sameAs
Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infectionVirus-specific CD8+ cytotoxic T-lymphocyte activity associated with control of viremia in primary human immunodeficiency virus type 1 infectionHIV-Host Interactions: Implications for Vaccine DesignViral Escape from Neutralizing Antibodies in Early Subtype A HIV-1 Infection Drives an Increase in Autologous Neutralization BreadthAntiviral pressure exerted by HIV-1-specific cytotoxic T lymphocytes (CTLs) during primary infection demonstrated by rapid selection of CTL escape virusCross-sectional detection of acute HIV infection: timing of transmission, inflammation and antiretroviral therapyObituary: Kuan-Teh Jeang.Loss of DNAM-1 contributes to CD8+ T-cell exhaustion in chronic HIV-1 infectionIntegrating in silico and in vitro analysis of peptide binding affinity to HLA-Cw*0102: a bioinformatic approach to the prediction of new epitopesElevation of intact and proteolytic fragments of acute phase proteins constitutes the earliest systemic antiviral response in HIV-1 infectionTransmission of single HIV-1 genomes and dynamics of early immune escape revealed by ultra-deep sequencing.Relationship between functional profile of HIV-1 specific CD8 T cells and epitope variability with the selection of escape mutants in acute HIV-1 infectionDefective B cell responses in the absence of SH2D1AMajor expansion of CD8+ T cells with a predominant V beta usage during the primary immune response to HIV.Distinct patterns of hepcidin and iron regulation during HIV-1, HBV, and HCV infections.Copy number variation of KIR genes influences HIV-1 control.Innate immunity against HIV: a priority target for HIV prevention researchEvidence of dysregulation of dendritic cells in primary HIV infection.Phenotypic properties of transmitted founder HIV-1Origin and evolution of HIV-1 in breast milk determined by single-genome amplification and sequencing.Fitness costs and diversity of the cytotoxic T lymphocyte (CTL) response determine the rate of CTL escape during acute and chronic phases of HIV infectionHeterosexual Transmission of Subtype C HIV-1 Selects Consensus-Like Variants without Increased Replicative Capacity or Interferon-α ResistanceDistinct kinetics of Gag-specific CD4+ and CD8+ T cell responses during acute HIV-1 infectionVirus-induced immunosuppression: immune system-mediated destruction of virus-infected dendritic cells results in generalized immune suppression.Discriminated selection among viral peptides with the appropriate anchor residues: implications for the size of the cytotoxic T-lymphocyte repertoire and control of viral infection.A single amino acid change in the glycoprotein of lymphocytic choriomeningitis virus is associated with the ability to cause growth hormone deficiency syndromeBlocking of feline immunodeficiency virus infection by a monoclonal antibody to CD9 is via inhibition of virus release rather than interference with receptor bindingCytokines induced during chronic hepatitis B virus infection promote a pathway for NK cell-mediated liver damage.Hepatitis C virus envelope glycoprotein fitness defines virus population composition following transmission to a new hostNoncytopathic clearance of lymphocytic choriomeningitis virus from the hepatocyte.Determinants of human immunodeficiency virus type 1 escape from the primary CD8+ cytotoxic T lymphocyte response.Vertical T cell immunodominance and epitope entropy determine HIV-1 escape.HIV-1 infection-induced apoptotic microparticles inhibit human DCs via CD44Defining the HLA class I-associated viral antigen repertoire from HIV-1-infected human cells.Virus-induced immunosuppression: kinetic analysis of the selection of a mutation associated with viral persistenceMolecular basis of viral persistence: a single amino acid change in the glycoprotein of lymphocytic choriomeningitis virus is associated with suppression of the antiviral cytotoxic T-lymphocyte response and establishment of persistence.HIV-1 gp41 envelope IgA is frequently elicited after transmission but has an initial short response half-life.Germinal Center B Cell and T Follicular Helper Cell Responses to Viral Vector and Protein-in-Adjuvant Vaccines.Innate immune responses in primary HIV-1 infection.The first T cell response to transmitted/founder virus contributes to the control of acute viremia in HIV-1 infection.
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