Upregulation of inducible nitric oxide synthase contributes to attenuated cutaneous vasodilation in essential hypertensive humans. - Test2 - elhamkhani - TriplyDB

Upregulation of inducible nitric oxide synthase contributes to attenuated cutaneous vasodilation in essential hypertensive humans.

Upregulation of inducible nitric oxide synthase contributes to attenuated cutaneous vasodilation in essential hypertensive humans.

http://www.wikidata.org/entity/Q34022778

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Altered Nitric Oxide System in Cardiovascular and Renal Diseases Age-related changes in brain support cells: Implications for stroke severity Mechanisms and time course of menthol-induced cutaneous vasodilation Tempol improves cutaneous thermal hyperemia through increasing nitric oxide bioavailability in young smokers.Altered skin flowmotion in hypertensive humans New approach to measure cutaneous microvascular function: an improved test of NO-mediated vasodilation by thermal hyperemia.Antihypertensive effects of inducible nitric oxide synthase inhibition in experimental pre-eclampsia.KCa channels and epoxyeicosatrienoic acids: major contributors to thermal hyperaemia in human skin.Sex- and limb-specific differences in the nitric oxide-dependent cutaneous vasodilation in response to local heating.Cutaneous microvascular dysfunction correlates with serum LDL and sLOX-1 receptor concentrations Impaired acetylcholine-induced cutaneous vasodilation in young smokers: roles of nitric oxide and prostanoids.Rho-Kinase activity and cutaneous vasoconstriction is upregulated in essential hypertensive humans.Endothelial nitric oxide synthase mediates the nitric oxide component of reflex cutaneous vasodilatation during dynamic exercise in humans.Augmented reflex cutaneous vasodilatation following short-term dietary nitrate supplementation in humans.Differential involvement of various sources of reactive oxygen species in thyroxin-induced hemodynamic changes and contractile dysfunction of the heart and diaphragm muscles.Endothelial nitric oxide synthase mediates cutaneous vasodilation during local heating and is attenuated in middle-aged human skin Differential modulation of nitric oxide synthases in aging: therapeutic opportunities.Passive heat therapy improves cutaneous microvascular function in sedentary humans via improved nitric oxide-dependent dilation.Endothelial-derived hyperpolarization contributes to acetylcholine-mediated vasodilation in human skin in a dose-dependent manner Functional Inducible Nitric Oxide Synthase Gene Variants Associate With Hypertension: A Case-Control Study in a Finnish Population-The TAMRISK Study Impaired Hydrogen Sulfide-Mediated Vasodilation Contributes to Microvascular Endothelial Dysfunction in Hypertensive Adults.iNOS-dependent sweating and eNOS-dependent cutaneous vasodilation are evident in younger adults, but are diminished in older adults exercising in the heat Current concepts of active vasodilation in human skin.Cutaneous vasodilator and vasoconstrictor mechanisms in temperature regulation.Nitric oxide synthase inhibitors: a review of patents from 2011 to the present.Effects of vehicle microdialysis solutions on cutaneous vascular responses to local heating.Increased Angiotensin II Sensitivity Contributes to Microvascular Dysfunction in Women Who Have Had Preeclampsia.Evidence for a functional vasodilatatory role for hydrogen sulphide in the human cutaneous microvasculature.Edward F. Adolph Distinguished Lecture: Skin-deep insights into vascular aging.Alterations in endothelin type B receptor contribute to microvascular dysfunction in women who have had preeclampsia.Endothelial function is impaired in the cutaneous microcirculation of adults with psoriasis through reductions in nitric oxide-dependent vasodilation.Menthol-Induced Cutaneous Vasodilation Is Preserved in Essential Hypertensive Men and Women.Blood pressure normalization via pharmacotherapy improves cutaneous microvascular function through NO-dependent and NO-independent mechanisms.Nicotinic receptor activation augments muscarinic receptor-mediated eccrine sweating but not cutaneous vasodilatation in young males.Acute lysyl oxidase inhibition alters microvascular function in normotensive, but not hypertensive, men and women.

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P2860

Upregulation of inducible nitric oxide synthase contributes to attenuated cutaneous vasodilation in essential hypertensive humans.

http://www.wikidata.org/entity/Q34022778

description

2011 nî lūn-bûn

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2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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Upregulation of inducible nitr ...... essential hypertensive humans.

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Upregulation of inducible nitr ...... essential hypertensive humans.

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Upregulation of inducible nitr ...... essential hypertensive humans.

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Upregulation of inducible nitr ...... essential hypertensive humans.

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Upregulation of inducible nitr ...... essential hypertensive humans.

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Upregulation of inducible nitr ...... essential hypertensive humans.

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Upregulation of inducible nitr ...... essential hypertensive humans.

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Upregulation of inducible nitr ...... essential hypertensive humans.

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Upregulation of inducible nitr ...... essential hypertensive humans.

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Upregulation of inducible nitr ...... essential hypertensive humans.

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Anna Stanhewicz

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Caroline J Smith

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Dan E Berkowitz

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Lacy A Holowatz

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Lakshmi Santhanam

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Rebecca S Bruning

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P2860

Neuronal nitric oxide synthase control mechanisms in the cutaneous vasculature of humans in vivo

Crystal structure of human arginase I at 1.29-A resolution and exploration of inhibition in the immune response.

Endothelial nitric oxide synthase in vascular disease: from marvel to menace

Inducible NO synthase dependent S-nitrosylation and activation of arginase1 contribute to age-related endothelial dysfunction

Acute ascorbate supplementation alone or combined with arginase inhibition augments reflex cutaneous vasodilation in aged human skin.

Noninvasive interrogation of microvasculature for signs of endothelial dysfunction in patients with chronic renal failure.

Mechanisms of acetylcholine-mediated vasodilatation in young and aged human skin.

L-Arginine supplementation or arginase inhibition augments reflex cutaneous vasodilatation in aged human skin.

The investigation of skin blood flowmotion: a new approach to study the microcirculatory impairment in vascular diseases?

Cold-induced cutaneous vasoconstriction is mediated by Rho kinase in vivo in human skin.

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21931069

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3209704

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