Bacterial Pili exploit integrin machinery to promote immune activation and efficient blood-brain barrier penetration. - Test2 - elhamkhani - TriplyDB

Bacterial Pili exploit integrin machinery to promote immune activation and efficient blood-brain barrier penetration.

Bacterial Pili exploit integrin machinery to promote immune activation and efficient blood-brain barrier penetration.

http://www.wikidata.org/entity/Q35366734

about

Defense at the border: the blood-brain barrier versus bacterial foreigners Pathogens penetrating the central nervous system: infection pathways and the cellular and molecular mechanisms of invasion Structure ofStreptococcus agalactiaetip pilin GBS104: a model for GBS pili assembly and host interactions Host-pathogen interactions in bacterial meningitis Gram-Positive Uropathogens, Polymicrobial Urinary Tract Infection, and the Emerging Microbiota of the Urinary Tract Entamoeba histolytica-Encoded Homolog of Macrophage Migration Inhibitory Factor Contributes to Mucosal Inflammation during Amebic Colitis.Identification of a group B streptococcal fibronectin binding protein, SfbA, that contributes to invasion of brain endothelium and development of meningitis.Contribution of pilus type 2b to invasive disease caused by a Streptococcus agalactiae ST-17 strain.Group B Streptococcus interactions with human meningeal cells and astrocytes in vitro.Group B Streptococcus hijacks the host plasminogen system to promote brain endothelial cell invasion The role of autophagy during group B Streptococcus infection of blood-brain barrier endothelium Streptococcus agalactiae infection in zebrafish larvae Structure and assembly of group B streptococcus pilus 2b backbone protein.Control of methicillin resistant Staphylococcus aureus infection utilizing a novel immunostimulatory peptide.Serine-rich repeat proteins and pili promote Streptococcus agalactiae colonization of the vaginal tract.Serine/threonine phosphatase Stp1 mediates post-transcriptional regulation of hemolysin, autolysis, and virulence of group B Streptococcus.Group B streptococcal infection and activation of human astrocytes.A Serine-Threonine Kinase (StkP) Regulates Expression of the Pneumococcal Pilus and Modulates Bacterial Adherence to Human Epithelial and Endothelial Cells In Vitro.Bacterial induction of Snail1 contributes to blood-brain barrier disruption.Identification of CiaR Regulated Genes That Promote Group B Streptococcal Virulence and Interaction with Brain Endothelial Cells Characterization of host immunity during persistent vaginal colonization by Group B Streptococcus.The metal ion-dependent adhesion site motif of the Enterococcus faecalis EbpA pilin mediates pilus function in catheter-associated urinary tract infection.Group B Streptococcus CovR regulation modulates host immune signalling pathways to promote vaginal colonization Concepts and mechanisms: crossing host barriers.Pilin and sortase residues critical for endocarditis- and biofilm-associated pilus biogenesis in Enterococcus faecalis Collagen-binding proteins of Streptococcus mutans and related streptococci.Increased Pilus Production Conferred by a Naturally Occurring Mutation Alters Host-Pathogen Interaction in Favor of Carriage in Streptococcus pyogenes.Perinatal Group B Streptococcal Infections: Virulence Factors, Immunity, and Prevention Strategies.The ancillary protein 1 of Streptococcus pyogenes FCT-1 pili mediates cell adhesion and biofilm formation through heterophilic as well as homophilic interactions.Quantitative assessment of the blood-brain barrier opening caused by Streptococcus agalactiae hyaluronidase in a BALB/c mouse model.Community-acquired bacterial meningitis.Modeling Group B Streptococcus and Blood-Brain Barrier Interaction by Using Induced Pluripotent Stem Cell-Derived Brain Endothelial Cells.Binding of Fibronectin to SsPepO Facilitates the Development of Streptococcus suis Meningitis.Characteristic Comparison of Meningitis and Non-meningitis of Streptococcus suis in an Experimentally Infected Porcine Model.Group B Streptococcal Maternal Colonization and Neonatal Disease: Molecular Mechanisms and Preventative Approaches.Characterization of a two-component system transcriptional regulator LtdR that impacts Group B Streptococcal colonization and disease.

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P2860

Bacterial Pili exploit integrin machinery to promote immune activation and efficient blood-brain barrier penetration.

http://www.wikidata.org/entity/Q35366734

description

2011 nî lūn-bûn

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2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Bacterial Pili exploit integri ...... ood-brain barrier penetration.

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Bacterial Pili exploit integri ...... ood-brain barrier penetration.

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Bacterial Pili exploit integri ...... ood-brain barrier penetration.

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Bacterial Pili exploit integri ...... ood-brain barrier penetration.

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Bacterial Pili exploit integri ...... ood-brain barrier penetration.

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Nature Communications

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Bacterial Pili exploit integri ...... ood-brain barrier penetration.

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Andrew S Cutting

http://www.w3.org/2001/XMLSchema#string

Anirban Banerjee

http://www.w3.org/2001/XMLSchema#string

Brandon J Kim

http://www.w3.org/2001/XMLSchema#string

Chris Carlos

http://www.w3.org/2001/XMLSchema#string

Ellese M Carmona

http://www.w3.org/2001/XMLSchema#string

Kelly S Doran

http://www.w3.org/2001/XMLSchema#string

Michael A Gurney

http://www.w3.org/2001/XMLSchema#string

Nemani V Prasadarao

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Ralph Feuer

http://www.w3.org/2001/XMLSchema#string

P2860

Group A Streptococcus produce pilus-like structures containing protective antigens and Lancefield T antigens.

A pneumococcal pilus influences virulence and host inflammatory responses

Corynebacterium diphtheriae employs specific minor pilins to target human pharyngeal epithelial cells

Sortase A utilizes an ancillary protein anchor for efficient cell wall anchoring of pili in Streptococcus agalactiae

Helicobacter pylori usurps cell polarity to turn the cell surface into a replicative niche

Novel fold and assembly of the repetitive B region of the Staphylococcus aureus collagen-binding surface protein

Structural basis of host cell recognition by the pilus adhesin from Streptococcus pneumoniae

Integrins: cell adhesives and modulators of cell function

Pneumococcal pili are composed of protofilaments exposing adhesive clusters of Rrg A

Focal adhesion kinase: the first ten years

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462

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scholarly article

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10.1038/NCOMMS1474

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P478

2

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2011-09-06T00:00:00Z

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51624676

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1026676652

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21897373

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blood–brain barrier

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3195231

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