Fibrosis of two: Epithelial cell-fibroblast interactions in pulmonary fibrosis - Test2 - elhamkhani - TriplyDB

Fibrosis of two: Epithelial cell-fibroblast interactions in pulmonary fibrosis

Fibrosis of two: Epithelial cell-fibroblast interactions in pulmonary fibrosis

http://www.wikidata.org/entity/Q33698776

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The Rho kinases: critical mediators of multiple profibrotic processes and rational targets for new therapies for pulmonary fibrosis Deficiency of α7 nicotinic acetylcholine receptor attenuates bleomycin-induced lung fibrosis in mice SDF-1 in Mammary Fibroblasts of Bovine with Mastitis Induces EMT and Inflammatory Response of Epithelial Cells Effect of P2X7 receptor knockout on AQP-5 expression of type I alveolar epithelial cells.Alveolar epithelium: beyond the barrier.Inhibition of CTGF ameliorates peritoneal fibrosis through suppression of fibroblast and myofibroblast accumulation and angiogenesis New therapeutic targets in idiopathic pulmonary fibrosis. Aiming to rein in runaway wound-healing responses.P190B RhoGAP overexpression in the developing mammary epithelium induces TGFβ-dependent fibroblast activation Heparan sulfate proteoglycans mediate factor XIIa binding to the cell surface Role of Areca Nut Induced TGF-β and Epithelial-Mesenchymal Interaction in the Pathogenesis of Oral Submucous Fibrosis.A Mathematical Model of Idiopathic Pulmonary Fibrosis.Pulmonary preconditioning, injury, and inflammation modulate expression of the candidate tumor suppressor gene ECRG4 in lung.miR-130b-3p Modulates Epithelial-Mesenchymal Crosstalk in Lung Fibrosis by Targeting IGF-1 MicroRNAs as potential targets for progressive pulmonary fibrosis Fibrogenic Lung Injury Induces Non-Cell-Autonomous Fibroblast Invasion miR-323a-3p regulates lung fibrosis by targeting multiple profibrotic pathways.A Systematic Review of the Role of Dysfunctional Wound Healing in the Pathogenesis and Treatment of Idiopathic Pulmonary Fibrosis.Future directions in idiopathic pulmonary fibrosis research. An NHLBI workshop report.The epithelium in idiopathic pulmonary fibrosis: breaking the barrier.Stem Cell-Based Therapy in Idiopathic Pulmonary Fibrosis.Inhibition of connective tissue growth factor attenuates paraquat-induced lung fibrosis in a human MRC-5 cell line.Lung Pericytes and Resident Fibroblasts: Busy Multitaskers.Immunoglobulin A in serum: an old acquaintance as a new prognostic biomarker in idiopathic pulmonary fibrosis.Plasma membrane wounding and repair in pulmonary diseases.The advancements of heparanase in fibrosis Curcumin Inhibits Transforming Growth Factor β Induced Differentiation of Mouse Lung Fibroblasts to Myofibroblasts.An immunofluorescence assay for extracellular matrix components highlights the role of epithelial cells in producing a stable, fibrillar extracellular matrix.Lysophosphatidic acid signaling through its receptor initiates profibrotic epithelial cell fibroblast communication mediated by epithelial cell derived connective tissue growth factor.Region-specific role for Pten in maintenance of epithelial phenotype and integrity.Adeno-Associated Virus 2 (AAV2)-Mediated Reintroduction of microRNA-19b Attenuates Hepatic Fibrosis.The Rho Kinase Isoforms ROCK1 and ROCK2 Each Contribute to the Development of Experimental Pulmonary Fibrosis.Fibroblasts in Hypoxic Conditions Mimic Laryngotracheal Stenosis.Effects of hypoxia and hyperoxia on the differential expression of VEGF-A isoforms and receptors in Idiopathic Pulmonary Fibrosis (IPF).FGF9 and FGF18 in idiopathic pulmonary fibrosis promote survival and migration and inhibit myofibroblast differentiation of human lung fibroblasts in vitro.Targeting metabolic abnormalities to reverse fibrosis in iatrogenic laryngotracheal stenosis.Differential Expression of VEGF-Axxx Isoforms Is Critical for Development of Pulmonary Fibrosis.Exposure to febrile-range hyperthermia potentiates Wnt signalling and epithelial-mesenchymal transition gene expression in lung epithelium.Tannic acid attenuates TGF-β1-induced epithelial-to-mesenchymal transition by effectively intervening TGF-β signaling in lung epithelial cells.Mitochondrial Dysfunction in Pulmonary Fibrosis.Modeling Idiopathic Pulmonary Fibrosis in Humanized SCID Mice.

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Fibrosis of two: Epithelial cell-fibroblast interactions in pulmonary fibrosis

http://www.wikidata.org/entity/Q33698776

description

2013 nî lūn-bûn

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2013 թուականի Մարտին հրատարակուած գիտական յօդուած

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2013 թվականի մարտին հրատարակված գիտական հոդված

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2013年の論文

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Fibrosis of two: Epithelial cell-fibroblast interactions in pulmonary fibrosis

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Fibrosis of two: Epithelial cell-fibroblast interactions in pulmonary fibrosis

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Fibrosis of two: Epithelial cell-fibroblast interactions in pulmonary fibrosis

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Fibrosis of two: Epithelial cell-fibroblast interactions in pulmonary fibrosis

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Fibrosis of two: Epithelial cell-fibroblast interactions in pulmonary fibrosis

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Fibrosis of two: Epithelial cell-fibroblast interactions in pulmonary fibrosis

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J.BBADIS.2013.03.001

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Biochimica et Biophysica Acta

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Fibrosis of two: Epithelial cell-fibroblast interactions in pulmonary fibrosis

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Andrew M Tager

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Norihiko Sakai

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Expression of the developmental Sonic hedgehog (Shh) signalling pathway is up-regulated in chronic lung fibrosis and the Shh receptor patched 1 is present in circulating T lymphocytes

Evidence from normal expression and targeted misexpression that bone morphogenetic protein (Bmp-4) plays a role in mouse embryonic lung morphogenesis

Transforming growth factor beta 1 null mutation in mice causes excessive inflammatory response and early death

A surfactant protein C precursor protein BRICHOS domain mutation causes endoplasmic reticulum stress, proteasome dysfunction, and caspase 3 activation

WNT1-inducible signaling protein-1 mediates pulmonary fibrosis in mice and is upregulated in humans with idiopathic pulmonary fibrosis

Angiotensin signalling in pulmonary fibrosis

Integrins: bidirectional, allosteric signaling machines

Integrins: versatility, modulation, and signaling in cell adhesion

Cyclooxygenase in biology and disease.

The integrin alpha v beta 6 binds and activates latent TGF beta 1: a mechanism for regulating pulmonary inflammation and fibrosis

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pulmonary fibrosis

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