The High Mobility Group A1 (HMGA1) Transcriptome in Cancer and Development. - Test2 - elhamkhani - TriplyDB

The High Mobility Group A1 (HMGA1) Transcriptome in Cancer and Development.

The High Mobility Group A1 (HMGA1) Transcriptome in Cancer and Development.

http://www.wikidata.org/entity/Q33613335

about

TRIM65 triggers β-Catenin signaling via ubiquitination of Axin1 to promote hepatocellular carcinoma.IGF2 mRNA binding protein-2 is a tumor promoter that drives cancer proliferation through its client mRNAs IGF2 and HMGA1 Insulin Receptor Isoforms in Physiology and Disease: An Updated View.Adenovirus-Mediated Delivery of Decoy Hyper Binding Sites Targeting Oncogenic HMGA1 Reduces Pancreatic and Liver Cancer Cell Viability.HMGA1 exacerbates tumor growth through regulating the cell cycle and accelerates migration/invasion via targeting miR-221/222 in cervical cancer.Transcriptional Regulation of Glucose Metabolism: The Emerging Role of the HMGA1 Chromatin Factor Rapid and Integrative Discovery of Retina Regulatory Molecules Role of Pseudogenes in Tumorigenesis

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P2860

The High Mobility Group A1 (HMGA1) Transcriptome in Cancer and Development.

http://www.wikidata.org/entity/Q33613335

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2016 nî lūn-bûn

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2016 թուականի Մարտին հրատարակուած գիտական յօդուած

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2016 թվականի մարտին հրատարակված գիտական հոդված

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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Human Th1 cells that express CD300a are polyfunctional and after stimulation up-regulate the T-box transcription factor eomesodermin

From bending DNA to diabetes: the curious case of HMGA1

Cyr61/CCN1 signaling is critical for epithelial-mesenchymal transition and stemness and promotes pancreatic carcinogenesis

Negative Regulation of BRCA1 Gene Expression by HMGA1 Proteins Accounts for the Reduced BRCA1 Protein Levels in Sporadic Breast Carcinoma

Hallmarks of Cancer: The Next Generation

The AT-hook of the chromatin architectural transcription factor high mobility group A1a is arginine-methylated by protein arginine methyltransferase 6

A novel role for high-mobility group a proteins in cellular senescence and heterochromatin formation

Inducible nitric oxide synthase binds, S-nitrosylates, and activates cyclooxygenase-2

SRF and myocardin regulate LRP-mediated amyloid-beta clearance in brain vascular cells

GATA6 mutations cause human cardiac outflow tract defects by disrupting semaphorin-plexin signaling

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