The SWI/SNF ATPases Are Required for Triple Negative Breast Cancer Cell Proliferation - Test2 - elhamkhani - TriplyDB

The SWI/SNF ATPases Are Required for Triple Negative Breast Cancer Cell Proliferation

The SWI/SNF ATPases Are Required for Triple Negative Breast Cancer Cell Proliferation

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BRG1 promotes hepatocarcinogenesis by regulating proliferation and invasiveness Identification of BRCA1-like triple-negative breast cancers by quantitative multiplex-ligation-dependent probe amplification (MLPA) analysis of BRCA1-associated chromosomal regions: a validation study.SMARCA4 regulates gene expression and higher-order chromatin structure in proliferating mammary epithelial cells.Targeting the chromatin remodeling enzyme BRG1 increases the efficacy of chemotherapy drugs in breast cancer cells The BET inhibitor JQ1 selectively impairs tumour response to hypoxia and downregulates CA9 and angiogenesis in triple negative breast cancer.Mammalian SWI/SNF Enzymes and the Epigenetics of Tumor Cell Metabolic Reprogramming.Epigenomic regulation of oncogenesis by chromatin remodeling.Panel sequencing of 264 candidate susceptibility genes and segregation analysis in a cohort of non-BRCA1, non-BRCA2 breast cancer families.BRM polymorphisms, pancreatic cancer risk and survival.The BRG1 chromatin remodeling enzyme links cancer cell metabolism and proliferation Diversity of cis-regulatory elements associated with auxin response in Arabidopsis thaliana.Brg1 inhibits E-cadherin expression in lung epithelial cells and disrupts epithelial integrity.A New Way Forward in Cancer Drug Discovery: Inhibiting the SWI/SNF Chromatin Remodelling Complex.High expression of SMARCA4 or SMARCA2 is frequently associated with an opposite prognosis in cancer.CRISPR-Cas9: a promising genetic engineering approach in cancer research.Epigenetic regulation in cell senescence.The BRG1 ATPase of human SWI/SNF chromatin remodeling enzymes as a driver of cancer.The Epigenetics of Triple-Negative and Basal-Like Breast Cancer: Current Knowledge

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The SWI/SNF ATPases Are Required for Triple Negative Breast Cancer Cell Proliferation

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2015 nî lūn-bûn

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2015 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2015 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

The SWI/SNF ATPases Are Required for Triple Negative Breast Cancer Cell Proliferation

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The SWI/SNF ATPases Are Required for Triple Negative Breast Cancer Cell Proliferation

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The SWI/SNF ATPases Are Required for Triple Negative Breast Cancer Cell Proliferation

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The SWI/SNF ATPases Are Required for Triple Negative Breast Cancer Cell Proliferation

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The SWI/SNF ATPases Are Required for Triple Negative Breast Cancer Cell Proliferation

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The SWI/SNF ATPases Are Required for Triple Negative Breast Cancer Cell Proliferation

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Journal of Cellular Physiology

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The SWI/SNF ATPases Are Required for Triple Negative Breast Cancer Cell Proliferation

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Andre J van Wijnen

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Anthony N Imbalzano

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Gary S Stein

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Georg E Winter

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Gillian Browne

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Greenfield Sluder

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Jacqueline Akech

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James E Bradner

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Jane B Lian

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Janet L Stein

http://www.w3.org/2001/XMLSchema#string

P2860

The spectrum of SWI/SNF mutations, ubiquitous in human cancers

Reconstitution of a core chromatin remodeling complex from SWI/SNF subunits

BRCA1 is associated with a human SWI/SNF-related complex: linking chromatin remodeling to breast cancer

SWI/SNF complex interacts with tumor suppressor p53 and is necessary for the activation of p53-mediated transcription

Purification and biochemical heterogeneity of the mammalian SWI-SNF complex

The retinoblastoma protein and BRG1 form a complex and cooperate to induce cell cycle arrest

Smurf2 up-regulation activates telomere-dependent senescence

Dissecting the genomic complexity underlying medulloblastoma

Comprehensive molecular portraits of human breast tumours

The first 30 years of p53: growing ever more complex

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10.1002/JCP.24991

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11

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230

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2015-11-01T00:00:00Z

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4516601

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