Genetic suppression of β2-adrenergic receptors ameliorates tau pathology in a mouse model of tauopathies.
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G Protein-Coupled Receptors (GPCRs) in Alzheimer's Disease: A Focus on BACE1 Related GPCRsAdministration of a selective β2 adrenergic receptor antagonist exacerbates neuropathology and cognitive deficits in a mouse model of Alzheimer's diseaseAmyloid β production is regulated by β2-adrenergic signaling-mediated post-translational modifications of the ryanodine receptor.One is the deadliest number: the detrimental effects of social isolation on cerebrovascular diseases and cognition.Post-translational remodeling of ryanodine receptor induces calcium leak leading to Alzheimer's disease-like pathologies and cognitive deficits.Genetics of Frontotemporal Lobar Degeneration: From the Bench to the Clinic.Scalable Production of iPSC-Derived Human Neurons to Identify Tau-Lowering Compounds by High-Content Screening.Kinetics of Human Mutant Tau Prion Formation in the Brains of 2 Transgenic Mouse Lines.
P2860
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P2860
Genetic suppression of β2-adrenergic receptors ameliorates tau pathology in a mouse model of tauopathies.
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2014 nî lūn-bûn
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2014 թուականի Մարտին հրատարակուած գիտական յօդուած
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2014 թվականի մարտին հրատարակված գիտական հոդված
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2014年の論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年論文
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2014年论文
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name
Genetic suppression of β2-adre ...... a mouse model of tauopathies.
@ast
Genetic suppression of β2-adre ...... a mouse model of tauopathies.
@en
type
label
Genetic suppression of β2-adre ...... a mouse model of tauopathies.
@ast
Genetic suppression of β2-adre ...... a mouse model of tauopathies.
@en
prefLabel
Genetic suppression of β2-adre ...... a mouse model of tauopathies.
@ast
Genetic suppression of β2-adre ...... a mouse model of tauopathies.
@en
P2093
P2860
P356
P1476
Genetic suppression of β2-adre ...... a mouse model of tauopathies.
@en
P2093
Elena V Wisely
Salvatore Oddo
Yang K Xiang
P2860
P304
P356
10.1093/HMG/DDU116
P577
2014-03-13T00:00:00Z