Angiotensin-converting enzyme N-terminal inactivation alleviates bleomycin-induced lung injury.
about
A modern understanding of the traditional and nontraditional biological functions of angiotensin-converting enzymeThe Effects of Angiotensin Converting Enzyme Inhibitors (ACE-I) on Human N-Acetyl-Seryl-Aspartyl-Lysyl-Proline (Ac-SDKP) Levels: A Systematic Review and Meta-AnalysisRediscovering ACE: novel insights into the many roles of the angiotensin-converting enzymeN-acetyl-seryl-aspartyl-lysyl-proline: a valuable endogenous anti-fibrotic peptide for combating kidney fibrosis in diabetesNovel mechanism of inhibition of human angiotensin-I-converting enzyme (ACE) by a highly specific phosphinic tripeptideFragment-based design for the development of N-domain-selective angiotensin-1-converting enzyme inhibitorsDifferent in vivo functions of the two catalytic domains of angiotensin-converting enzyme (ACE).Spironolactone attenuates bleomycin-induced pulmonary injury partially via modulating mononuclear phagocyte phenotype switching in circulating and alveolar compartments.A novel angiotensin I-converting enzyme mutation (S333W) impairs N-domain enzymatic cleavage of the anti-fibrotic peptide, AcSDKP.Increased angiotensin II-induced hypertension and inflammatory cytokines in mice lacking angiotensin-converting enzyme N domain activity.Nontraditional roles of angiotensin-converting enzyme.Structural basis of Ac-SDKP hydrolysis by Angiotensin-I converting enzyme.Acetylated α-Tubulin Regulated by N-Acetyl-Seryl-Aspartyl-Lysyl-Proline(Ac-SDKP) Exerts the Anti-fibrotic Effect in Rat Lung Fibrosis Induced by SilicaElevation of the antifibrotic peptide N-acetyl-seryl-aspartyl-lysyl-proline: a blood pressure-independent beneficial effect of angiotensin I-converting enzyme inhibitorsAntifibrotic peptide N-acetyl-Ser-Asp-Lys-Pro (Ac-SDKP): opportunities for angiotensin-converting enzyme inhibitor design.The biological significance of angiotensin-converting enzyme inhibition to combat kidney fibrosis.Preventive and therapeutic effects of thymosin β4 N-terminal fragment Ac-SDKP in the bleomycin model of pulmonary fibrosis.Effect of Antifibrotic MicroRNAs Crosstalk on the Action of N-acetyl-seryl-aspartyl-lysyl-proline in Diabetes-related Kidney FibrosisN-acetyl-seryl-aspartyl-lysyl-proline inhibits diabetes-associated kidney fibrosis and endothelial-mesenchymal transition.Angiotensin-converting enzyme defines matrikine-regulated inflammation and fibrosis.
P2860
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P2860
Angiotensin-converting enzyme N-terminal inactivation alleviates bleomycin-induced lung injury.
description
2010 nî lūn-bûn
@nan
2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Angiotensin-converting enzyme ...... bleomycin-induced lung injury.
@ast
Angiotensin-converting enzyme ...... bleomycin-induced lung injury.
@en
Angiotensin-converting enzyme ...... bleomycin-induced lung injury.
@nl
type
label
Angiotensin-converting enzyme ...... bleomycin-induced lung injury.
@ast
Angiotensin-converting enzyme ...... bleomycin-induced lung injury.
@en
Angiotensin-converting enzyme ...... bleomycin-induced lung injury.
@nl
prefLabel
Angiotensin-converting enzyme ...... bleomycin-induced lung injury.
@ast
Angiotensin-converting enzyme ...... bleomycin-induced lung injury.
@en
Angiotensin-converting enzyme ...... bleomycin-induced lung injury.
@nl
P2093
P2860
P1476
Angiotensin-converting enzyme ...... bleomycin-induced lung injury.
@en
P2093
Anthony Gal
Frank S Ong
Hong D Xiao
Jesse Roman
Jianguo Xu
Sebastien Fuchs
P2860
P304
P356
10.2353/AJPATH.2010.081127
P407
P577
2010-07-22T00:00:00Z