogens.Unregulated expression of c-Jun or c-Fos proteins but not Jun D inhibits oestrogen receptor activity in human breast cancer derived cells
P2860
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P2860
Characterization of junD: a new member of the jun proto-oncogene family
description
1989 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի մայիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 1989
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scientific journal article
@en
wetenschappelijk artikel (gepubliceerd op 1989/05/01)
@nl
wissenschaftlicher Artikel
@de
наукова стаття, опублікована в травні 1989
@uk
مقالة علمية (نشرت في مايو 1989)
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name
Characterization of junD: a new member of the jun proto-oncogene family
@ast
Characterization of junD: a new member of the jun proto-oncogene family
@en
type
label
Characterization of junD: a new member of the jun proto-oncogene family
@ast
Characterization of junD: a new member of the jun proto-oncogene family
@en
prefLabel
Characterization of junD: a new member of the jun proto-oncogene family
@ast
Characterization of junD: a new member of the jun proto-oncogene family
@en
P2093
P2860
P1433
P1476
Characterization of junD: a new member of the jun proto-oncogene family
@en
P2093
P2860
P304
P407
P577
1989-05-01T00:00:00Z