Point mutations in the tumor suppressor Smad4/DPC4 enhance its phosphorylation by GSK3 and reversibly inactivate TGF-β signaling
about
Antitumor effects of recombinant human adenovirus-p53 against human cutaneous squamous cell carcinoma in mice.Genetic variants in the TGFβ-signaling pathway influence expression of miRNAs in colon and rectal normal mucosa and tumor tissue.Combination immunohistochemistry for SMAD4 and Runt-related transcription factor 3 may identify a favorable prognostic subgroup of pancreatic ductal adenocarcinomas.Multispectral imaging reveals hyper active TGF-β signaling in colorectal cancer.A partnership with the proteasome; the destructive nature of GSK3.
P2860
Point mutations in the tumor suppressor Smad4/DPC4 enhance its phosphorylation by GSK3 and reversibly inactivate TGF-β signaling
description
2016 nî lūn-bûn
@nan
2016 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2016 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
name
Point mutations in the tumor s ...... bly inactivate TGF-β signaling
@ast
Point mutations in the tumor s ...... bly inactivate TGF-β signaling
@en
type
label
Point mutations in the tumor s ...... bly inactivate TGF-β signaling
@ast
Point mutations in the tumor s ...... bly inactivate TGF-β signaling
@en
prefLabel
Point mutations in the tumor s ...... bly inactivate TGF-β signaling
@ast
Point mutations in the tumor s ...... bly inactivate TGF-β signaling
@en
P2860
P1476
Point mutations in the tumor s ...... bly inactivate TGF-β signaling
@en
P2093
Hadrien Demagny
P2860
P304
P356
10.1080/23723556.2015.1025181
P407
P577
2016-01-01T00:00:00Z