Transactivation by the E2 protein of oncogenic human papillomavirus type 31 is not essential for early and late viral functions.
about
Bromodomain protein 4 mediates the papillomavirus E2 transcriptional activation functionMechanisms of virus immune evasion lead to development from chronic inflammation to cancer formation associated with human papillomavirus infectionThe Role of the DNA Damage Response throughout the Papillomavirus Life CycleInteraction of NCOR/SMRT Repressor Complexes with Papillomavirus E8^E2C Proteins Inhibits Viral ReplicationPapillomavirus genomes associate with BRD4 to replicate at fragile sites in the host genome.DNA replication of human papillomavirus type 31 is modulated by elements of the upstream regulatory region that lie 5' of the minimal originMechanisms of human papillomavirus E2-mediated repression of viral oncogene expression and cervical cancer cell growth inhibition.Differentiation-dependent chromatin rearrangement coincides with activation of human papillomavirus type 31 late gene expression.Cellular changes induced by low-risk human papillomavirus type 11 in keratinocytes that stably maintain viral episomesEarly polyadenylation signals of human papillomavirus type 31 negatively regulate capsid gene expression.Human papillomavirus type 16 E1circumflexE4 contributes to multiple facets of the papillomavirus life cycle.Nuclear export of human papillomavirus type 31 E1 is regulated by Cdk2 phosphorylation and required for viral genome maintenance.A transactivator function of cottontail rabbit papillomavirus e2 is essential for tumor induction in rabbits.Long-term effect of interferon on keratinocytes that maintain human papillomavirus type 31.Human papillomavirus type 16 E2 protein has no effect on transcription from episomal viral DNA.Human papillomavirus type 31 replication modes during the early phases of the viral life cycle depend on transcriptional and posttranscriptional regulation of E1 and E2 expression.Brd4 is displaced from HPV replication factories as they expand and amplify viral DNABrd4 is required for e2-mediated transcriptional activation but not genome partitioning of all papillomaviruses.Replication and partitioning of papillomavirus genomes.Development of quantitative and high-throughput assays of polyomavirus and papillomavirus DNA replicationRecent Insights into the Control of Human Papillomavirus (HPV) Genome Stability, Loss, and Degradation.Recent advances in the search for antiviral agents against human papillomaviruses.Human papillomavirus: gene expression, regulation and prospects for novel diagnostic methods and antiviral therapiesProduction of infectious bovine papillomavirus from cloned viral DNA by using an organotypic raft/xenograft techniqueCurrent understanding of the role of the Brd4 protein in the papillomavirus lifecycle.The papillomavirus E2 proteinsHuman Papillomavirus E2 Protein: Linking Replication, Transcription, and RNA Processing.Brd4 Activates Early Viral Transcription upon Human Papillomavirus 18 Infection of Primary Keratinocytes.Identification of the E9/E2C cDNA and functional characterization of the gene product reveal a new repressor of transcription and replication in cottontail rabbit papillomavirus.Inhibition of transcription and DNA replication by the papillomavirus E8-E2C protein is mediated by interaction with corepressor molecules.Evidence supporting a role for TopBP1 and Brd4 in the initiation but not continuation of human papillomavirus 16 E1/E2-mediated DNA replication.The E8E2C protein, a negative regulator of viral transcription and replication, is required for extrachromosomal maintenance of human papillomavirus type 31 in keratinocytes.The E8 domain confers a novel long-distance transcriptional repression activity on the E8E2C protein of high-risk human papillomavirus type 31.Human papillomavirus type 31b infection of human keratinocytes and the onset of early transcription.Human papillomavirus type 16 E2 protein transcriptionally activates the promoter of a key cellular splicing factor, SF2/ASF.Human Papillomavirus E2 Regulates SRSF3 (SRp20) To Promote Capsid Protein Expression in Infected Differentiated Keratinocytes.Modification of papillomavirus E2 proteins by the small ubiquitin-like modifier family members (SUMOs).Dimerization of the human papillomavirus type 16 E2 N terminus results in DNA looping within the upstream regulatory region.The papillomavirus E8-E2C protein represses DNA replication from extrachromosomal origins.The Deacetylase SIRT1 Regulates the Replication Properties of Human Papillomavirus 16 E1 and E2.
P2860
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P2860
Transactivation by the E2 protein of oncogenic human papillomavirus type 31 is not essential for early and late viral functions.
description
1998 nî lūn-bûn
@nan
1998 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Transactivation by the E2 prot ...... arly and late viral functions.
@ast
Transactivation by the E2 prot ...... arly and late viral functions.
@en
type
label
Transactivation by the E2 prot ...... arly and late viral functions.
@ast
Transactivation by the E2 prot ...... arly and late viral functions.
@en
prefLabel
Transactivation by the E2 prot ...... arly and late viral functions.
@ast
Transactivation by the E2 prot ...... arly and late viral functions.
@en
P2093
P2860
P1433
P1476
Transactivation by the E2 prot ...... arly and late viral functions.
@en
P2093
Colbert AM
Laimins LA
Stubenrauch F
P2860
P304
P577
1998-10-01T00:00:00Z