PRL-3 promotes cell adhesion by interacting with JAM2 in colon cancer. - Test2 - elhamkhani - TriplyDB

PRL-3 promotes cell adhesion by interacting with JAM2 in colon cancer.

PRL-3 promotes cell adhesion by interacting with JAM2 in colon cancer.

http://www.wikidata.org/entity/Q37204478

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Transcriptome profiling analysis reveals biomarkers in colon cancer samples of various differentiation.Knockdown of PRL-3 increases mitochondrial superoxide anion production through transcriptional regulation of RAP1

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PRL-3 promotes cell adhesion by interacting with JAM2 in colon cancer.

http://www.wikidata.org/entity/Q37204478

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 11 July 2016

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vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

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name

PRL-3 promotes cell adhesion by interacting with JAM2 in colon cancer.

@en

PRL-3 promotes cell adhesion by interacting with JAM2 in colon cancer.

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type

label

PRL-3 promotes cell adhesion by interacting with JAM2 in colon cancer.

@en

PRL-3 promotes cell adhesion by interacting with JAM2 in colon cancer.

@nl

prefLabel

PRL-3 promotes cell adhesion by interacting with JAM2 in colon cancer.

@en

PRL-3 promotes cell adhesion by interacting with JAM2 in colon cancer.

@nl

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Oncology Letters

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PRL-3 promotes cell adhesion by interacting with JAM2 in colon cancer.

@en

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Chengchao Shou

http://www.w3.org/2001/XMLSchema#string

Like Qu

http://www.w3.org/2001/XMLSchema#string

Lin Meng

http://www.w3.org/2001/XMLSchema#string

Shenyi Lian

http://www.w3.org/2001/XMLSchema#string

Xiaofang Xing

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Yongyong Yang

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Metastasis-associated PRL-3 induces EGFR activation and addiction in cancer cells

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Microenvironmental regulation of tumor progression and metastasis

JAM-C regulates unidirectional monocyte transendothelial migration in inflammation.

A role of autophagy in PTP4A3-driven cancer progression.

Movers and shakers: cell cytoskeleton in cancer metastasis.

Putative "stemness" gene jam-B is not required for maintenance of stem cell state in embryonic, neural, or hematopoietic stem cells.

The role of adhesion molecules as biomarkers for the aggressive prostate cancer phenotype.

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1661-1666

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scholarly article

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10.3892/OL.2016.4836

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3

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12

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2016-07-11T00:00:00Z

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27588115

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4998106

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