The cystic fibrosis transmembrane conductance regulator (CFTR) is expressed in maturation stage ameloblasts, odontoblasts and bone cells - Test2 - elhamkhani - TriplyDB

The cystic fibrosis transmembrane conductance regulator (CFTR) is expressed in maturation stage ameloblasts, odontoblasts and bone cells

The cystic fibrosis transmembrane conductance regulator (CFTR) is expressed in maturation stage ameloblasts, odontoblasts and bone cells

http://www.wikidata.org/entity/Q33742138

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Anoctamin-6 controls bone mineralization by activating the calcium transporter NCX1 SLC26A Gene Family Participate in pH Regulation during Enamel Maturation Osteoblast CFTR inactivation reduces differentiation and osteoprotegerin expression in a mouse model of cystic fibrosis-related bone disease.Multiple Calcium Export Exchangers and Pumps Are a Prominent Feature of Enamel Organ Cells.Elevated incidence of dental caries in a mouse model of cystic fibrosis.Enamel pathology resulting from loss of function in the cystic fibrosis transmembrane conductance regulator in a porcine animal model The sodium bicarbonate cotransporter (NBCe1) is essential for normal development of mouse dentition.Functional interaction between CFTR and the sodium-phosphate co-transport type 2a in Xenopus laevis oocytes Genome-wide analysis of miRNA and mRNA transcriptomes during amelogenesis.NBCe1 in mouse and human ameloblasts may be indirectly regulated by fluoride.Composition of mineralizing incisor enamel in cystic fibrosis transmembrane conductance regulator-deficient mice.Identification of novel candidate genes involved in mineralization of dental enamel by genome-wide transcript profiling.Murine ameloblasts are immunonegative for Tcirg1, the v-H-ATPase subunit essential for the osteoclast plasma proton pump Requirements for ion and solute transport, and pH regulation during enamel maturation.Partial Restoration of CFTR Function in cftr-Null Mice following Targeted Cell Replacement Therapy.Slc26a3/Dra and Slc26a6 in Murine Ameloblasts.Developmental expression of solute carrier family 26A member 4 (SLC26A4/pendrin) during amelogenesis in developing rodent teeth Null mutation of chloride channel 7 (Clcn7) impairs dental root formation but does not affect enamel mineralization New paradigms on the transport functions of maturation-stage ameloblasts.Cystic Fibrosis Transmembrane Conductance Regulator in Sarcoplasmic Reticulum of Airway Smooth Muscle. Implications for Airway Contractility V-type ATPase proton pump expression during enamel formation.NBCe1 (SLC4A4) a potential pH regulator in enamel organ cells during enamel development in the mouse.Ion channels, channelopathies, and tooth formation.Importance of bicarbonate transport in pH control during amelogenesis - need for functional studies.Deletion of Slc26a1 and Slc26a7 Delays Enamel Mineralization in Mice.DENTAL ENAMEL FORMATION AND IMPLICATIONS FOR ORAL HEALTH AND DISEASE.Ion Transport by Ameloblasts during Amelogenesis.Enamel: Molecular identity of its transepithelial ion transport system.Buffering of protons released by mineral formation during amelogenesis in mice.Stim1 Regulates Enamel Mineralization and Ameloblast Modulation.Orai1 expression pattern in tooth and craniofacial ectodermal tissues and potential functions during ameloblast differentiation.Increased NF-κB Activity and Decreased Wnt/β-Catenin Signaling Mediate Reduced Osteoblast Differentiation and Function in ΔF508 Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Mice Novel role of cystic fibrosis transmembrane conductance regulator in maintaining adult mouse olfactory neuronal homeostasis.Bicarbonate Transport During Enamel Maturation.CFTR induces extracellular acid sensing in Xenopus oocytes which activates endogenous Ca²⁺-activated Cl⁻ conductance.Genetic deletion of keratin 8 corrects the altered bone formation and osteopenia in a mouse model of cystic fibrosis.Ion transporters in secretory and cyclically modulating ameloblasts: a new hypothesis for cellular control of preeruptive enamel maturation.The Importance of Connexin 43 in Enamel Development and Mineralization.How pH is regulated during amelogenesis in dental fluorosis

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The cystic fibrosis transmembrane conductance regulator (CFTR) is expressed in maturation stage ameloblasts, odontoblasts and bone cells

http://www.wikidata.org/entity/Q33742138

description

2009 nî lūn-bûn

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2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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The cystic fibrosis transmembr ...... s, odontoblasts and bone cells

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The cystic fibrosis transmembr ...... s, odontoblasts and bone cells

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The cystic fibrosis transmembr ...... s, odontoblasts and bone cells

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The cystic fibrosis transmembr ...... s, odontoblasts and bone cells

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J.BONE.2009.12.002

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The cystic fibrosis transmembr ...... s, odontoblasts and bone cells

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Antonius Bronckers

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Behrouz Zandieh-Doulabi

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Donacian M Lyaruu

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Hugo de Jonge

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Huub J N Jorna

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Lida Kalogeraki

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Martina Wilke

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Pamela Denbesten

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Theodore J Bervoets

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P2860

Structure of nucleotide-binding domain 1 of the cystic fibrosis transmembrane conductance regulator

The SLC26 gene family of multifunctional anion exchangers

Immunohistochemical localization of carbonic anhydrase isozyme II in rat incisor epithelial cells at various stages of amelogenesis

Subcellular localization of CFTR to endosomes in a ductal epithelium

Phosphorylation of the cystic fibrosis transmembrane conductance regulator

Dynamic control of cystic fibrosis transmembrane conductance regulator Cl(-)/HCO3(-) selectivity by external Cl(-)

Evidence by signal peptide trap technology for the expression of carbonic anhydrase 6 in rat incisor enamel organs.

Lack of CFTR in skeletal muscle predisposes to muscle wasting and diaphragm muscle pump failure in cystic fibrosis mice

Ameloblast modulation in the maturation zone of the rat incisor enamel organ. A light and electron microscopic study.

Na+:HCO(3-) cotransporters (NBC): cloning and characterization.

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1188-1196

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scholarly article

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10.1016/J.BONE.2009.12.002

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4

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cystic fibrosis

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