Increased NF-κB Activity and Decreased Wnt/β-Catenin Signaling Mediate Reduced Osteoblast Differentiation and Function in ΔF508 Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Mice
about
Defective CFTR- β-catenin interaction promotes NF-κB nuclear translocation and intestinal inflammation in cystic fibrosis.Chronic skin inflammation leads to bone loss by IL-17-mediated inhibition of Wnt signaling in osteoblasts.Excessive Activation of TLR4/NF-κB Interactively Suppresses the Canonical Wnt/β-catenin Pathway and Induces SANFH in SD Rats.Reduced bone length, growth plate thickness, bone content, and IGF-I as a model for poor growth in the CFTR-deficient rat.CFTR-β-catenin interaction regulates mouse embryonic stem cell differentiation and embryonic development.Genetic deletion of keratin 8 corrects the altered bone formation and osteopenia in a mouse model of cystic fibrosis.Dissection of the Role of VIMP in Endoplasmic Reticulum-Associated Degradation of CFTRΔF508.
P2860
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P2860
Increased NF-κB Activity and Decreased Wnt/β-Catenin Signaling Mediate Reduced Osteoblast Differentiation and Function in ΔF508 Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Mice
description
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name
Increased NF-κB Activity and D ...... ductance Regulator (CFTR) Mice
@en
type
label
Increased NF-κB Activity and D ...... ductance Regulator (CFTR) Mice
@en
prefLabel
Increased NF-κB Activity and D ...... ductance Regulator (CFTR) Mice
@en
P2093
P2860
P50
P921
P356
P1476
Increased NF-κB Activity and D ...... ductance Regulator (CFTR) Mice
@en
P2093
Carole Le Henaff
Caroline Marty
Emmanuel Laplantine
Nadine Tarantino
Pierre J Marie
Rafik Mansouri
P2860
P304
18009-18017
P356
10.1074/JBC.M115.646208
P407
P577
2015-06-09T00:00:00Z