Helicobacter pylori CagA induces Ras-independent morphogenetic response through SHP-2 recruitment and activation.
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Gastric cancer development after the successful eradication of Helicobacter pyloriEffect of Helicobacter pylori Infection on the Composition of Gastric Microbiota in the Development of Gastric CancerHelicobacter pylori Eradication in Patients with Immune Thrombocytopenic Purpura: A Review and the Role of BiogeographyEchoes of a distant past: The cag pathogenicity island of Helicobacter pyloriAn Update on Helicobacter pylori as the Cause of Gastric CancerPolymorphism in the Helicobacter pylori CagA and VacA toxins and diseaseThe Helicobacter pylori cag Pathogenicity IslandDramatic increase in SHP2 binding activity of Helicobacter pylori Western CagA by EPIYA-C duplication: its implications in gastric carcinogenesisTyrosine phosphatase SHP2 regulates the expression of acyl-CoA synthetase ACSL4Oncogenic mechanisms of the Helicobacter pylori CagA proteinHelicobacter pylori CagA: a new paradigm for bacterial carcinogenesisA specific A/T polymorphism in Western tyrosine phosphorylation B-motifs regulates Helicobacter pylori CagA epithelial cell interactionsAssociation of CagA EPIYA-D or EPIYA-C phosphorylation sites with peptic ulcer and gastric cancer risks: A meta-analysis.Helicobacter pylori CagA induces a transition from polarized to invasive phenotypes in MDCK cells.Helicobacter pylori CagA induces AGS cell elongation through a cell retraction defect that is independent of Cdc42, Rac1, and Arp2/3.Host-pathogen systems biology: logical modelling of hepatocyte growth factor and Helicobacter pylori induced c-Met signal transductionDiscovering differential protein expression caused by CagA-induced ERK pathway activation in AGS cells using the SELDI-ProteinChip platform.In vivo accumulation of Helicobacter pylori products, NOD1, ubiquitinated proteins and proteasome in a novel cytoplasmic structureGenetic Alterations in Gastric Cancer Associated with Helicobacter pylori Infection.Epidemiological link between gastric disease and polymorphisms in VacA and CagA.Adherence and invasion of mouse-adapted H pylori in different epithelial cell lines.Helicobacter pylori CagA phosphorylation status determines the gp130-activated SHP2/ERK and JAK/STAT signal transduction pathways in gastric epithelial cells.NF-kappaB activation and potentiation of proinflammatory responses by the Helicobacter pylori CagA protein.Helicobacter pylori CagA triggers expression of the bactericidal lectin REG3γ via gastric STAT3 activationHelicobacter pylori and gastric cancer: factors that modulate disease risk.Heme oxygenase-1 dysregulates macrophage polarization and the immune response to Helicobacter pylori.Helicobacter pylori exploits cholesterol-rich microdomains for induction of NF-kappaB-dependent responses and peptidoglycan delivery in epithelial cellsHigher glucose level can enhance the H. pylori adhesion and virulence related with type IV secretion system in AGS cells.Gastric cancer, Helicobacter pylori infection and other risk factors.Polymorphisms in the intermediate region of VacA impact Helicobacter pylori-induced disease development.Helicobacter pylori initiates a mesenchymal transition through ZEB1 in gastric epithelial cells.Helicobacter pylori homB, but not cagA, is associated with gastric cancer in IranHelicobacter pylori cag pathogenicity island's role in B7-H1 induction and immune evasion.Modification of Helicobacter pylori Peptidoglycan Enhances NOD1 Activation and Promotes Cancer of the Stomach.Grb2-associated binder 1 polymorphism was associated with the risk of Helicobactor pylori infection and gastric atrophyHelicobacter pylori modulates host cell survival regulation through the serine-threonine kinase, 3-phosphoinositide dependent kinase 1 (PDK-1).SHP-2 Mediates Cryptosporidium parvum Infectivity in Human Intestinal Epithelial Cells.H. pylori infection: bacterial virulence factors and cytokine gene polymorphisms as determinants of infection outcome.Orchestration of aberrant epithelial signaling by Helicobacter pylori CagA.An RGD helper sequence in CagL of Helicobacter pylori assists in interactions with integrins and injection of CagA
P2860
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P2860
Helicobacter pylori CagA induces Ras-independent morphogenetic response through SHP-2 recruitment and activation.
description
2004 nî lūn-bûn
@nan
2004 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Helicobacter pylori CagA induc ...... -2 recruitment and activation.
@ast
Helicobacter pylori CagA induc ...... -2 recruitment and activation.
@en
Helicobacter pylori CagA induc ...... -2 recruitment and activation.
@nl
type
label
Helicobacter pylori CagA induc ...... -2 recruitment and activation.
@ast
Helicobacter pylori CagA induc ...... -2 recruitment and activation.
@en
Helicobacter pylori CagA induc ...... -2 recruitment and activation.
@nl
prefLabel
Helicobacter pylori CagA induc ...... -2 recruitment and activation.
@ast
Helicobacter pylori CagA induc ...... -2 recruitment and activation.
@en
Helicobacter pylori CagA induc ...... -2 recruitment and activation.
@nl
P2093
P2860
P356
P1476
Helicobacter pylori CagA induc ...... -2 recruitment and activation.
@en
P2093
Akihiro Nakaya
Atsushi Takahashi
Hideaki Higashi
Kazuyuki Yokoyama
Masanori Hatakeyama
Megumi Higuchi
Ryouhei Tsutsumi
Shinya Tanaka
Susumu Ishikawa
Takeshi Azuma
P2860
P304
17205-17216
P356
10.1074/JBC.M309964200
P407
P577
2004-02-12T00:00:00Z