Temporal and regional alterations in NMDA receptor expression in Mecp2-null mice. - Test2 - elhamkhani - TriplyDB

Temporal and regional alterations in NMDA receptor expression in Mecp2-null mice.

Temporal and regional alterations in NMDA receptor expression in Mecp2-null mice.

http://www.wikidata.org/entity/Q34004048

about

Recent advances in understanding synaptic abnormalities in Rett syndrome Abnormalities of the DNA methylation mark and its machinery: an emerging cause of neurologic dysfunction Targeted pharmacological treatment of autism spectrum disorders: fragile X and Rett syndromes Rett Syndrome: Reaching for Clinical Trials Mechanisms of Functional Hypoconnectivity in the Medial Prefrontal Cortex of Mecp2 Null Mice.Preweaning sensorimotor deficits and adolescent hypersociability in Grin1 knockdown mice.Twenty-four hour quantitative-EEG and in-vivo glutamate biosensor detects activity and circadian rhythm dependent biomarkers of pathogenesis in Mecp2 null mice.Brain injury in canine models of cardiac surgery Rett syndrome: genes, synapses, circuits, and therapeutics Heightened Delta Power during Slow-Wave-Sleep in Patients with Rett Syndrome Associated with Poor Sleep Efficiency.Cell-Specific Regulation of N-Methyl-D-Aspartate Receptor Maturation by Mecp2 in Cortical Circuits.Enhancement of postsynaptic GABAA and extrasynaptic NMDA receptor-mediated responses in the barrel cortex of Mecp2-null mice.The first mecp2-null zebrafish model shows altered motor behaviors Neurobiologically-based treatments in Rett syndrome: opportunities and challenges.Temporal- and Location-Specific Alterations of the GABA Recycling System in Mecp2 KO Mouse Brains.Detecting autonomic response to pain in Rett syndrome.Randomized open-label trial of dextromethorphan in Rett syndrome.Dendrimer-mediated delivery of N-acetyl cysteine to microglia in a mouse model of Rett syndrome.NMDA Receptors in the Central Nervous System.MeCP2 Modulates Sex Differences in the Postsynaptic Development of the Valproate Animal Model of Autism.Rett Syndrome: Coming to Terms with Treatment

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Temporal and regional alterations in NMDA receptor expression in Mecp2-null mice.

http://www.wikidata.org/entity/Q34004048

description

2011 nî lūn-bûn

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2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Temporal and regional alterations in NMDA receptor expression in Mecp2-null mice.

@ast

Temporal and regional alterations in NMDA receptor expression in Mecp2-null mice.

@en

Temporal and regional alterations in NMDA receptor expression in Mecp2-null mice.

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type

label

Temporal and regional alterations in NMDA receptor expression in Mecp2-null mice.

@ast

Temporal and regional alterations in NMDA receptor expression in Mecp2-null mice.

@en

Temporal and regional alterations in NMDA receptor expression in Mecp2-null mice.

@nl

prefLabel

Temporal and regional alterations in NMDA receptor expression in Mecp2-null mice.

@ast

Temporal and regional alterations in NMDA receptor expression in Mecp2-null mice.

@en

Temporal and regional alterations in NMDA receptor expression in Mecp2-null mice.

@nl

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The Anatomical Record: Advances in Integrative Anatomy and Evolutionary Biology

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Temporal and regional alterations in NMDA receptor expression in Mecp2-null mice.

@en

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Charlotte Eyring

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Cliona O'driscoll

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Joseph Bressler

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Mary E Blue

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Michael V Johnston

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Sakkubai Naidu

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Walter E Kaufmann

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P2860

Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2

Reduced cortical activity due to a shift in the balance between excitation and inhibition in a mouse model of Rett syndrome

Rett syndrome: revised diagnostic criteria and nomenclature

Advances in the treatment of fragile X syndrome

MeCP2 expression and function during brain development: implications for Rett syndrome's pathogenesis and clinical evolution

A mouse Mecp2-null mutation causes neurological symptoms that mimic Rett syndrome

Learning and memory and synaptic plasticity are impaired in a mouse model of Rett syndrome

MeCP2 mutation causes distinguishable phases of acute and chronic defects in synaptogenesis and maintenance, respectively

Hippocampal synaptic plasticity is impaired in the Mecp2-null mouse model of Rett syndrome

The transcriptional repressor Mecp2 regulates terminal neuronal differentiation

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10.1002/AR.21380

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10

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294

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