Immunopathology of cerebral malaria: morphological evidence of parasite sequestration in murine brain microvasculature
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Cerebral malaria: mysteries at the blood-brain barrierPerivascular Arrest of CD8+ T Cells Is a Signature of Experimental Cerebral MalariaA rapid murine coma and behavior scale for quantitative assessment of murine cerebral malariaExperimental cerebral malaria pathogenesis--hemodynamics at the blood brain barrierGenome wide analysis of inbred mouse lines identifies a locus containing Ppar-gamma as contributing to enhanced malaria survivalNeuroimmunological blood brain barrier opening in experimental cerebral malariaAbsence of apolipoprotein E protects mice from cerebral malariaPlasmodium cysteine repeat modular proteins 1-4: complex proteins with roles throughout the malaria parasite life cyclePharmacologic inhibition of CXCL10 in combination with anti-malarial therapy eliminates mortality associated with murine model of cerebral malariaInhaled nitric oxide reduces endothelial activation and parasite accumulation in the brain, and enhances survival in experimental cerebral malariaSequestration and Tissue Accumulation of Human Malaria Parasites: Can We Learn Anything from Rodent Models of Malaria?Host erythrocyte environment influences the localization of exported protein 2, an essential component of the Plasmodium translocon.Murine malaria parasite sequestration: CD36 is the major receptor, but cerebral pathology is unlinked to sequestration.Pathogenesis of cerebral malaria: recent experimental data and possible applications for humans.Role of CD40-CVD40L in mouse severe malaria.Oral activated charcoal prevents experimental cerebral malaria in mice and in a randomized controlled clinical trial in man did not interfere with the pharmacokinetics of parenteral artesunate.Host Resistance to Plasmodium-Induced Acute Immune Pathology Is Regulated by Interleukin-10 Receptor Signaling.Unlike the synchronous Plasmodium falciparum and P. chabaudi infection, the P. berghei and P. yoelii asynchronous infections are not affected by melatonin.Electron microscopic features of brain edema in rodent cerebral malaria in relation to glial fibrillary acidic protein expression.Natural regulatory T cells in malaria: host or parasite allies?CD8+ T cells and IFN-γ mediate the time-dependent accumulation of infected red blood cells in deep organs during experimental cerebral malaria.Species-specific inhibition of cerebral malaria in mice coinfected with Plasmodium spp.Global proteomic analysis of plasma from mice infected with Plasmodium berghei ANKA using two dimensional gel electrophoresis and matrix assisted laser desorption ionization-time of flight mass spectrometry.CD8(+)-T-cell depletion ameliorates circulatory shock in Plasmodium berghei-infected mice.Susceptibility to experimental cerebral malaria induced by Plasmodium berghei ANKA in inbred mouse strains recently derived from wild stock.Inflammatory responses associated with the induction of cerebral malaria: lessons from experimental murine modelsPersistent cognitive and motor deficits after successful antimalarial treatment in murine cerebral malariaP-selectin contributes to severe experimental malaria but is not required for leukocyte adhesion to brain microvasculatureChemokine receptor CCR2 is not essential for the development of experimental cerebral malaria.Pathogenic role of P-selectin in experimental cerebral malaria: importance of the endothelial compartmentIron prevents the development of experimental cerebral malaria by attenuating CXCR3-mediated T cell chemotaxis.Phosphatidylinositol 3-Kinase γ is required for the development of experimental cerebral malaria.The CTLA-4 and PD-1/PD-L1 inhibitory pathways independently regulate host resistance to Plasmodium-induced acute immune pathology.The Impact of HIV Coinfection on Cerebral Malaria Pathogenesis.Anti-plasmodial effects of Azadirachta indica in experimental cerebral malaria: Apoptosis of cerebellar Purkinje cells of mice as a markerReview Article: blood-brain barrier in falciparum malaria.Experimental Models of Microvascular Immunopathology: The Example of Cerebral Malaria.Inhibition of platelet adherence to brain microvasculature protects against severe Plasmodium berghei malaria.Protective and pathogenic roles of CD8+ T cells during malaria infection.Plasmodium berghei bio-burden correlates with parasite lactate dehydrogenase: application to murine Plasmodium diagnostics.
P2860
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P2860
Immunopathology of cerebral malaria: morphological evidence of parasite sequestration in murine brain microvasculature
description
2000 nî lūn-bûn
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2000 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի սեպտեմբերին հրատարակված գիտական հոդված
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2000年の論文
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2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Immunopathology of cerebral ma ...... murine brain microvasculature
@ast
Immunopathology of cerebral ma ...... murine brain microvasculature
@en
type
label
Immunopathology of cerebral ma ...... murine brain microvasculature
@ast
Immunopathology of cerebral ma ...... murine brain microvasculature
@en
prefLabel
Immunopathology of cerebral ma ...... murine brain microvasculature
@ast
Immunopathology of cerebral ma ...... murine brain microvasculature
@en
P2093
P2860
P1476
Immunopathology of cerebral ma ...... murine brain microvasculature
@en
P2093
P2860
P304
P356
10.1128/IAI.68.9.5364-5376.2000
P407
P577
2000-09-01T00:00:00Z