A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.
about
Mechanisms of gene duplication and amplificationTelomere dysfunction and chromosome instabilityDNA end resection: many nucleases make light workReplication stalling at unstable inverted repeats: interplay between DNA hairpins and fork stabilizing proteinsNew approaches to the analysis of palindromic sequences from the human genome: evolution and polymorphism of an intronic site at the NF1 locusInverted repeat-stimulated sister-chromatid exchange events are RAD1-independent but reduced in a msh2 mutant.Replication protein A prevents promiscuous annealing between short sequence homologies: Implications for genome integritySae2 is an endonuclease that processes hairpin DNA cooperatively with the Mre11/Rad50/Xrs2 complex.Phosphorylation-regulated transitions in an oligomeric state control the activity of the Sae2 DNA repair enzyme.Minding the gap: the underground functions of BRCA1 and BRCA2 at stalled replication forksDNA repair pathway selection caused by defects in TEL1, SAE2, and de novo telomere addition generates specific chromosomal rearrangement signatures.Fusion of nearby inverted repeats by a replication-based mechanism leads to formation of dicentric and acentric chromosomes that cause genome instability in budding yeast.GAP-Seq: a method for identification of DNA palindromes.CtIP maintains stability at common fragile sites and inverted repeats by end resection-independent endonuclease activity.End resection at double-strand breaks: mechanism and regulation.The distribution of inverted repeat sequences in the Saccharomyces cerevisiae genome.The role of replication bypass pathways in dicentric chromosome formation in budding yeastRPA coordinates DNA end resection and prevents formation of DNA hairpins.The mitochondrial genome of the glomeromycete Rhizophagus sp. DAOM 213198 reveals an unusual organization consisting of two circular chromosomes.RPA antagonizes microhomology-mediated repair of DNA double-strand breaks.Replication fork integrity and intra-S phase checkpoint suppress gene amplification.Intrastrand annealing leads to the formation of a large DNA palindrome and determines the boundaries of genomic amplification in human cancerMre11-Sae2 and RPA Collaborate to Prevent Palindromic Gene AmplificationE. coli SbcCD and RecA control chromosomal rearrangement induced by an interrupted palindrome.DNA palindromes with a modest arm length of greater, similar 20 base pairs are a significant target for recombinant adeno-associated virus vector integration in the liver, muscles, and heart in mice.Multiple pathways of duplication formation with and without recombination (RecA) in Salmonella enterica.Palindromic amplification of the ERBB2 oncogene in primary HER2-positive breast tumorsGene copy-number variation in haploid and diploid strains of the yeast Saccharomyces cerevisiaeGlioblastoma multiforme: the role of DSB repair between genotype and phenotype.De novo-generated small palindromes are characteristic of amplicon boundary junction of double minutes.Mechanism and regulation of DNA end resection in eukaryotesXRCC2 and XRCC3 regulate the balance between short- and long-tract gene conversions between sister chromatids.Homology-mediated end-capping as a primary step of sister chromatid fusion in the breakage-fusion-bridge cycles.Chromosome aberrations resulting from double-strand DNA breaks at a naturally occurring yeast fragile site composed of inverted ty elements are independent of Mre11p and Sae2p.Mechanisms and regulation of DNA end resection.Making the best of the loose ends: Mre11/Rad50 complexes and Sae2 promote DNA double-strand break resection.Unprecedented large inverted repeats at the replication terminus of circular bacterial chromosomes suggest a novel mode of chromosome rescue.Differential regulation of short- and long-tract gene conversion between sister chromatids by Rad51C.Functions and regulation of the MRX complex at DNA double-strand breaks.Large inverted duplications in the human genome form via a fold-back mechanism.
P2860
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P2860
A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.
description
2005 nî lūn-bûn
@nan
2005 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.
@ast
A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.
@en
type
label
A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.
@ast
A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.
@en
prefLabel
A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.
@ast
A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.
@en
P2093
P2860
P356
P1433
P1476
A mechanism of palindromic gene amplification in Saccharomyces cerevisiae.
@en
P2093
Alison J Rattray
Beena Neelam
Brenda K Shafer
Jeffrey N Strathern
P2860
P304
P356
10.1101/GAD.1315805
P577
2005-06-01T00:00:00Z