Modulation of GABAergic transmission in development and neurodevelopmental disorders: investigating physiology and pathology to gain therapeutic perspectives.
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Brain-specific transcriptional regulator T-brain-1 controls brain wiring and neuronal activity in autism spectrum disordersMolecular aspects of age-related cognitive decline: the role of GABA signalingDevelopmental and adult expression patterns of the G-protein-coupled receptor GPR88 in the rat: Establishment of a dual nuclear-cytoplasmic localizationDNA methylation: a mechanism linking environmental chemical exposures to risk of autism spectrum disorders?A Role for Oxytocin in the Etiology and Treatment of Schizophrenia.Pharmacological approaches to improving cognitive function in Down syndrome: current status and considerations.Leptin potentiates GABAergic synaptic transmission in the developing rodent hippocampus.Twenty years of fluorescence imaging of intracellular chlorideGABAergic/glutamatergic imbalance relative to excessive neuroinflammation in autism spectrum disorders.Early depolarizing GABA controls critical-period plasticity in the rat visual cortex.Association between 5q23.2-located polymorphism of CTXN3 gene (Cortexin 3) and schizophrenia in European-Caucasian males; implications for the aetiology of schizophrenia.Regulatory domain or CpG site variation in SLC12A5, encoding the chloride transporter KCC2, in human autism and schizophreniaFine Mapping of a Dravet Syndrome Modifier Locus on Mouse Chromosome 5 and Candidate Gene Analysis by RNA-SeqA Chimera Na+-Pump Rhodopsin as an Effective Optogenetic Silencer.Kinase-KCC2 coupling: Cl- rheostasis, disease susceptibility, therapeutic target.During postnatal development endogenous neurosteroids influence GABA-ergic neurotransmission of mouse cortical neurons.The basolateral amygdala γ-aminobutyric acidergic system in health and disease.γ-Aminobutyric Acid Type A (GABAA) Receptor Subunits Play a Direct Structural Role in Synaptic Contact Formation via Their N-terminal Extracellular Domains.The GABAergic Hypothesis for Cognitive Disabilities in Down SyndromeCalretinin and parvalbumin in schizophrenia and affective disorders: a mini-review, a perspective on the evolutionary role of calretinin in schizophrenia, and a preliminary post-mortem study of calretinin in the septal nuclei.Altering the course of schizophrenia: progress and perspectives.Inhibitory neurotransmission regulates vagal efferent activity and gastric motility.Developmentally regulated neurosteroid synthesis enhances GABAergic neurotransmission in mouse thalamocortical neurones.Testing the excitation/inhibition imbalance hypothesis in a mouse model of the autism spectrum disorder: in vivo neurospectroscopy and molecular evidence for regional phenotypes.Bumetanide treatment during early development rescues maternal separation-induced susceptibility to stress.GABAA receptor activity shapes the formation of inhibitory synapses between developing medium spiny neurons.Simultaneous two-photon imaging of intracellular chloride concentration and pH in mouse pyramidal neurons in vivo.Neural Hyperexcitability in Autism Spectrum Disorders.The female epilepsy protein PCDH19 is a new GABAAR binding partner that regulates GABAergic transmission as well as migration and morphological maturation of hippocampal neurons.Decreasing the Expression of GABAA α5 Subunit-Containing Receptors Partially Improves Cognitive, Electrophysiological, and Morphological Hippocampal Defects in the Ts65Dn Model of Down Syndrome.Activation of the anti-inflammatory reflex blocks lipopolysaccharide-induced decrease in synaptic inhibition in the temporal cortex of the rat.Guanidinoacetic acid loading affects plasma γ-aminobutyric acid in healthy men.[Mutations in NONO lead to syndromic intellectual disability and inhibitory synaptic defects].Paradoxical effect of baclofen on social behavior in the fragile X syndrome mouse model.Chloride Homeostasis in Neurons With Special Emphasis on the Olivocerebellar System: Differential Roles for Transporters and Channels.Structure, Function, and Modulation of γ-Aminobutyric Acid Transporter 1 (GAT1) in Neurological Disorders: A Pharmacoinformatic Prospective
P2860
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P2860
Modulation of GABAergic transmission in development and neurodevelopmental disorders: investigating physiology and pathology to gain therapeutic perspectives.
description
2014 nî lūn-bûn
@nan
2014 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Modulation of GABAergic transm ...... gain therapeutic perspectives.
@ast
Modulation of GABAergic transm ...... gain therapeutic perspectives.
@en
type
label
Modulation of GABAergic transm ...... gain therapeutic perspectives.
@ast
Modulation of GABAergic transm ...... gain therapeutic perspectives.
@en
prefLabel
Modulation of GABAergic transm ...... gain therapeutic perspectives.
@ast
Modulation of GABAergic transm ...... gain therapeutic perspectives.
@en
P2860
P356
P1476
Modulation of GABAergic transm ...... gain therapeutic perspectives.
@en
P2093
Ignacio F Bozarth
P2860
P356
10.3389/FNCEL.2014.00119
P577
2014-05-22T00:00:00Z