Multiple Rad5 activities mediate sister chromatid recombination to bypass DNA damage at stalled replication forks. - Test2 - elhamkhani - TriplyDB

Multiple Rad5 activities mediate sister chromatid recombination to bypass DNA damage at stalled replication forks.

Multiple Rad5 activities mediate sister chromatid recombination to bypass DNA damage at stalled replication forks.

http://www.wikidata.org/entity/Q33921111

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Unwinding and rewinding: double faces of helicase?The Role of PCNA Posttranslational Modifications in Translesion Synthesis Replication-Associated Recombinational Repair: Lessons from Budding Yeast DNA damage tolerance by recombination: Molecular pathways and DNA structures Rescuing stalled or damaged replication forks DNA bending facilitates the error-free DNA damage tolerance pathway and upholds genome integrity.Length-dependent processing of telomeres in the absence of telomerase.Rad5 template switch pathway of DNA damage tolerance determines synergism between cisplatin and NSC109268 in Saccharomyces cerevisiae DNA repair mechanisms and the bypass of DNA damage in Saccharomyces cerevisiae A structure-function analysis of the yeast Elg1 protein reveals the importance of PCNA unloading in genome stability maintenance Both RAD5-dependent and independent pathways are involved in DNA damage-associated sister chromatid exchange in budding yeast.NuA4 initiates dynamic histone H4 acetylation to promote high-fidelity sister chromatid recombination at postreplication gaps.Chronic treatment with cisplatin induces replication-dependent sister chromatid recombination to confer cisplatin-resistant phenotype in nasopharyngeal carcinoma.Visualization of recombination-mediated damage bypass by template switching.In vivo bypass of 8-oxodG.Restriction of replication fork regression activities by a conserved SMC complex.Replication fork stalling by bulky DNA damage: localization at active origins and checkpoint modulation Branching out with DNA helicases.Concerted and differential actions of two enzymatic domains underlie Rad5 contributions to DNA damage tolerance.Repeat instability during DNA repair: Insights from model systems HLTF's Ancient HIRAN Domain Binds 3' DNA Ends to Drive Replication Fork Reversal.Mechanism of DNA damage tolerance.Rad5-dependent DNA repair functions of the Saccharomyces cerevisiae FANCM protein homolog Mph1.A Novel Rrm3 Function in Restricting DNA Replication via an Orc5-Binding Domain Is Genetically Separable from Rrm3 Function as an ATPase/Helicase in Facilitating Fork Progression.Impaired replication elongation in Tetrahymena mutants deficient in histone H3 Lys 27 monomethylation.Oxidative damage and mutagenesis in Saccharomyces cerevisiae: genetic studies of pathways affecting replication fidelity of 8-oxoguanine Replicating damaged DNA in eukaryotes.Esc2 promotes Mus81 complex-activity via its SUMO-like and DNA binding domains.Regulation of Replication Fork Advance and Stability by Nucleosome Assembly.Mechanisms of Post-Replication DNA Repair Monoubiquitylation of histone H2B contributes to the bypass of DNA damage during and after DNA replication Ubiquitin family modifications and template switching.Impediments to replication fork movement: stabilisation, reactivation and genome instability.Replication fork regression and its regulation.Homologous recombination maintenance of genome integrity during DNA damage tolerance.Role of recombination and replication fork restart in repeat instability.Premature Cdk1/Cdc5/Mus81 pathway activation induces aberrant replication and deleterious crossover Rad51 replication fork recruitment is required for DNA damage tolerance.Expansion of Interstitial Telomeric Sequences in Yeast.Irc3 is a mitochondrial DNA branch migration enzyme

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Multiple Rad5 activities mediate sister chromatid recombination to bypass DNA damage at stalled replication forks.

http://www.wikidata.org/entity/Q33921111

description

2010 nî lūn-bûn

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2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

@wuu

name

Multiple Rad5 activities media ...... at stalled replication forks.

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Multiple Rad5 activities media ...... at stalled replication forks.

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type

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Multiple Rad5 activities media ...... at stalled replication forks.

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Multiple Rad5 activities media ...... at stalled replication forks.

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Multiple Rad5 activities media ...... at stalled replication forks.

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Multiple Rad5 activities media ...... at stalled replication forks.

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J.MOLCEL.2010.03.020

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Multiple Rad5 activities media ...... at stalled replication forks.

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David Kowalski

http://www.w3.org/2001/XMLSchema#string

Eugen C Minca

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P2860

Human SHPRH is a ubiquitin ligase for Mms2-Ubc13-dependent polyubiquitylation of proliferating cell nuclear antigen

Functional uncoupling of MCM helicase and DNA polymerase activities activates the ATR-dependent checkpoint.

Polyubiquitination of proliferating cell nuclear antigen by HLTF and SHPRH prevents genomic instability from stalled replication forks

Eukaryotic translesion polymerases and their roles and regulation in DNA damage tolerance

The RING finger ATPase Rad5p of Saccharomyces cerevisiae contributes to DNA double-strand break repair in a ubiquitin-independent manner

Holliday junctions accumulate in replication mutants via a RecA homolog-independent mechanism.

Activation of ubiquitin-dependent DNA damage bypass is mediated by replication protein a.

Yeast Rad5 protein required for postreplication repair has a DNA helicase activity specific for replication fork regression.

The Saccharomyces cerevisiae RAD6 group is composed of an error-prone and two error-free postreplication repair pathways

RAD6-dependent DNA repair is linked to modification of PCNA by ubiquitin and SUMO.

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649-661

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scholarly article

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10.1016/J.MOLCEL.2010.03.020

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