Relationship between tau pathology and neuroinflammation in Alzheimer's disease. - Test2 - elhamkhani - TriplyDB

Relationship between tau pathology and neuroinflammation in Alzheimer's disease.

Relationship between tau pathology and neuroinflammation in Alzheimer's disease.

http://www.wikidata.org/entity/Q33995490

about

Unified theory of Alzheimer's disease (UTAD): implications for prevention and curative therapy Neuroinflammation and Depression: Microglia Activation, Extracellular Microvesicles and microRNA Dysregulation Air pollution & the brain: Subchronic diesel exhaust exposure causes neuroinflammation and elevates early markers of neurodegenerative disease Protection of TGF-β1 against neuroinflammation and neurodegeneration in Aβ1-42-induced Alzheimer's disease model rats.Rheumatoid arthritis-associated polymorphisms are not protective against Alzheimer's disease.Amyloid-β oligomers unveil a novel primate model of sporadic Alzheimer's disease.OPTIMIZING DIAGNOSIS AND MANANGEMENT IN MILD-TO-MODERATE ALZHEIMER'S DISEASE.Tau mediated neurodegeneration: an insight into Alzheimer's disease pathology.3-(2,4-dimethoxybenzylidene)-anabaseine: a promising candidate drug for Alzheimer's disease?Telomere shortening and Alzheimer's disease.Neuroinflammation in Alzheimer's disease: an understanding of physiology and pathology.Tau in Late-Life Depression: A Systematic Review and Meta-Analysis.Protein Phosphorylation is a Key Mechanism in Alzheimer's Disease.Involvement of tau phosphorylation in traumatic brain injury patients.Insulin sensitizers improve learning and attenuate tau hyperphosphorylation and neuroinflammation in 3xTg-AD mice.Tau hyperphosphorylation is associated with spatial learning and memory after exposure to benzo[a]pyrene in SD rats.Linagliptin, a Dipeptidyl Peptidase-4 Inhibitor, Mitigates Cognitive Deficits and Pathology in the 3xTg-AD Mouse Model of Alzheimer's Disease.Asiaticoside Attenuates Cell Growth Inhibition and Apoptosis Induced by Aβ1-42 via Inhibiting the TLR4/NF-κB Signaling Pathway in Human Brain Microvascular Endothelial Cells.Two novel blood-based biomarker candidates measuring degradation of tau are associated with dementia: A prospective study.Novel Key Players in the Development of Tau Neuropathology: Focus on the 5-Lipoxygenase.Chia Seed Does Not Improve Cognitive Impairment in SAMP8 Mice Fed with High Fat Diet

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Relationship between tau pathology and neuroinflammation in Alzheimer's disease.

http://www.wikidata.org/entity/Q33995490

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2010 nî lūn-bûn

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2010 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2010 թվականի հունվարին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Relationship between tau pathology and neuroinflammation in Alzheimer's disease.

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Relationship between tau pathology and neuroinflammation in Alzheimer's disease.

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Relationship between tau pathology and neuroinflammation in Alzheimer's disease.

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Relationship between tau pathology and neuroinflammation in Alzheimer's disease.

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Relationship between tau pathology and neuroinflammation in Alzheimer's disease.

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Relationship between tau pathology and neuroinflammation in Alzheimer's disease.

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Mount Sinai Journal of Medicine

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Relationship between tau pathology and neuroinflammation in Alzheimer's disease.

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Maria E Figueiredo-Pereira

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Maria Jose Metcalfe

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P2860

The prolyl isomerase Pin1 restores the function of Alzheimer-associated phosphorylated tau protein

Sequestosome 1/p62 shuttles polyubiquitinated tau for proteasomal degradation

Caspase cleavage of tau: linking amyloid and neurofibrillary tangles in Alzheimer's disease

Regulated phosphorylation and dephosphorylation of tau protein: effects on microtubule interaction, intracellular trafficking and neurodegeneration

Akt and CHIP coregulate tau degradation through coordinated interactions

Tau fragmentation, aggregation and clearance: the dual role of lysosomal processing

Synapse loss and microglial activation precede tangles in a P301S tauopathy mouse model

CHIP-Hsc70 complex ubiquitinates phosphorylated tau and enhances cell survival

Frameshift mutants of beta amyloid precursor protein and ubiquitin-B in Alzheimer's and Down patients

Phosphorylation of tau at both Thr 231 and Ser 262 is required for maximal inhibition of its binding to microtubules

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50-58

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10.1002/MSJ.20163

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1

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77

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41140019

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20101714

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Alzheimer's disease

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