Expression of SHANK3 in the Temporal Neocortex of Patients with Intractable Temporal Epilepsy and Epilepsy Rat Models. - Test2 - elhamkhani - TriplyDB

Expression of SHANK3 in the Temporal Neocortex of Patients with Intractable Temporal Epilepsy and Epilepsy Rat Models.

Expression of SHANK3 in the Temporal Neocortex of Patients with Intractable Temporal Epilepsy and Epilepsy Rat Models.

http://www.wikidata.org/entity/Q30008868

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Interactions between GHRH and GABAARs in the brains of patients with epilepsy and in animal models of epilepsy.nArgBP2-SAPAP-SHANK, the core postsynaptic triad associated with psychiatric disorders.

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Expression of SHANK3 in the Temporal Neocortex of Patients with Intractable Temporal Epilepsy and Epilepsy Rat Models.

http://www.wikidata.org/entity/Q30008868

description

2016 nî lūn-bûn

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2016 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2016 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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Expression of SHANK3 in the Te ...... lepsy and Epilepsy Rat Models.

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Expression of SHANK3 in the Te ...... lepsy and Epilepsy Rat Models.

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Expression of SHANK3 in the Te ...... lepsy and Epilepsy Rat Models.

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Expression of SHANK3 in the Te ...... lepsy and Epilepsy Rat Models.

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Expression of SHANK3 in the Te ...... lepsy and Epilepsy Rat Models.

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Expression of SHANK3 in the Te ...... lepsy and Epilepsy Rat Models.

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Cellular and Molecular Neurobiology

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Expression of SHANK3 in the Te ...... lepsy and Epilepsy Rat Models.

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Baobing Gao

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Fangshuo Zheng

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Xin Xu

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Yan Xiong

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Yanke Zhang

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Yida Hu

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P2860

Haploinsufficiency of the autism-associated Shank3 gene leads to deficits in synaptic function, social interaction, and social communication

Synaptic dysfunction and abnormal behaviors in mice lacking major isoforms of Shank3

AMPA receptors as a molecular target in epilepsy therapy

Hyperactive mTOR signals in the proopiomelanocortin-expressing hippocampal neurons cause age-dependent epilepsy and premature death in mice

Association between deletion size and important phenotypes expands the genomic region of interest in Phelan-McDermid syndrome (22q13 deletion syndrome)

An architectural framework that may lie at the core of the postsynaptic density

22q13.2q13.32 genomic regions associated with severity of speech delay, developmental delay, and physical features in Phelan-McDermid syndrome

A patient with the classic features of Phelan-McDermid syndrome and a high immunoglobulin E level caused by a cryptic interstitial 0.72-Mb deletion in the 22q13.2 region

Neuroligins determine synapse maturation and function

Phelan-McDermid Syndrome and SHANK3: Implications for Treatment.

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10.1007/S10571-016-0423-7

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1019446654

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27592227

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