A key role for autophagy and the autophagy gene Atg16l1 in mouse and human intestinal Paneth cells - Test2 - elhamkhani - TriplyDB

A key role for autophagy and the autophagy gene Atg16l1 in mouse and human intestinal Paneth cells

A key role for autophagy and the autophagy gene Atg16l1 in mouse and human intestinal Paneth cells

http://www.wikidata.org/entity/Q29614565

about

Central role of the gut epithelial barrier in the pathogenesis of chronic intestinal inflammation: lessons learned from animal models and human genetics Recent Insights Into the Genetics of Inflammatory Bowel Disease Yip1 domain family, member 6 (Yipf6) mutation induces spontaneous intestinal inflammation in mice OATL1, a novel autophagosome-resident Rab33B-GAP, regulates autophagosomal maturation A mammalian autophagosome maturation mechanism mediated by TECPR1 and the Atg12-Atg5 conjugate The LRR and RING domain protein LRSAM1 is an E3 ligase crucial for ubiquitin-dependent autophagy of intracellular Salmonella Typhimurium Autophagy in immunity and inflammation Exercise-induced BCL2-regulated autophagy is required for muscle glucose homeostasis Virus-plus-susceptibility gene interaction determines Crohn's disease gene Atg16L1 phenotypes in intestine Autophagy protects against active tuberculosis by suppressing bacterial burden and inflammation Homeostasis and inflammation in the intestine Human IRGM regulates autophagy and cell-autonomous immunity functions through mitochondria Horror autoinflammaticus: the molecular pathophysiology of autoinflammatory disease (*)Atg9a controls dsDNA-driven dynamic translocation of STING and the innate immune response The Mucosal Immune System and Its Regulation by Autophagy Paneth cell α-defensins and enteric microbiota in health and disease The clearance of dying cells: table for two Mechanisms of Microbe-Host Interaction in Crohn's Disease: Dysbiosis vs. Pathobiont Selection Looking for Pyroptosis-Modulating miRNAs as a Therapeutic Target for Improving Myocardium Survival Intestinal Microbiota and the Innate Immune System - A Crosstalk in Crohn's Disease Pathogenesis Mitochondrial dysfunction in inflammatory bowel disease Apoptosis, Necrosis, and Necroptosis in the Gut and Intestinal Homeostasis The Intestinal Microbiota in Inflammatory Bowel Disease Gut inflammation and immunity: what is the role of the human gut virome?Autophagy and the regulation of the immune response Development of autophagy inducers in clinical medicine FADD: an endogenous inhibitor of RIP3-driven regulated necrosis New insights into the dichotomous role of innate cytokines in gut homeostasis and inflammation Dual roles for autophagy: degradation and secretion of Alzheimer's disease Aβ peptide The virome in host health and disease Endoplasmic reticulum stress and inflammation Role of endoplasmic reticulum stress and autophagy as interlinking pathways in the pathogenesis of inflammatory bowel disease Autophagy, nutrition and immunology Autophagy and aging Mitochondria and the autophagy-inflammation-cell death axis in organismal aging Autophagy and disease: always two sides to a problem Regulation of osteoclasts by membrane-derived lipid mediators PAMPs and DAMPs: signal 0s that spur autophagy and immunity Autophagy modulation as a potential therapeutic target for diverse diseases Mechanisms of mitochondria and autophagy crosstalk

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A key role for autophagy and the autophagy gene Atg16l1 in mouse and human intestinal Paneth cells

http://www.wikidata.org/entity/Q29614565

description

2008 nî lūn-bûn

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2008 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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name

A key role for autophagy and t ...... human intestinal Paneth cells

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A key role for autophagy and t ...... human intestinal Paneth cells

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A key role for autophagy and t ...... human intestinal Paneth cells

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A key role for autophagy and t ...... human intestinal Paneth cells

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Chieko Kishi

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Christian D Stone

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Elizabeth M Brunt

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Ellen Li

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Herbert W Virgin

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Javier A Carrero

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Jochen K Lennerz

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John Y Liu

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Joy Loh

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P2860

Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease

Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice

Gene set enrichment analysis: a knowledge-based approach for interpreting genome-wide expression profiles.

Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls

The Atg16L complex specifies the site of LC3 lipidation for membrane biogenesis in autophagy

Genome-wide association study identifies new susceptibility loci for Crohn disease and implicates autophagy in disease pathogenesis

Autophagy in the Pathogenesis of Disease

Formation of the approximately 350-kDa Apg12-Apg5.Apg16 multimeric complex, mediated by Apg16 oligomerization, is essential for autophagy in yeast.

Suppression of basal autophagy in neural cells causes neurodegenerative disease in mice

Mouse Apg16L, a novel WD-repeat protein, targets to the autophagic isolation membrane with the Apg12-Apg5 conjugate

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259-263

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10.1038/NATURE07416

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7219

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Mizushima Noboru

Hiroyuki Miyoshi

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2008-10-05T00:00:00Z

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