Genomic prevalence of heterochromatic H3K9me2 and transcription do not discriminate pluripotent from terminally differentiated cells. - Test2 - elhamkhani - TriplyDB

Genomic prevalence of heterochromatic H3K9me2 and transcription do not discriminate pluripotent from terminally differentiated cells.

Genomic prevalence of heterochromatic H3K9me2 and transcription do not discriminate pluripotent from terminally differentiated cells.

http://www.wikidata.org/entity/Q33926822

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Histones and histone modifications in perinuclear chromatin anchoring: from yeast to man Chromatin states and nuclear organization in development--a view from the nuclear lamina Higher order chromatin organization in cancer Enhancer repertoires are reshaped independently of early priming and heterochromatin dynamics during B cell differentiation Spatial partitioning of the regulatory landscape of the X-inactivation centre Cytomegalovirus latency and reactivation: recent insights into an age old problem.Revisiting heterochromatin in embryonic stem cells Large-scale functional organization of long-range chromatin interaction networks Genome-wide chromatin state transitions associated with developmental and environmental cues.Chromatin modifiers and remodellers: regulators of cellular differentiation Epigenetic dysregulation by nickel through repressive chromatin domain disruption.Euchromatin islands in large heterochromatin domains are enriched for CTCF binding and differentially DNA-methylated regions.Transcription factors that convert adult cell identity are differentially polycomb repressed Recognition of H3K9 methylation by GLP is required for efficient establishment of H3K9 methylation, rapid target gene repression, and mouse viability.Chromatin landscapes of retroviral and transposon integration profiles The epigenome in early vertebrate development.Target genes of Topoisomerase IIβ regulate neuronal survival and are defined by their chromatin state.Transient bursts of Zscan4 expression are accompanied by the rapid derepression of heterochromatin in mouse embryonic stem cells Computational inference of mRNA stability from histone modification and transcriptome profiles SPOC1 modulates DNA repair by regulating key determinants of chromatin compaction and DNA damage response.Chromatin dynamics and the role of G9a in gene regulation and enhancer silencing during early mouse development.Histone H3K9 methyltransferase G9a represses PPARγ expression and adipogenesis.G9a/GLP Complex Maintains Imprinted DNA Methylation in Embryonic Stem Cells Widespread and extensive lengthening of 3' UTRs in the mammalian brain.The KRAB-ZFP/KAP1 system contributes to the early embryonic establishment of site-specific DNA methylation patterns maintained during development.Polycomb associates genome-wide with a specific RNA polymerase II variant, and regulates metabolic genes in ESCs.Non-polyadenylated transcription in embryonic stem cells reveals novel non-coding RNA related to pluripotency and differentiation Intragenic epigenetic changes modulate NCAM alternative splicing in neuronal differentiation.Epigenetic regulation of miR-302 by JMJD1C inhibits neural differentiation of human embryonic stem cells.The methyltransferase G9a regulates HoxA9-dependent transcription in AML.Jmjd2c facilitates the assembly of essential enhancer-protein complexes at the onset of embryonic stem cell differentiation.Reprogramming chromatin.Current trends in epigenetic drug discovery.Acetylation of histone H3 at lysine 64 regulates nucleosome dynamics and facilitates transcription.Regulation of cell differentiation and function by the euchromatin histone methyltranserfases G9a and GLP.H3K9me3-Dependent Heterochromatin: Barrier to Cell Fate Changes.RNA Binding to CBP Stimulates Histone Acetylation and Transcription.The expanding role of the Ehmt2/G9a complex in neurodevelopment.p63 Transcription Factor Regulates Nuclear Shape and Expression of Nuclear Envelope-Associated Genes in Epidermal Keratinocytes The chromatin remodelling factor ATRX suppresses R-loops in transcribed telomeric repeats.

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Genomic prevalence of heterochromatic H3K9me2 and transcription do not discriminate pluripotent from terminally differentiated cells.

http://www.wikidata.org/entity/Q33926822

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2011 nî lūn-bûn

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Genomic prevalence of heteroch ...... rminally differentiated cells.

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Genomic prevalence of heteroch ...... rminally differentiated cells.

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Genomic prevalence of heteroch ...... rminally differentiated cells.

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Dimos Gaidatzis

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Dirk Schübeler

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Fabio Mohn

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Florian Lienert

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Michael B Stadler

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Tim C Roloff

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Tuncay Baubec

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Vijay K Tiwari

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P2860

Identification and analysis of functional elements in 1% of the human genome by the ENCODE pilot project

Most "dark matter" transcripts are associated with known genes

Model-based analysis of ChIP-Seq (MACS)

Genome-wide analysis of KAP1 binding suggests autoregulation of KRAB-ZNFs

Genome-scale DNA methylation maps of pluripotent and differentiated cells

Genome-wide maps of chromatin state in pluripotent and lineage-committed cells

Human DNA methylomes at base resolution show widespread epigenomic differences

The landscape of histone modifications across 1% of the human genome in five human cell lines

Linear models and empirical bayes methods for assessing differential expression in microarray experiments

Induction of pluripotent stem cells from mouse embryonic and adult fibroblast cultures by defined factors

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