Contribution of pilus type 2b to invasive disease caused by a Streptococcus agalactiae ST-17 strain. - Test2 - elhamkhani - TriplyDB

Contribution of pilus type 2b to invasive disease caused by a Streptococcus agalactiae ST-17 strain.

Contribution of pilus type 2b to invasive disease caused by a Streptococcus agalactiae ST-17 strain.

http://www.wikidata.org/entity/Q33868597

about

Codevelopment of Microbiota and Innate Immunity and the Risk for Group B Streptococcal Disease.Group B Streptococcal Colonization, Molecular Characteristics, and Epidemiology.Development of experimental GBS vaccine for mucosal immunization.

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Contribution of pilus type 2b to invasive disease caused by a Streptococcus agalactiae ST-17 strain.

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description

2017 nî lūn-bûn

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2017 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2017 թվականի հուլիսին հրատարակված գիտական հոդված

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年論文

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2017年论文

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name

Contribution of pilus type 2b ...... occus agalactiae ST-17 strain.

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Contribution of pilus type 2b ...... occus agalactiae ST-17 strain.

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type

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Contribution of pilus type 2b ...... occus agalactiae ST-17 strain.

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Contribution of pilus type 2b ...... occus agalactiae ST-17 strain.

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Contribution of pilus type 2b ...... occus agalactiae ST-17 strain.

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Contribution of pilus type 2b ...... occus agalactiae ST-17 strain.

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Contribution of pilus type 2b ...... coccus agalactiae ST-17 strain

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C Daniela Rinaudo

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Claudia Ghezzo

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Kelly S Doran

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Maddalena Lazzarin

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Monica Fabbrini

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P2860

Identification of a group B streptococcal fibronectin binding protein, SfbA, that contributes to invasion of brain endothelium and development of meningitis.

Pilus distribution among lineages of group b streptococcus: an evolutionary and clinical perspective

Group B streptococcal disease in infants aged younger than 3 months: systematic review and meta-analysis.

The spectrum of perinatal group B streptococcal disease.

Identification of novel genomic islands coding for antigenic pilus-like structures in Streptococcus agalactiae.

Subtractive hybridization identifies a novel predicted protein mediating epithelial cell invasion by virulent serotype III group B Streptococcus agalactiae.

Bacterial Pili exploit integrin machinery to promote immune activation and efficient blood-brain barrier penetration.

Phylogenetic lineage and pilus protein Spb1/SAN1518 affect opsonin-independent phagocytosis and intracellular survival of Group B Streptococcus.

Regulation and function of pilus island 1 in group B streptococcus

Streptococcus agalactiae clones infecting humans were selected and fixed through the extensive use of tetracycline.

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Immaculada Margarit

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