ChePep controls Helicobacter pylori Infection of the gastric glands and chemotaxis in the Epsilonproteobacteria.
about
Helicobacter pylori infection: An overview of bacterial virulence factors and pathogenesisHelicobacter and salmonella persistent infection strategiesChemodetection and Destruction of Host Urea Allows Helicobacter pylori to Locate the Epithelium.Motility and chemotaxis mediate the preferential colonization of gastric injury sites by Helicobacter pyloriMultiple peptidoglycan modification networks modulate Helicobacter pylori's cell shape, motility, and colonization potentialHelicobacter pylori CheZ(HP) and ChePep form a novel chemotaxis-regulatory complex distinct from the core chemotaxis signaling proteins and the flagellar motor.The 3.2 Å Resolution Structure of a Receptor:CheA:CheW Signaling Complex Defines Overlapping Binding Sites and Key Residue Interactions within Bacterial Chemosensory ArraysStructure and proposed mechanism for the pH-sensing Helicobacter pylori chemoreceptor TlpBDynamics of Lewis b binding and sequence variation of the babA adhesin gene during chronic Helicobacter pylori infection in humansHelicobacter pylori strains vary cell shape and flagellum number to maintain robust motility in viscous environmentsHelical and rod-shaped bacteria swim in helical trajectories with little additional propulsion from helical shape.Pathobiology of Helicobacter pylori-Induced Gastric Cancer.Signal balancing by the CetABC and CetZ chemoreceptors controls energy taxis in Campylobacter jejuni.Comparative genomic analysis of East Asian and non-Asian Helicobacter pylori strains identifies rapidly evolving genes.In vitro expansion of human gastric epithelial stem cells and their responses to bacterial infectionSurvival in hostile territory: the microbiota of the stomach.A gastric pathogen moves chemotaxis in a new direction.Bacterial chemotaxis modulates host cell apoptosis to establish a T-helper cell, type 17 (Th17)-dominant immune response in Helicobacter pylori infection.Quantitative Imaging of Gut Microbiota Spatial Organization.Chemorepulsion from the Quorum Signal Autoinducer-2 Promotes Helicobacter pylori Biofilm Dispersal.Systems-wide analyses of mucosal immune responses to Helicobacter pylori at the interface between pathogenicity and symbiosisRetraction of enteropathogenic E. coli type IV pili promotes efficient host cell colonization, effector translocation and tight junction disruptionMultiple Acid Sensors Control Helicobacter pylori Colonization of the StomachA supplemented soft agar chemotaxis assay demonstrates the Helicobacter pylori chemotactic response to zinc and nickelStaying in Shape: the Impact of Cell Shape on Bacterial Survival in Diverse EnvironmentsLife in the human stomach: persistence strategies of the bacterial pathogen Helicobacter pylori.The Helicobacter pylori CZB Cytoplasmic Chemoreceptor TlpD Forms an Autonomous Polar Chemotaxis Signaling Complex That Mediates a Tactic Response to Oxidative Stress.Spatial and Temporal Shifts in Bacterial Biogeography and Gland Occupation during the Development of a Chronic Infection.How Helicobacter pylori senses, targets and interacts with the gastric epithelium.A Conserved Helicobacter pylori Gene, HP0102, Is Induced Upon Contact With Gastric Cells and Has Multiple Roles in Pathogenicity.Phosphorylation-dependent and Phosphorylation-independent Regulation of Helicobacter pylori Acid Acclimation by the ArsRS Two-component System.Recent Advances in Helicobacter pylori Replication: Possible Implications in Adaptation to a Pathogenic Lifestyle and Perspectives for Drug Design.Role of Flagella in the Pathogenesis of Helicobacter pyloriLocalisation and protein-protein interactions of the Helicobacter pylori taxis sensor TlpD and their connection to metabolic functionsImaging the motility and chemotaxis machineries in Helicobacter pylori by cryo-electron tomography.Colonization, localization, and inflammation: the roles of H. pylori chemotaxis in vivo.Campylobacter jejuni transducer like proteins: Chemotaxis and beyond.Chemotaxis allows bacteria to overcome host-generated reactive oxygen species that constrain gland colonization.Helicobacter pylori Biofilm Formation and Its Potential Role in Pathogenesis.Influence of the viscosity of healthy and diseased human mucins on the motility of Helicobacter pylori.
P2860
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P2860
ChePep controls Helicobacter pylori Infection of the gastric glands and chemotaxis in the Epsilonproteobacteria.
description
2011 nî lūn-bûn
@nan
2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
ChePep controls Helicobacter p ...... in the Epsilonproteobacteria.
@ast
ChePep controls Helicobacter p ...... in the Epsilonproteobacteria.
@en
type
label
ChePep controls Helicobacter p ...... in the Epsilonproteobacteria.
@ast
ChePep controls Helicobacter p ...... in the Epsilonproteobacteria.
@en
prefLabel
ChePep controls Helicobacter p ...... in the Epsilonproteobacteria.
@ast
ChePep controls Helicobacter p ...... in the Epsilonproteobacteria.
@en
P2093
P2860
P356
P1433
P1476
ChePep controls Helicobacter p ...... in the Epsilonproteobacteria.
@en
P2093
Josephine Y Lee
Karen M Ottemann
Manuel R Amieva
Michael R Howitt
Roger Vogelmann
P2860
P356
10.1128/MBIO.00098-11
P577
2011-07-26T00:00:00Z