JAM-C regulates unidirectional monocyte transendothelial migration in inflammation. - Test2 - elhamkhani - TriplyDB

JAM-C regulates unidirectional monocyte transendothelial migration in inflammation.

JAM-C regulates unidirectional monocyte transendothelial migration in inflammation.

http://www.wikidata.org/entity/Q30480098

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Acquired immunodeficiency syndrome and the blood-brain barrier Crossing the Vascular Wall: Common and Unique Mechanisms Exploited by Different Leukocyte Subsets during Extravasation Divergent JAM-C Expression Accelerates Monocyte-Derived Cell Exit from Atherosclerotic Plaques How leukocytes trigger opening and sealing of gaps in the endothelial barrier The regulation of transendothelial migration: new knowledge and new questions Endothelial cells use dynamic actin to facilitate lymphocyte transendothelial migration and maintain the monolayer barrier.LFA-1 and Mac-1 define characteristically different intralumenal crawling and emigration patterns for monocytes and neutrophils in situ.The junctional adhesion molecule JAM-C regulates polarized transendothelial migration of neutrophils in vivo.Activated tumor cell integrin αvβ3 cooperates with platelets to promote extravasation and metastasis from the blood stream.Monocyte Subsets Coregulate Inflammatory Responses by Integrated Signaling through TNF and IL-6 at the Endothelial Cell Interface.Sertoli-germ cell junctions in the testis: a review of recent data Blockade but not overexpression of the junctional adhesion molecule C influences virus-induced type 1 diabetes in mice.Blocking junctional adhesion molecule C enhances dendritic cell migration and boosts the immune responses against Leishmania major.Effects of HIV-1 infection in vitro on transendothelial migration by monocytes and monocyte-derived macrophages.Local activation of dendritic cells alters the pathogenesis of autoimmune disease in the retina.Neutrophil reverse migration becomes transparent with zebrafish.Monocyte trafficking across the vessel wall Direct comparison of microglial dynamics and inflammatory profile in photothrombotic and arterial occlusion evoked stroke.Neutrophil heterogeneity in health and disease: a revitalized avenue in inflammation and immunity.Reverse-migrated neutrophils regulated by JAM-C are involved in acute pancreatitis-associated lung injury.Monocyte ADAM17 promotes diapedesis during transendothelial migration: identification of steps and substrates targeted by metalloproteinases Direct observations of the kinetics of migrating T cells suggest active retention by endothelial cells with continual bidirectional migration.Gene expression profiling in autoimmune noninfectious uveitis disease.Endothelial signaling in paracellular and transcellular leukocyte transmigration Reverse leukocyte migration can be attractive or repulsive.Microbial induction of vascular pathology in the CNS.PRL-3 promotes cell adhesion by interacting with JAM2 in colon cancer.Oxaprozin-induced apoptosis on CD40 ligand-treated human primary monocytes is associated with the modulation of defined intracellular pathways.Synovial tissues concentrate secreted APRIL.Recent developments and complexities in neutrophil transmigration Monocyte and macrophage dynamics during atherogenesis.Function of junctional adhesion molecules (JAMs) in leukocyte migration and homeostasis.Shedding light on cutaneous innate immune responses: the intravital microscopy approach.Neutrophil and monocyte recruitment by PECAM, CD99, and other molecules via the LBRC Tight junction dynamics: the role of junctional adhesion molecules (JAMs).Similarities and differences in the regulation of leukocyte extravasation and vascular permeability.JAM-related proteins in mucosal homeostasis and inflammation How endothelial cells regulate transmigration of leukocytes in the inflammatory response.Endothelial Cell Junctional Adhesion Molecules: Role and Regulation of Expression in Inflammation.Reverse transendothelial cell migration in inflammation: to help or to hinder?

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P2860

JAM-C regulates unidirectional monocyte transendothelial migration in inflammation.

http://www.wikidata.org/entity/Q30480098

description

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2007年の論文

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2007年学术文章

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2007年学术文章

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2007年学术文章

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2007年學術文章

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name

JAM-C regulates unidirectional monocyte transendothelial migration in inflammation.

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JAM-C regulates unidirectional monocyte transendothelial migration in inflammation.

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type

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JAM-C regulates unidirectional monocyte transendothelial migration in inflammation.

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JAM-C regulates unidirectional monocyte transendothelial migration in inflammation.

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JAM-C regulates unidirectional monocyte transendothelial migration in inflammation.

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JAM-C regulates unidirectional monocyte transendothelial migration in inflammation.

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JAM-C regulates unidirectional monocyte transendothelial migration in inflammation.

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Christiane Ody

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Christoph Scheiermann

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Francis W Luscinskas

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G Ed Rainger

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Gerard B Nash

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Marijana Miljkovic-Licina

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Paul F Bradfield

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Sussan Nourshargh

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P2860

JAM-C is a component of desmosomes and a ligand for CD11b/CD18-mediated neutrophil transepithelial migration

Antibody against junctional adhesion molecule-C inhibits angiogenesis and tumor growth

The junctional adhesion molecule 3 (JAM-3) on human platelets is a counterreceptor for the leukocyte integrin Mac-1

Cloning of human junctional adhesion molecule 3 (JAM3) and its identification as the JAM2 counter-receptor

Junctional adhesion molecule-2 (JAM-2) promotes lymphocyte transendothelial migration

JAM2 interacts with alpha4beta1. Facilitation by JAM3

The JAM family of junctional adhesion molecules

Heterogeneity of endothelial junctions is reflected by differential expression and specific subcellular localization of the three JAM family members

Junctional adhesion molecules and interendothelial junctions

Vascular endothelial-junctional adhesion molecule (VE-JAM)/JAM 2 interacts with T, NK, and dendritic cells through JAM 3

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7

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110

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Michel Aurrand-Lions

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6213265

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