Oxidative stress-induced JNK activation contributes to proinflammatory phenotype of aging diabetic mesangial cells.

Oxidative stress-induced JNK activation contributes to proinflammatory phenotype of aging diabetic mesangial cells.

Item
http://www.wikidata.org/entity/Q33570113
Induction of diabetes in aged C57B6 mice results in severe nephropathy: an association with oxidative stress, endoplasmic reticulum stress, and inflammationThe modified selenenyl amide, M-hydroxy ebselen, attenuates diabetic nephropathy and diabetes-associated atherosclerosis in ApoE/GPx1 double knockout miceElevated muscle TLR4 expression and metabolic endotoxemia in human aging.Resveratrol retards progression of diabetic nephropathy through modulations of oxidative stress, proinflammatory cytokines, and AMP-activated protein kinase.Hypoxia Potentiates Palmitate-induced Pro-inflammatory Activation of Primary Human Macrophages.A gene expression study of normal and damaged cartilage in anteromedial gonarthrosis, a phenotype of osteoarthritis.Relationship between oxidative stress and inflammatory cytokines in diabetic nephropathy.Oxidative stress in the etiology of age-associated decline in glucose metabolism.Targeted functional investigations guided by integrative proteome network analysis revealed significant perturbations of renal tubular cell functions induced by high glucose.Mechanisms of MPP⁺-induced PC12 cell apoptosis via reactive oxygen species.Heme oxygenase-1 protects spinal cord neurons from hydrogen peroxide-induced apoptosis via suppression of Cdc42/MLK3/MKK7/JNK3 signaling.Identification of PTPN1 as a novel negative regulator of the JNK MAPK pathway using a synthetic screening for pathway-specific phosphatasesBradykinin inhibits high glucose- and growth factor-induced collagen synthesis in mesangial cells through the B2-kinin receptor.Dissecting cellular senescence and SASP in Drosophila.p-Cresol sulfate and indoxyl sulfate induce similar cellular inflammatory gene expressions in cultured proximal renal tubular cells.Optimal LDR to Protect the Kidney From Diabetes: Whole-Body Exposure to 25 mGy X-rays Weekly for 8 Weeks Efficiently Attenuates Renal Damage in Diabetic Mice
P2860

Oxidative stress-induced JNK activation contributes to proinflammatory phenotype of aging diabetic mesangial cells.

Item
http://www.wikidata.org/entity/Q33570113
description
2009 nî lūn-bûn
@nan
2009 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Oxidative stress-induced JNK a ...... ging diabetic mesangial cells.
@ast
Oxidative stress-induced JNK a ...... ging diabetic mesangial cells.
@en
Oxidative stress-induced JNK a ...... ging diabetic mesangial cells.
@nl
type
label
Oxidative stress-induced JNK a ...... ging diabetic mesangial cells.
@ast
Oxidative stress-induced JNK a ...... ging diabetic mesangial cells.
@en
Oxidative stress-induced JNK a ...... ging diabetic mesangial cells.
@nl
prefLabel
Oxidative stress-induced JNK a ...... ging diabetic mesangial cells.
@ast
Oxidative stress-induced JNK a ...... ging diabetic mesangial cells.
@en
Oxidative stress-induced JNK a ...... ging diabetic mesangial cells.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P433
P478
P577
P5875
P698
P932
P356
P1433
P1476
Oxidative stress-induced JNK a ...... ging diabetic mesangial cells.
@en
P2093
P2860
P304
P31
P356
10.1152/AJPRENAL.00078.2009
P433
P478
P577
P5875
P698
P932