Epigallocatechin-3 gallate prevents cardiac hypertrophy induced by pressure overload in rats - Test2 - elhamkhani - TriplyDB

Epigallocatechin-3 gallate prevents cardiac hypertrophy induced by pressure overload in rats

Epigallocatechin-3 gallate prevents cardiac hypertrophy induced by pressure overload in rats

http://www.wikidata.org/entity/Q33860907

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Insights into restrictive cardiomyopathy from clinical and animal studies.Polyphenols: benefits to the cardiovascular system in health and in aging Oxidative stress and cardiac hypertrophy: a review.Oxidative stress and cardiovascular health: therapeutic potential of polyphenols.Potential benefits of green tea polyphenol EGCG in the prevention and treatment of vascular inflammation in rheumatoid arthritis.Catechins and Its Role in Chronic Diseases.EGCG blocks TGFβ1-induced CCN2 by suppressing JNK and p38 in buccal fibroblasts.Cardioprotective Effect of Ulmus wallichiana Planchon in β-Adrenergic Agonist Induced Cardiac Hypertrophy Oral sophocarpine protects rat heart against pressure overload-induced cardiac fibrosis.Green tea polyphenols improve cardiac muscle mRNA and protein levels of signal pathways related to insulin and lipid metabolism and inflammation in insulin-resistant rats.(+)-Catechin in a 1:2 Complex with Lysine Inhibits Cancer Cell Migration and Metastatic Take in Mice.Phase 2 trial of daily, oral epigallocatechin gallate in patients with light-chain amyloidosis.Positive inotropic effects of epigallocatechin-3-gallate (EGCG) involve activation of Na+/H+ and Na+/Ca2+ exchangers.Green tea extract intake during lactation modified cardiac macrophage infiltration and AMP-activated protein kinase phosphorylation in weanling rats from undernourished mother during gestation and lactation.A Comparison of Phenomenologic Growth Laws for Myocardial Hypertrophy.Epigallocatechingallate attenuates myocardial injury in a mouse model of heart failure through TGF‑β1/Smad3 signaling pathway.Epigallocatechin-3-gallate improves cardiac hypertrophy and short-term memory deficits in a Williams-Beuren syndrome mouse model.(‑)‑Epicatechin protects against myocardial ischemia‑induced cardiac injury via activation of the PTEN/PI3K/AKT pathway.

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P2860

Epigallocatechin-3 gallate prevents cardiac hypertrophy induced by pressure overload in rats

http://www.wikidata.org/entity/Q33860907

description

2007 nî lūn-bûn

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2007 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի հունիսին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Epigallocatechin-3 gallate pre ...... d by pressure overload in rats

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Epigallocatechin-3 gallate pre ...... d by pressure overload in rats

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Epigallocatechin-3 gallate pre ...... d by pressure overload in rats

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JVS.2007.8.2.121

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Journal of Veterinary Science

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Epigallocatechin-3 gallate pre ...... d by pressure overload in rats

@en

P2093

Chan-Hyung Kim

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Hee-Yul Ahn

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Jia Hao

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Tae-Sun Ha

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P2860

On the part played by the suprarenals in the normal vascular reactions of the body

Mortality by cause for eight regions of the world: Global Burden of Disease Study

Mechanical stress-induced cardiac hypertrophy: mechanisms and signal transduction pathways.

Differential effects of green tea-derived catechin on developing versus established atherosclerosis in apolipoprotein E-null mice.

A constituent of green tea, epigallocatechin-3-gallate, activates endothelial nitric oxide synthase by a phosphatidylinositol-3-OH-kinase-, cAMP-dependent protein kinase-, and Akt-dependent pathway and leads to endothelial-dependent vasorelaxation.

Targeted deletion of apoptosis signal-regulating kinase 1 attenuates left ventricular remodeling.

Epigallocathechin-3 gallate selectively inhibits the PDGF-BB-induced intracellular signaling transduction pathway in vascular smooth muscle cells and inhibits transformation of sis-transfected NIH 3T3 fibroblasts and human glioblastoma cells (A172).

Mechanisms of hypertension in cardiac transplantation and the role of cyclosporine.

Cell type-specific angiotensin II-evoked signal transduction pathways: critical roles of Gbetagamma subunit, Src family, and Ras in cardiac fibroblasts.

Reactive oxygen species mediate amplitude-dependent hypertrophic and apoptotic responses to mechanical stretch in cardiac myocytes.

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121-129

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scholarly article

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10.4142/JVS.2007.8.2.121

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6312361

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17519564

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2872709

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