HIF-1alpha is essential for myeloid cell-mediated inflammation - Test2 - elhamkhani - TriplyDB

HIF-1alpha is essential for myeloid cell-mediated inflammation

HIF-1alpha is essential for myeloid cell-mediated inflammation

http://www.wikidata.org/entity/Q29617581

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Oxidation in rheumatoid arthritis.miR-17-92 expression in differentiated T cells - implications for cancer immunotherapy Hyperbaric treatment for children with autism: a multicenter, randomized, double-blind, controlled trial The streptococcal collagen-like protein-1 (Scl1) is a significant determinant for biofilm formation by group a Streptococcus The group A streptococcal collagen-like protein-1, Scl1, mediates biofilm formation by targeting the extra domain A-containing variant of cellular fibronectin expressed in wounded tissue Ubiquitin-specific protease 19 (USP19) regulates hypoxia-inducible factor 1α (HIF-1α) during hypoxia Mint3 enhances the activity of hypoxia-inducible factor-1 (HIF-1) in macrophages by suppressing the activity of factor inhibiting HIF-1 Endothelial catabolism of extracellular adenosine during hypoxia: the role of surface adenosine deaminase and CD26 SENP3 is responsible for HIF-1 transactivation under mild oxidative stress via p300 de-SUMOylation Systemic VHL gene functions and the VHL disease Hypoxia-inducible factors and the response to hypoxic stress Deletion of vascular endothelial growth factor in myeloid cells accelerates tumorigenesis MicroRNAs in immune regulation--opportunities for cancer immunotherapy Treating lung inflammation with agonists of the adenosine A2A receptor: promises, problems and potential solutions Regulation of type II collagen synthesis during osteoarthritis by prolyl-4-hydroxylases: possible influence of low oxygen levels Myeloid progenitors differentiate into microglia and promote vascular repair in a model of ischemic retinopathy Coordinated adenine nucleotide phosphohydrolysis and nucleoside signaling in posthypoxic endothelium: role of ectonucleotidases and adenosine A2B receptors Oxidative metabolism and PGC-1beta attenuate macrophage-mediated inflammation Hypoxia: not merely a regulator of angiogenesis?Loss of skeletal muscle HIF-1alpha results in altered exercise endurance Immunobiology of hepatocarcinogenesis: Ways to go or almost there?Hypoxia-Inducible Factor (HIF) as a Target for Novel Therapies in Rheumatoid Arthritis Sickness-Associated Anorexia: Mother Nature's Idea of Immunonutrition?Hypoxia and its implications in rheumatoid arthritis Hallmarks of cancer and AU-rich elements Glucose Metabolism in T Cells and Monocytes: New Perspectives in HIV Pathogenesis Modulation of Host Angiogenesis as a Microbial Survival Strategy and Therapeutic Target The Response of Macrophages and Neutrophils to Hypoxia in the Context of Cancer and Other Inflammatory Diseases A Metabolic Immune Checkpoint: Adenosine in Tumor Microenvironment Hypoxia-inducible factors as molecular targets for liver diseases NF-κB and HIF crosstalk in immune responses Exploring the HIFs, buts and maybes of hypoxia signalling in disease: lessons from zebrafish models Pathogenesis of Myeloproliferative Neoplasms: Role and Mechanisms of Chronic Inflammation Control of macrophage metabolism and activation by mTOR and Akt signaling Bench-to-bedside review: the effects of hyperoxia during critical illness Regulating the Intersection of Metabolism and Pathogenesis in Gram-positive Bacteria Complement and macrophage crosstalk during process of angiogenesis in tumor progression Extrinsic and intrinsic control of macrophage inflammatory responses Drosophila blood as a model system for stress sensing mechanisms Redox regulation of stem/progenitor cells and bone marrow niche

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P2860

HIF-1alpha is essential for myeloid cell-mediated inflammation

http://www.wikidata.org/entity/Q29617581

description

2003 nî lūn-bûn

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2003 թուականի Մարտին հրատարակուած գիտական յօդուած

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2003 թվականի մարտին հրատարակված գիտական հոդված

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2003年の論文

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2003年論文

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2003年論文

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2003年論文

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2003年論文

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name

HIF-1alpha is essential for myeloid cell-mediated inflammation

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HIF-1alpha is essential for myeloid cell-mediated inflammation

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HIF-1alpha is essential for myeloid cell-mediated inflammation

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HIF-1alpha is essential for myeloid cell-mediated inflammation

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prefLabel

HIF-1alpha is essential for myeloid cell-mediated inflammation

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HIF-1alpha is essential for myeloid cell-mediated inflammation

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Clausen BE

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Cramer T

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The tumour suppressor protein VHL targets hypoxia-inducible factors for oxygen-dependent proteolysis

Invasion of brain microvascular endothelial cells by group B streptococci

The Expression and Distribution of the Hypoxia-Inducible Factors HIF-1α and HIF-2α in Normal Human Tissues, Cancers, and Tumor-Associated Macrophages

The biochemical basis of phagocytosis. I. Metabolic changes during the ingestion of particles by polymorphonuclear leukocytes

The von Hippel-Lindau tumor suppressor gene

HIF-1 alpha is required for solid tumor formation and embryonic vascularization.

Innate immunity

The peritoneal environment during infection. The effect of monomicrobial and polymicrobial bacteria on pO2 and pH.

Transcription factor HIF-1 is a necessary mediator of the pasteur effect in mammalian cells.

Macrophage responses to hypoxia: relevance to disease mechanisms.

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