Impaired astrocytic gap junction coupling and potassium buffering in a mouse model of tuberous sclerosis complex. - Test2 - elhamkhani - TriplyDB

Impaired astrocytic gap junction coupling and potassium buffering in a mouse model of tuberous sclerosis complex.

Impaired astrocytic gap junction coupling and potassium buffering in a mouse model of tuberous sclerosis complex.

http://www.wikidata.org/entity/Q30491063

about

Transplantation of specific human astrocytes promotes functional recovery after spinal cord injury Ion Homeostasis in Rhythmogenesis: The Interplay Between Neurons and Astroglia Roles of gap junctions, connexins, and pannexins in epilepsy Epilepsy related to developmental tumors and malformations of cortical development Role of astrocytes in epilepsy Precursor cell biology and the development of astrocyte transplantation therapies: lessons from spinal cord injury New vistas on astroglia in convulsive and non-convulsive epilepsy highlight novel astrocytic targets for treatment Inflammatory mechanisms contribute to the neurological manifestations of tuberous sclerosis complex.Stochastic model of Tsc1 lesions in mouse brain Tsc2 gene inactivation causes a more severe epilepsy phenotype than Tsc1 inactivation in a mouse model of tuberous sclerosis complex.Reciprocal regulation of epileptiform neuronal oscillations and electrical synapses in the rat hippocampus.Intracellular polyamines enhance astrocytic coupling.Autism spectrum disorders in tuberous sclerosis: pathogenetic pathways and implications for treatment.Mechanisms of neurocognitive dysfunction and therapeutic considerations in tuberous sclerosis complex.Astrocyte dysfunction in temporal lobe epilepsy: K+ channels and gap junction coupling.Tuberous sclerosis and epilepsy: role of astrocytes.Mechanisms underlying blood-brain barrier dysfunction in brain pathology and epileptogenesis: role of astroglia.Evolving neurobiology of tuberous sclerosis complex.Novel astrocyte targets: new avenues for the therapeutic treatment of epilepsy The impact of the glial spatial buffering on the K(+) Nernst potential.Noradrenergic β-Adrenoceptor-Mediated Intracellular Molecular Mechanism of Na-K ATPase Subunit Expression in C6 Cells."Targeting astrocytes in CNS injury and disease: A translational research approach".Review: Roles for astrocytes in epilepsy: insights from malformations of cortical development.Microglial activation during epileptogenesis in a mouse model of tuberous sclerosis complex.Temporary sequestration of potassium by mitochondria in astrocytes Spinal astrocytes stimulated by tumor necrosis factor-α and/or interferon-γ attenuate connexin 43-gap junction via c-jun terminal kinase activity.Postnatal reduction of tuberous sclerosis complex 1 expression in astrocytes and neurons causes seizures in an age-dependent manner.Epilepsy and astrocyte energy metabolism.Astrocytic Kir4.1 channels and gap junctions account for spontaneous epileptic seizure.

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P2860

Impaired astrocytic gap junction coupling and potassium buffering in a mouse model of tuberous sclerosis complex.

http://www.wikidata.org/entity/Q30491063

description

2009 nî lūn-bûn

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2009 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2009 թվականի մայիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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Impaired astrocytic gap juncti ...... of tuberous sclerosis complex.

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Impaired astrocytic gap juncti ...... of tuberous sclerosis complex.

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Impaired astrocytic gap juncti ...... of tuberous sclerosis complex.

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Impaired astrocytic gap juncti ...... of tuberous sclerosis complex.

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J.NBD.2009.01.010

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Neurobiology of Disease

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Impaired astrocytic gap juncti ...... of tuberous sclerosis complex.

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Lin Xu

http://www.w3.org/2001/XMLSchema#string

Ling-Hui Zeng

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Michael Wong

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P2860

Epileptogenesis and reduced inward rectifier potassium current in tuberous sclerosis complex-1-deficient astrocytes

The tuberous sclerosis complex

Rapamycin prevents epilepsy in a mouse model of tuberous sclerosis complex.

Upregulation in astrocytic connexin 43 gap junction levels may exacerbate generalized seizures in mesial temporal lobe epilepsy.

Reduction of K+ uptake in glia prevents long-term depression maintenance and causes epileptiform activity.

Functional specialization and topographic segregation of hippocampal astrocytes.

Astrocytes in the epileptic brain.

Seizure-promoting effect of blood-brain barrier disruption.

Expression of connexin 43 and connexin 32 gap-junction proteins in epilepsy-associated brain tumors and in the perilesional epileptic cortex.

Tuberous sclerosis: from tubers to mTOR.

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291-299

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10.1016/J.NBD.2009.01.010

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