JunD protects against chronic kidney disease by regulating paracrine mitogens.
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Menin suppresses osteoblast differentiation by antagonizing the AP-1 factor, JunDStimulating healthy tissue regeneration by targeting the 5-HT₂B receptor in chronic liver disease.Polyamines regulate the stability of JunD mRNA by modulating the competitive binding of its 3' untranslated region to HuR and AUF1Spindle pole cohesion requires glycosylation-mediated localization of NuMA.Lipocalin 2 is essential for chronic kidney disease progression in mice and humans.TGF-alpha mediates genetic susceptibility to chronic kidney disease.Increased renal iron accumulation in hypertensive nephropathy of salt-loaded hypertensive ratsJunD enhances miR-29b levels transcriptionally and posttranscriptionally to inhibit proliferation of intestinal epithelial cells.A transcriptional network underlies susceptibility to kidney disease progression.JunD represses transcription and translation of the tight junction protein zona occludens-1 modulating intestinal epithelial barrier function.Transplant-induced reactivation of murine cytomegalovirus immediate early gene expression is associated with recruitment of NF-κB and AP-1 to the major immediate early promoter.The role of the EGF family of ligands and receptors in renal development, physiology and pathophysiologySignal transduction in podocytes--spotlight on receptor tyrosine kinases.Role of kidney biomarkers of chronic kidney disease: An update.Induced JunD in intestinal epithelial cells represses CDK4 transcription through its proximal promoter region following polyamine depletion.Novel Biomarkers in the Diagnosis of Chronic Kidney Disease and the Prediction of Its Outcome.Transcriptional regulation of importin-α1 by JunD modulates subcellular localization of RNA-binding protein HuR in intestinal epithelial cells.A susceptibility gene for kidney disease in an obese mouse model of type II diabetes maps to chromosome 8.Role of TGF-alpha in the progression of diabetic kidney disease.JunD regulates lymphocyte proliferation and T helper cell cytokine expressionA look at transactivation of the EGF receptor by angiotensin II.Hypertensive renal injury is associated with gene variation affecting immune signaling.Jun and JunD-dependent functions in cell proliferation and stress response.Loss of ACE2 accelerates time-dependent glomerular and tubulointerstitial damage in streptozotocin-induced diabetic mice.EGFR signaling in podocytes at the root of glomerular disease.JunD is a profibrogenic transcription factor regulated by Jun N-terminal kinase-independent phosphorylation.The JNK Signaling Pathway in Renal Fibrosis.Stat3 Controls Tubulointerstitial Communication during CKD.β-Arrestin scaffolds and signaling elements essential for the obestatin/GPR39 system that determine the myogenic program in human myoblast cells.Association between renal iron accumulation and renal interstitial fibrosis in a rat model of chronic kidney disease.
P2860
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P2860
JunD protects against chronic kidney disease by regulating paracrine mitogens.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
2003年论文
@zh
2003年论文
@zh-cn
name
JunD protects against chronic kidney disease by regulating paracrine mitogens.
@ast
JunD protects against chronic kidney disease by regulating paracrine mitogens.
@en
type
label
JunD protects against chronic kidney disease by regulating paracrine mitogens.
@ast
JunD protects against chronic kidney disease by regulating paracrine mitogens.
@en
prefLabel
JunD protects against chronic kidney disease by regulating paracrine mitogens.
@ast
JunD protects against chronic kidney disease by regulating paracrine mitogens.
@en
P2093
P2860
P356
P1476
JunD protects against chronic kidney disease by regulating paracrine mitogens
@en
P2093
Carla Martino
Evangéline Pillebout
Fabiola Terzi
Martine Burtin
Moshe Yaniv
Pierre Federici
P2860
P304
P356
10.1172/JCI200317647
P407
P577
2003-09-01T00:00:00Z