Role of p38 and JNK mitogen-activated protein kinases in the activation of ternary complex factors
about
M-Ras/R-Ras3, a transforming ras protein regulated by Sos1, GRF1, and p120 Ras GTPase-activating protein, interacts with the putative Ras effector AF6Targeting of p38 mitogen-activated protein kinases to MEF2 transcription factorsDistinct carboxy-termini confer divergent characteristics to the mitogen-activated protein kinase p38alpha and its splice isoform Mxi2Activation of mitogen-activated protein kinase p38 and extracellular signal-regulated kinase is involved in glass fiber-induced tumor necrosis factor-alpha production in macrophagesTranscriptional regulation of the cytosolic chaperonin theta subunit gene, Cctq, by Ets domain transcription factors Elk-1, Sap-1a, and Net in the absence of serum response factorPRAK, a novel protein kinase regulated by the p38 MAP kinaseTranscriptional repressor ERF is a Ras/mitogen-activated protein kinase target that regulates cellular proliferation.CDC42 and FGD1 cause distinct signaling and transforming activities.c-Jun regulates cell cycle progression and apoptosis by distinct mechanismsMultiple mitogen-activated protein kinase signaling pathways connect the cot oncoprotein to the c-jun promoter and to cellular transformationThe net repressor is regulated by nuclear export in response to anisomycin, UV, and heat shockFusion toxin BLyS-gelonin inhibits growth of malignant human B cell lines in vitro and in vivoNemo-like kinase suppresses a wide range of transcription factors, including nuclear factor-kappaBDeficiency of the stress kinase p38alpha results in embryonic lethality: characterization of the kinase dependence of stress responses of enzyme-deficient embryonic stem cellsDecreased sensitivity of tristetraprolin-deficient cells to p38 inhibitors suggests the involvement of tristetraprolin in the p38 signaling pathwayCell fate determination factor DACH1 inhibits c-Jun-induced contact-independent growthRequirement of mitogen-activated protein kinase kinase 3 (MKK3) for tumor necrosis factor-induced cytokine expressionDefective IL-12 production in mitogen-activated protein (MAP) kinase kinase 3 (Mkk3)-deficient miceRole of the stress-activated protein kinases in endothelin-induced cardiomyocyte hypertrophyWithdrawal of survival factors results in activation of the JNK pathway in neuronal cells leading to Fas ligand induction and cell deathThe Ras/Rac1/Cdc42/SEK/JNK/c-Jun cascade is a key pathway by which agonists stimulate DNA synthesis in primary cultures of rat hepatocytesObesity is associated with impaired ventricular protein kinase C-MAP kinase signaling and altered ANP mRNA expression in the heart of adult Zucker ratsSTAT4 serine phosphorylation is critical for IL-12-induced IFN-gamma production but not for cell proliferationMAPKAP kinase 2 phosphorylates serum response factor in vitro and in vivoInvolvement of Elk-1 in L6E9 skeletal muscle differentiationSelective activation of p38 mitogen-activated protein (MAP) kinase isoforms by the MAP kinase kinases MKK3 and MKK6AMPAR removal underlies Abeta-induced synaptic depression and dendritic spine lossNovel membrane-targeted ERK1 and ERK2 chimeras which act as dominant negative, isotype-specific mitogen-activated protein kinase inhibitors of Ras-Raf-mediated transcriptional activation of c-fos in NIH 3T3 cells.Requirement for Ras/Rac1-mediated p38 and c-Jun N-terminal kinase signaling in Stat3 transcriptional activity induced by the Src oncoprotein.Selective induction of p38 mitogen-activated protein kinase activity following A6H co-stimulation in primary human CD4(+) T cells.Cross-interactions of two p38 mitogen-activated protein (MAP) kinase inhibitors and two cholecystokinin (CCK) receptor antagonists with the CCK1 receptor and p38 MAP kinase.The thrombin receptor, PAR-1, causes transformation by activation of Rho-mediated signaling pathways.Activation of transcription of the human cytomegalovirus early UL4 promoter by the Ets transcription factor binding element.The potential of p38 MAPK inhibitors to modulate periodontal infections.Inhibition of anchorage-independent growth of transformed NIH3T3 cells by epithelial protein lost in neoplasm (EPLIN) requires localization of EPLIN to actin cytoskeleton.The Phytochemical Bergenin Enhances T Helper 1 Responses and Anti-Mycobacterial Immunity by Activating the MAP Kinase Pathway in MacrophagesSelective unresponsiveness to the inhibition of p38 MAPK activation by cAMP helps L929 fibroblastoma cells escape TNF-alpha-induced cell death.A network of mitogen-activated protein kinases links G protein-coupled receptors to the c-jun promoter: a role for c-Jun NH2-terminal kinase, p38s, and extracellular signal-regulated kinase 5.Tumor suppression by p53: making cells senescent.Role of MLK3 in the regulation of mitogen-activated protein kinase signaling cascades.
P2860
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P2860
Role of p38 and JNK mitogen-activated protein kinases in the activation of ternary complex factors
description
1997 nî lūn-bûn
@nan
1997 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
1997 թվականի մայիսին հրատարակված գիտական հոդված
@hy
1997年の論文
@ja
1997年論文
@yue
1997年論文
@zh-hant
1997年論文
@zh-hk
1997年論文
@zh-mo
1997年論文
@zh-tw
1997年论文
@wuu
name
Role of p38 and JNK mitogen-ac ...... ion of ternary complex factors
@ast
Role of p38 and JNK mitogen-ac ...... ion of ternary complex factors
@en
type
label
Role of p38 and JNK mitogen-ac ...... ion of ternary complex factors
@ast
Role of p38 and JNK mitogen-ac ...... ion of ternary complex factors
@en
prefLabel
Role of p38 and JNK mitogen-ac ...... ion of ternary complex factors
@ast
Role of p38 and JNK mitogen-ac ...... ion of ternary complex factors
@en
P2093
P2860
P356
P1476
Role of p38 and JNK mitogen-ac ...... ion of ternary complex factors
@en
P2093
P2860
P304
P356
10.1128/MCB.17.5.2360
P407
P577
1997-05-01T00:00:00Z