Murine malaria parasite sequestration: CD36 is the major receptor, but cerebral pathology is unlinked to sequestration. - Test2 - elhamkhani - TriplyDB

Murine malaria parasite sequestration: CD36 is the major receptor, but cerebral pathology is unlinked to sequestration.

Murine malaria parasite sequestration: CD36 is the major receptor, but cerebral pathology is unlinked to sequestration.

http://www.wikidata.org/entity/Q30476076

about

Human malarial disease: a consequence of inflammatory cytokine release CD36: implications in cardiovascular disease In vivo bioluminescent imaging (BLI): noninvasive visualization and interrogation of biological processes in living animals Both functional LTbeta receptor and TNF receptor 2 are required for the development of experimental cerebral malaria CD36 and Fyn kinase mediate malaria-induced lung endothelial barrier dysfunction in mice infected with Plasmodium berghei The machinery underlying malaria parasite virulence is conserved between rodent and human malaria parasites IP-10-mediated T cell homing promotes cerebral inflammation over splenic immunity to malaria infection A rapid murine coma and behavior scale for quantitative assessment of murine cerebral malaria Experimental cerebral malaria pathogenesis--hemodynamics at the blood brain barrier The exported protein PbCP1 localises to cleft-like structures in the rodent malaria parasite Plasmodium berghei Genome wide analysis of inbred mouse lines identifies a locus containing Ppar-gamma as contributing to enhanced malaria survival Visualisation and quantitative analysis of the rodent malaria liver stage by real time imaging Neuroimmunological blood brain barrier opening in experimental cerebral malaria Absence of apolipoprotein E protects mice from cerebral malaria The role of animal models for research on severe malaria STEVOR is a Plasmodium falciparum erythrocyte binding protein that mediates merozoite invasion and rosetting Enlightening the malaria parasite life cycle: bioluminescent Plasmodium in fundamental and applied research Bioluminescent imaging of Trypanosoma brucei shows preferential testis dissemination which may hamper drug efficacy in sleeping sickness A novel 'gene insertion/marker out' (GIMO) method for transgene expression and gene complementation in rodent malaria parasites Salivary gland-specific P. berghei reporter lines enable rapid evaluation of tissue-specific sporozoite loads in mosquitoes Pharmacologic inhibition of CXCL10 in combination with anti-malarial therapy eliminates mortality associated with murine model of cerebral malaria Glucagon-like peptide-1 analogue, liraglutide, in experimental cerebral malaria: implications for the role of oxidative stress in cerebral malaria Sequestration and Tissue Accumulation of Human Malaria Parasites: Can We Learn Anything from Rodent Models of Malaria?Proteomic and genetic analyses demonstrate that Plasmodium berghei blood stages export a large and diverse repertoire of proteins Host erythrocyte environment influences the localization of exported protein 2, an essential component of the Plasmodium translocon.A role for natural regulatory T cells in the pathogenesis of experimental cerebral malaria.Imaging Plasmodium immunobiology in the liver, brain, and lung.Effect of mature blood-stage Plasmodium parasite sequestration on pathogen biomass in mathematical and in vivo models of malaria.Infection by and protective immune responses against Plasmodium berghei ANKA are not affected in macrophage scavenger receptors A deficient mice.Simultaneous host and parasite expression profiling identifies tissue-specific transcriptional programs associated with susceptibility or resistance to experimental cerebral malaria Imaging host-pathogen interactions.Parasite burden and CD36-mediated sequestration are determinants of acute lung injury in an experimental malaria model.Murine cerebral malaria development is independent of toll-like receptor signaling A novel role for von Willebrand factor in the pathogenesis of experimental cerebral malaria Uric acid is a mediator of the Plasmodium falciparum-induced inflammatory response.Plasmepsin 4-deficient Plasmodium berghei are virulence attenuated and induce protective immunity against experimental malaria.The murine cerebral malaria phenomenon ICAM-1 is a key receptor mediating cytoadherence and pathology in the Plasmodium chabaudi malaria model Dual effect of Plasmodium-infected erythrocytes on dendritic cell maturation.CD4+ natural regulatory T cells prevent experimental cerebral malaria via CTLA-4 when expanded in vivo

P2860

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P2860

Murine malaria parasite sequestration: CD36 is the major receptor, but cerebral pathology is unlinked to sequestration.

http://www.wikidata.org/entity/Q30476076

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2005 nî lūn-bûn

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2005 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2005年の論文

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Proceedings of the National Academy of Sciences of the United States of America

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Murine malaria parasite seques ...... is unlinked to sequestration.

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Andrew P Waters

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Jai Ramesar

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Margarida Cunha-Rodrigues

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Maria Febbraio

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Marion A M den Boer

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Peter J Voshol

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Sjoerd G van Duinen

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P2860

Plasmodium falciparum MAEBL is a unique member of the ebl family

Plasmodium chabaudi-infected erythrocytes adhere to CD36 and bind to microvascular endothelial cells in an organ-specific way

Immunopathology of cerebral malaria: morphological evidence of parasite sequestration in murine brain microvasculature

Molecular mechanisms of cytoadherence in malaria.

A nonpolio enterovirus with respiratory tropism causes poliomyelitis in intercellular adhesion molecule 1 transgenic mice.

The development of genetic tools for dissecting the biology of malaria parasites.

Platelet-mediated clumping of Plasmodium falciparum-infected erythrocytes is a common adhesive phenotype and is associated with severe malaria.

CD36: a class B scavenger receptor involved in angiogenesis, atherosclerosis, inflammation, and lipid metabolism.

Cerebral malaria: the contribution of studies in animal models to our understanding of immunopathogenesis.

Cytoadherence and sequestration in Plasmodium falciparum: defining the ties that bind.

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11468-11473

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10.1073/PNAS.0503386102

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32

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Blandine Franke-Fayard

Philippe Büscher

Maria Manuel Dias da Mota

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