Adaptive immune neuroprotection in G93A-SOD1 amyotrophic lateral sclerosis mice. - Test2 - elhamkhani - TriplyDB

Adaptive immune neuroprotection in G93A-SOD1 amyotrophic lateral sclerosis mice.

Adaptive immune neuroprotection in G93A-SOD1 amyotrophic lateral sclerosis mice.

http://www.wikidata.org/entity/Q33354106

about

Is spinal muscular atrophy a disease of the motor neurons only: pathogenesis and therapeutic implications?Neuroinflammation in motor neuron disease Bridge between neuroimmunity and traumatic brain injury Early functional deficit and microglial disturbances in a mouse model of amyotrophic lateral sclerosis Targeting miR-155 restores abnormal microglia and attenuates disease in SOD1 mice.Ablation of proliferating cells in the CNS exacerbates motor neuron disease caused by mutant superoxide dismutase Excess circulating alternatively activated myeloid (M2) cells accelerate ALS progression while inhibiting experimental autoimmune encephalomyelitis Mechanisms underlying inflammation in neurodegeneration Regulatory T cells attenuate Th17 cell-mediated nigrostriatal dopaminergic neurodegeneration in a model of Parkinson's disease.Murine motor and behavior functional evaluations for acute 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP) intoxication.Immune dysregulation may contribute to disease pathogenesis in spinal muscular atrophy mice Inflammation in ALS and SMA: sorting out the good from the evil.Control of neuroinflammation as a therapeutic strategy for amyotrophic lateral sclerosis and other neurodegenerative disorders Functional proteomic analysis for regulatory T cell surveillance of the HIV-1-infected macrophage.Effects of facial nerve axotomy on Th2-associated and Th1-associated chemokine mRNA expression in the facial motor nucleus of wild-type and presymptomatic SOD1 mice NP001 regulation of macrophage activation markers in ALS: a phase I clinical and biomarker study Abnormal changes in NKT cells, the IGF-1 axis, and liver pathology in an animal model of ALS.SOD1(G93A) transgenic mouse CD4(+) T cells mediate neuroprotection after facial nerve axotomy when removed from a suppressive peripheral microenvironment.Glycans in sera of amyotrophic lateral sclerosis patients and their role in killing neuronal cells Neuroinflammation modulates distinct regional and temporal clinical responses in ALS mice.Endogenous regulatory T lymphocytes ameliorate amyotrophic lateral sclerosis in mice and correlate with disease progression in patients with amyotrophic lateral sclerosis.Early-stage treatment with Withaferin A reduces levels of misfolded superoxide dismutase 1 and extends lifespan in a mouse model of amyotrophic lateral sclerosis.Dual destructive and protective roles of adaptive immunity in neurodegenerative disorders.The double-edged sword of autoimmunity: lessons from multiple sclerosis.Effect of thymic stimulation of CD4+ T cell expansion on disease onset and progression in mutant SOD1 mice.The Role of immune and inflammatory mechanisms in ALS.Protective and Toxic Neuroinflammation in Amyotrophic Lateral Sclerosis.A lifespan approach to neuroinflammatory and cognitive disorders: a critical role for glia.Humoral factors in ALS patients during disease progression.Modulating inflammatory monocytes with a unique microRNA gene signature ameliorates murine ALS.Motor neuron-immune interactions: the vicious circle of ALS.Brain regeneration in physiology and pathology: the immune signature driving therapeutic plasticity of neural stem cells Regulatory T-lymphocytes mediate amyotrophic lateral sclerosis progression and survival Neuroprotective and Angiogenic Effects of Bone Marrow Transplantation Combined With Granulocyte Colony-Stimulating Factor in a Mouse Model of Amyotrophic Lateral Sclerosis.Th17 Cell Response in SOD1G93A Mice following Motor Nerve Injury Increased ratio of circulating neutrophils to monocytes in amyotrophic lateral sclerosis.Infiltration of CD4+ lymphocytes into the brain contributes to neurodegeneration in a mouse model of Parkinson disease.Nitrated {alpha}-synuclein-induced alterations in microglial immunity are regulated by CD4+ T cell subsets CD 4+ T cells in the pathobiology of neurodegenerative disorders.Thymic involution, a co-morbidity factor in amyotrophic lateral sclerosis.

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Adaptive immune neuroprotection in G93A-SOD1 amyotrophic lateral sclerosis mice.

http://www.wikidata.org/entity/Q33354106

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Adaptive immune neuroprotection in G93A-SOD1 amyotrophic lateral sclerosis mice.

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Adaptive immune neuroprotection in G93A-SOD1 amyotrophic lateral sclerosis mice.

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type

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Adaptive immune neuroprotection in G93A-SOD1 amyotrophic lateral sclerosis mice.

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Adaptive immune neuroprotection in G93A-SOD1 amyotrophic lateral sclerosis mice.

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Adaptive immune neuroprotection in G93A-SOD1 amyotrophic lateral sclerosis mice.

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Adaptive immune neuroprotection in G93A-SOD1 amyotrophic lateral sclerosis mice.

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Adaptive immune neuroprotection in G93A-SOD1 amyotrophic lateral sclerosis mice.

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Ashley D Reynolds

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Howard E Gendelman

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R Lee Mosley

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P2860

SOD1 mutations disrupt redox-sensitive Rac regulation of NADPH oxidase in a familial ALS model

Glial cells as intrinsic components of non-cell-autonomous neurodegenerative disease

Glatiramer acetate fights against Alzheimer's disease by inducing dendritic-like microglia expressing insulin-like growth factor 1

Therapeutic vaccine for acute and chronic motor neuron diseases: implications for amyotrophic lateral sclerosis.

Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis

Design, power, and interpretation of studies in the standard murine model of ALS

Effect of nitric oxide on lymphocytes from sporadic amyotrophic lateral sclerosis patients: toxic or protective role?

Modified expression of Bcl-2 and SOD1 proteins in lymphocytes from sporadic ALS patients

Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation

ALS: a disease of motor neurons and their nonneuronal neighbors

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