Toward pluripotency by reprogramming: mechanisms and application - Test2 - elhamkhani - TriplyDB

Toward pluripotency by reprogramming: mechanisms and application

Toward pluripotency by reprogramming: mechanisms and application

http://www.wikidata.org/entity/Q28833688

about

iPSC-derived forebrain neurons from FXS individuals show defects in initial neurite outgrowth iPSCs as a major opportunity to understand and cure age-related diseases.A Comparative View on Easy to Deploy non-Integrating Methods for Patient-Specific iPSC Production Protein kinases and associated pathways in pluripotent state and lineage differentiation.Induced Pluripotent Stem Cells for Disease Modeling and Drug Discovery in Neurodegenerative Diseases.MicroRNA-Mediated Reprogramming of Somatic Cells into Neural Stem Cells or Neurons.Functional AdoMet Isosteres Resistant to Classical AdoMet Degradation Pathways.Do circulating cells transdifferentiate and replenish stem cell pools in the brain and periphery?

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P2860

Toward pluripotency by reprogramming: mechanisms and application

http://www.wikidata.org/entity/Q28833688

description

2013 nî lūn-bûn

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2013 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

Toward pluripotency by reprogramming: mechanisms and application

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Toward pluripotency by reprogramming: mechanisms and application

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Toward pluripotency by reprogramming: mechanisms and application

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Toward pluripotency by reprogramming: mechanisms and application

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Toward pluripotency by reprogramming: mechanisms and application

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Peng Jin

http://www.w3.org/2001/XMLSchema#string

Stephen T Warren

http://www.w3.org/2001/XMLSchema#string

Tao Wang

http://www.w3.org/2001/XMLSchema#string

P2860

DNA methyltransferases Dnmt3a and Dnmt3b are essential for de novo methylation and mammalian development

Prediction of novel families of enzymes involved in oxidative and other complex modifications of bases in nucleic acids

Progressive degeneration of human neural stem cells caused by pathogenic LRRK2

Conversion of 5-methylcytosine to 5-hydroxymethylcytosine in mammalian DNA by MLL partner TET1

Thymine DNA glycosylase can rapidly excise 5-formylcytosine and 5-carboxylcytosine: potential implications for active demethylation of CpG sites

UHRF1 plays a role in maintaining DNA methylation in mammalian cells

RNA-guided human genome engineering via Cas9

Reprogramming towards pluripotency requires AID-dependent DNA demethylation

Multiplex genome engineering using CRISPR/Cas systems

MicroRNAs: target recognition and regulatory functions

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