Severe learning deficits of IRSp53 mutant mice are caused by altered NMDA receptor dependent signal transduction.
about
Redundant functions of I-BAR family members, IRSp53 and IRTKS, are essential for embryonic development.The neuronal insulin receptor in its environment.IRSp53 accumulates at the postsynaptic density under excitatory conditions.Comprehensive behavioral analysis of the Cdkl5 knockout mice revealed significant enhancement in anxiety- and fear-related behaviors and impairment in both acquisition and long-term retention of spatial reference memory.O-GlcNAc cycling in the developing, adult and geriatric brain.
P2860
Severe learning deficits of IRSp53 mutant mice are caused by altered NMDA receptor dependent signal transduction.
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2015 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
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2015 թվականի նոյեմբերին հրատարակված գիտական հոդված
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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name
Severe learning deficits of IR ...... dependent signal transduction.
@ast
Severe learning deficits of IR ...... dependent signal transduction.
@en
type
label
Severe learning deficits of IR ...... dependent signal transduction.
@ast
Severe learning deficits of IR ...... dependent signal transduction.
@en
prefLabel
Severe learning deficits of IR ...... dependent signal transduction.
@ast
Severe learning deficits of IR ...... dependent signal transduction.
@en
P2860
P356
P1476
Severe learning deficits of IR ...... dependent signal transduction.
@en
P2093
Hans-Jürgen Kreienkamp
Kristin Bobsin
P2860
P356
10.1111/JNC.13428
P407
P577
2015-11-12T00:00:00Z