Reduced severity of oxygen-induced retinopathy in eNOS-deficient mice.
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The matricellular protein cysteine-rich protein 61 (CCN1/Cyr61) enhances physiological adaptation of retinal vessels and reduces pathological neovascularization associated with ischemic retinopathyMore weapons in the arsenal against ischemic retinopathyN-CAM exhibits a regulatory function in pathological angiogenesis in oxygen induced retinopathyRegulation of pathologic retinal angiogenesis in mice and inhibition of VEGF-VEGFR2 binding by soluble heparan sulfateRetinal features predictive of progressive stage 4 retinopathy of prematurity.Vascular dysfunction in retinopathy-an emerging role for arginaseRetinopathy of prematurity: understanding ischemic retinal vasculopathies at an extreme of life.eNOS-beta-actin interaction contributes to increased peroxynitrite formation during hyperoxia in pulmonary artery endothelial cells and mouse lungs.The role of genetic polymorphisms in antioxidant enzymes and potential antioxidant therapies in neonatal lung diseaseArginase 2 deficiency prevents oxidative stress and limits hyperoxia-induced retinal vascular degeneration.Novel peptide for attenuation of hyperoxia-induced disruption of lung endothelial barrier and pulmonary edema via modulating peroxynitrite formationDocosahexaenoic acid improves the nitroso-redox balance and reduces VEGF-mediated angiogenic signaling in microvascular endothelial cells.Arginase in retinopathy.Inhibition of NAD(P)H oxidase activity blocks vascular endothelial growth factor overexpression and neovascularization during ischemic retinopathy.Knockout of insulin and IGF-1 receptors on vascular endothelial cells protects against retinal neovascularization.Deletion of thioredoxin interacting protein (TXNIP) augments hyperoxia-induced vaso-obliteration in a mouse model of oxygen induced-retinopathy.Experimental diabetes causes breakdown of the blood-retina barrier by a mechanism involving tyrosine nitration and increases in expression of vascular endothelial growth factor and urokinase plasminogen activator receptor.Essential role of sphingosine 1-phosphate receptor 2 in pathological angiogenesis of the mouse retina.MEF2C ablation in endothelial cells reduces retinal vessel loss and suppresses pathologic retinal neovascularization in oxygen-induced retinopathyeNOS overexpression exacerbates vascular closure in the obliterative phase of OIR and increases angiogenic drive in the subsequent proliferative stageHypoxia-induced oxidative stress in ischemic retinopathyEndothelial nitric oxide synthase gene T-786C and 27-bp repeat gene polymorphisms in retinopathy of prematurity.Attenuation of retinal vascular development and neovascularization in PECAM-1-deficient miceNeutralizing VEGF decreases tortuosity and alters endothelial cell division orientation in arterioles and veins in a rat model of ROP: relevance to plus diseaseTrans-arachidonic acids generated during nitrative stress induce a thrombospondin-1-dependent microvascular degenerationHigh glucose promotes retinal endothelial cell migration through activation of Src, PI3K/Akt1/eNOS, and ERKs.Role of Nrf2 in retinal vascular development and the vaso-obliterative phase of oxygen-induced retinopathyBioelectric impact of pathological angiogenesis on vascular function.The effects of oxygen stresses on the development of features of severe retinopathy of prematurity: knowledge from the 50/10 OIR model.Mechanisms and management of retinopathy of prematurity.Arginase: an old enzyme with new tricks.Inhibition of tumor necrosis factor-alpha improves physiological angiogenesis and reduces pathological neovascularization in ischemic retinopathy.Advances in understanding and management of retinopathy of prematurity.Dimethylarginine dimethylaminohydrolase-2 deficiency promotes vascular regeneration and attenuates pathological angiogenesis.Signaling pathways triggered by oxidative stress that mediate features of severe retinopathy of prematurity.The role of supplemental oxygen and JAK/STAT signaling in intravitreous neovascularization in a ROP rat model.Candesartan stimulates reparative angiogenesis in ischemic retinopathy model: role of hemeoxygenase-1 (HO-1).Ischemic Retinopathies: Oxidative Stress and Inflammation.BH4-Mediated Enhancement of Endothelial Nitric Oxide Synthase Activity Reduces Hyperoxia-Induced Endothelial Damage and Preserves Vascular Integrity in the Neonate.A Pathogenic Relationship of Bronchopulmonary Dysplasia and Retinopathy of Prematurity? A Review of Angiogenic Mediators in Both Diseases.
P2860
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P2860
Reduced severity of oxygen-induced retinopathy in eNOS-deficient mice.
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Reduced severity of oxygen-induced retinopathy in eNOS-deficient mice.
@ast
Reduced severity of oxygen-induced retinopathy in eNOS-deficient mice.
@en
type
label
Reduced severity of oxygen-induced retinopathy in eNOS-deficient mice.
@ast
Reduced severity of oxygen-induced retinopathy in eNOS-deficient mice.
@en
prefLabel
Reduced severity of oxygen-induced retinopathy in eNOS-deficient mice.
@ast
Reduced severity of oxygen-induced retinopathy in eNOS-deficient mice.
@en
P2093
P1476
Reduced severity of oxygen-induced retinopathy in eNOS-deficient mice
@en
P2093
Caldwell RB
P304
P407
P577
2001-01-01T00:00:00Z