Cathepsin D regulates lipid metabolism in murine steatohepatitis. - Test2 - elhamkhani - TriplyDB

Cathepsin D regulates lipid metabolism in murine steatohepatitis.

Cathepsin D regulates lipid metabolism in murine steatohepatitis.

http://www.wikidata.org/entity/Q33799445

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Cathepsin D regulates lipid metabolism in murine steatohepatitis.

http://www.wikidata.org/entity/Q33799445

description

2017 nî lūn-bûn

@nan

2017 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2017 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2017年の論文

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2017年論文

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2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

name

Cathepsin D regulates lipid metabolism in murine steatohepatitis.

@ast

Cathepsin D regulates lipid metabolism in murine steatohepatitis.

@en

type

label

Cathepsin D regulates lipid metabolism in murine steatohepatitis.

@ast

Cathepsin D regulates lipid metabolism in murine steatohepatitis.

@en

prefLabel

Cathepsin D regulates lipid metabolism in murine steatohepatitis.

@ast

Cathepsin D regulates lipid metabolism in murine steatohepatitis.

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Scientific Reports

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Cathepsin D regulates lipid metabolism in murine steatohepatitis.

@en

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Albert V Bitorina

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Dieter Lütjohann

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Jogchum Plat

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Marion J J Gijbels

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Marten H Hofker

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Silvia Friedrichs

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Sofie M A Walenbergh

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Tim Hendrikx

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Yvonne Oligschlaeger

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P2860

From NAFLD in clinical practice to answers from guidelines

Lysosomal enzymes are released from cultured human macrophages, hydrolyze LDL in vitro, and are present extracellularly in human atherosclerotic lesions

Cathepsin D--many functions of one aspartic protease

TFEB links autophagy to lysosomal biogenesis

Small intestinal CD103+ dendritic cells display unique functional properties that are conserved between mice and humans.

Enzymatically active lysosomal proteases are associated with amyloid deposits in Alzheimer brain

Bile acids lower triglyceride levels via a pathway involving FXR, SHP, and SREBP-1c

LDL receptor knock-out mice are a physiological model particularly vulnerable to study the onset of inflammation in non-alcoholic fatty liver disease.

Internalization of modified lipids by CD36 and SR-A leads to hepatic inflammation and lysosomal cholesterol storage in Kupffer cells.

Human cholesterol 7alpha-hydroxylase (CYP7A1) deficiency has a hypercholesterolemic phenotype.

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scholarly article

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10.1038/S41598-017-03796-5

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1

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7

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Ronit Shiri-Sverdlov

Frank G. Schaap

Patrick J van Gorp

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2017-06-14T00:00:00Z

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1085943329

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28615690

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Steatohepatitis

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