Aryl hydrocarbon receptor activation reduces dendritic cell function during influenza virus infection. - Test2 - elhamkhani - TriplyDB

Aryl hydrocarbon receptor activation reduces dendritic cell function during influenza virus infection.

Aryl hydrocarbon receptor activation reduces dendritic cell function during influenza virus infection.

http://www.wikidata.org/entity/Q34000825

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Association between Environmental Dioxin-Related Toxicants Exposure and Adverse Pregnancy Outcome: Systematic Review and Meta-Analysis New insights into the aryl hydrocarbon receptor as a modulator of host responses to infection Autoimmunity and infection in Sjögren's syndrome Toward understanding the role of aryl hydrocarbon receptor in the immune system: current progress and future trends Aryl hydrocarbon receptor (AhR) regulates silica-induced inflammation but not fibrosis New insights into the role of the aryl hydrocarbon receptor in the function of CD11c⁺ cells during respiratory viral infection Developmental exposure to bisphenol A modulates innate but not adaptive immune responses to influenza A virus infection Consequences of AhR activation in steady-state dendritic cells.AhR deficiency impairs expression of LPS-induced inflammatory genes in mice Mechanistic exploration of AhR-mediated host protection against Streptococcus pneumoniae infection.The influence of 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD) on hematological parameters during experimentally induced pleuritis in rats.Balancing intestinal and systemic inflammation through cell type-specific expression of the aryl hydrocarbon receptor repressor Role of the aryl hydrocarbon receptor in carcinogenesis and potential as a drug target.Aryl hydrocarbon receptor signaling regulates NF-κB RelB activation during dendritic-cell differentiation Differential influences of the aryl hydrocarbon receptor on Th17 mediated responses in vitro and in vivo.Cross-talk between aryl hydrocarbon receptor and the inflammatory response: a role for nuclear factor-κB.The aryl hydrocarbon receptor and food allergy.Aryl hydrocarbon receptor signaling modifies Toll-like receptor-regulated responses in human dendritic cells.Interrelated Roles for the Aryl Hydrocarbon Receptor and Hypoxia Inducible Factor-1α in the Immune Response to Infection.Biology and function of the aryl hydrocarbon receptor: report of an international and interdisciplinary conference.Endogenous ligands of the aryl hydrocarbon receptor regulate lung dendritic cell function.Immunology: Mammalian watchdog targets bacteria.

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P2860

Aryl hydrocarbon receptor activation reduces dendritic cell function during influenza virus infection.

http://www.wikidata.org/entity/Q34000825

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Aryl hydrocarbon receptor acti ...... ing influenza virus infection.

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Aryl hydrocarbon receptor acti ...... ing influenza virus infection.

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Aryl hydrocarbon receptor acti ...... ing influenza virus infection.

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Aryl hydrocarbon receptor acti ...... ing influenza virus infection.

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Aryl hydrocarbon receptor acti ...... ing influenza virus infection.

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Toxicological Sciences

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Aryl hydrocarbon receptor acti ...... ing influenza virus infection.

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P2093

Amanda J Moore

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B Paige Lawrence

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Guang-Bi Jin

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Jennifer L Head

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Joshua J Neumiller

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P2860

The aryl hydrocarbon receptor: a perspective on potential roles in the immune system

Distinct migrating and nonmigrating dendritic cell populations are involved in MHC class I-restricted antigen presentation after lung infection with virus

Respiratory dendritic cell subsets differ in their capacity to support the induction of virus-specific cytotoxic CD8+ T cell responses.

2,3,7,8-Tetrachlorodibenzo-p-dioxin suppresses tumor necrosis factor-alpha and anti-CD40-induced activation of NF-kappaB/Rel in dendritic cells: p50 homodimer activation is not affected

Reduced antibody responses to vaccinations in children exposed to polychlorinated biphenyls

Accelerated migration of respiratory dendritic cells to the regional lymph nodes is limited to the early phase of pulmonary infection.

Frequency, specificity, and sites of expansion of CD8+ T cells during primary pulmonary influenza virus infection.

2,3,7,8-Tetrachlorodibenzo-p-dioxin affects the number and function of murine splenic dendritic cells and their expression of accessory molecules.

Primary peripheral T cells become susceptible to 2,3,7,8-tetrachlorodibenzo-p-dioxin-mediated apoptosis in vitro upon activation and in the presence of dendritic cells

Effects of TCDD on the fate of naive dendritic cells.

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514-522

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10.1093/TOXSCI/KFQ153

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2

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44626051

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20498003

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2905408

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