Huntingtin is required for normal excitatory synapse development in cortical and striatal circuits
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Quantitative Analysis of Axonal Branch Dynamics in the Developing Nervous SystemGeneration and Characterization of Knock-in Mouse Models Expressing Versions of Huntingtin with Either an N17 or a Combined PolyQ and Proline-Rich Region Deletion.Is Huntingtin Dispensable in the Adult Brain?Gestational Exposure to Air Pollution Alters Cortical Volume, Microglial Morphology, and Microglia-Neuron Interactions in a Sex-Specific Manner.Human Umbilical Tissue-Derived Cells Promote Synapse Formation and Neurite Outgrowth via Thrombospondin Family ProteinsEnhanced Store-Operated Calcium Entry Leads to Striatal Synaptic Loss in a Huntington's Disease Mouse Model.Selective expression of mutant huntingtin during development recapitulates characteristic features of Huntington's diseaseAbnormal degradation of the neuronal stress-protective transcription factor HSF1 in Huntington's disease.Huntington Disease as a Neurodevelopmental Disorder and Early Signs of the Disease in Stem Cells.Induced Pluripotent Stem Cells in Huntington's Disease Research: Progress and Opportunity.The interplay between neurons and glia in synapse development and plasticity.Huntington's Disease: Mechanisms of Pathogenesis and Therapeutic Strategies.The Generation of Mouse and Human Huntington Disease iPS Cells Suitable for In vitro Studies on Huntingtin FunctionMinor splicing snRNAs are enriched in the developing mouse CNS and are crucial for survival of differentiating retinal neurons.Synaptophysin depletion and intraneuronal Aβ in organotypic hippocampal slice cultures from huAPP transgenic mice.Allele-Specific Reduction of the Mutant Huntingtin Allele Using Transcription Activator-Like Effectors in Human Huntington's Disease Fibroblasts.Topoisomerase 1 inhibitor topotecan delays the disease progression in a mouse model of Huntington's disease.Effect of early embryonic deletion of huntingtin from pyramidal neurons on the development and long-term survival of neurons in cerebral cortex and striatum.Boysenberry ingestion supports fibrolytic macrophages with the capacity to ameliorate chronic lung remodeling.Synaptic dysfunction in neurodegenerative and neurodevelopmental diseases: an overview of induced pluripotent stem-cell-based disease models
P2860
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P2860
Huntingtin is required for normal excitatory synapse development in cortical and striatal circuits
description
2014 nî lūn-bûn
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2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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name
Huntingtin is required for nor ...... cortical and striatal circuits
@ast
Huntingtin is required for nor ...... cortical and striatal circuits
@en
type
label
Huntingtin is required for nor ...... cortical and striatal circuits
@ast
Huntingtin is required for nor ...... cortical and striatal circuits
@en
prefLabel
Huntingtin is required for nor ...... cortical and striatal circuits
@ast
Huntingtin is required for nor ...... cortical and striatal circuits
@en
P2093
P2860
P50
P1476
Huntingtin is required for nor ...... cortical and striatal circuits
@en
P2093
Henry H Yin
Ioannis Dragatsis
Karol Serafin-Molina
M Ilcim Ozlu
Volodya Y Hayrapetyan
P2860
P304
P356
10.1523/JNEUROSCI.4699-13.2014
P407
P50
P577
2014-07-01T00:00:00Z