Morphological and functional abnormalities in mitochondria associated with synaptic degeneration in prion disease. - Test2 - elhamkhani - TriplyDB

Morphological and functional abnormalities in mitochondria associated with synaptic degeneration in prion disease.

Morphological and functional abnormalities in mitochondria associated with synaptic degeneration in prion disease.

http://www.wikidata.org/entity/Q34086982

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Unfolded Protein Response and Macroautophagy in Alzheimer's, Parkinson's and Prion Diseases Redox control of protein degradation Amyloid oligomer neurotoxicity, calcium dysregulation, and lipid rafts The type of Aβ-related neuronal degeneration differs between amyloid precursor protein (APP23) and amyloid β-peptide (APP48) transgenic mice Quantitative quadruple-label immunofluorescence of mitochondrial and cytoplasmic proteins in single neurons from human midbrain tissue.Molecular structure of amyloid fibrils controls the relationship between fibrillar size and toxicity.Proteomics analysis of amyloid and nonamyloid prion disease phenotypes reveals both common and divergent mechanisms of neuropathogenesis Sod1 deficiency reduces incubation time in mouse models of prion disease Brain region specific pre-synaptic and post-synaptic degeneration are early components of neuropathology in prion disease.Analyses of the mitochondrial mutations in the Chinese patients with sporadic Creutzfeldt-Jakob disease The neutral sphingomyelinase pathway regulates packaging of the prion protein into exosomes.Striatal pathology underlies prion infection-mediated hyperactivity in mice.Mitochondrial regulation of epigenetics and its role in human diseases.Antioxidant peroxiredoxin 6 protein rescues toxicity due to oxidative stress and cellular hypoxia in vitro, and attenuates prion-related pathology in vivo Exposure of rats to environmental tobacco smoke during cerebellar development alters behavior and perturbs mitochondrial energetics Differential molecular chaperone response associated with various mouse adapted scrapie strains.Cytosolic caspases mediate mislocalised SOD2 depletion in an in vitro model of chronic prion infection.RML prions act through Mahogunin and Attractin-independent pathways.Mitochondrial Abnormalities and Synaptic Loss Underlie Memory Deficits Seen in Mouse Models of Obesity and Alzheimer's Disease Cellular prion protein is present in mitochondria of healthy mice.The role of microglia in synaptic stripping and synaptic degeneration: a revised perspective.Metal attenuating therapies in neurodegenerative disease.Mechanisms underlying synaptic vulnerability and degeneration in neurodegenerative disease.Role of autophagy in prion protein-induced neurodegenerative diseases.Multispectral imaging: a review of its technical aspects and applications in anatomic pathology.Proteomics applications in prion biology and structure.Melatonin-mediated β-catenin activation protects neuron cells against prion protein-induced neurotoxicity.Gingerol-induced hypoxia-inducible factor 1 alpha inhibits human prion peptide-mediated neurotoxicity.Melatonin-induced autophagy protects against human prion protein-mediated neurotoxicity.Losing sleep over mitochondria: a new player in the pathophysiology of fatal familial insomnia.Melatonin regulates the autophagic flux via activation of alpha-7 nicotinic acetylcholine receptors.Aberrant Alterations of Mitochondrial Factors Drp1 and Opa1 in the Brains of Scrapie Experiment Rodents.Statins are ineffective at reducing neuroinflammation or prolonging survival in scrapie-infected mice.Mitochondrial Respiration Is Impaired during Late-Stage Hamster Prion Infection.Cytosolically expressed PrP GPI-signal peptide interacts with mitochondria.A role for microglia in synaptic plasticity?The ultrastructural characteristics of porcine hepatocytes donated after cardiac death and preserved with warm machine perfusion preservation ALS--dying forward, backward or outward?Persistence of intact retinal ganglion cell terminals after axonal transport loss in the DBA/2J mouse model of glaucoma.p62-Keap1-NRF2-ARE Pathway: A Contentious Player for Selective Targeting of Autophagy, Oxidative Stress and Mitochondrial Dysfunction in Prion Diseases

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Morphological and functional abnormalities in mitochondria associated with synaptic degeneration in prion disease.

http://www.wikidata.org/entity/Q34086982

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2010年の論文

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2010年論文

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2010年論文

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2010年论文

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Morphological and functional a ...... degeneration in prion disease.

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Morphological and functional a ...... degeneration in prion disease.

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Morphological and functional a ...... degeneration in prion disease.

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AJPATH.2010.091037

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The American Journal of Pathology

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Morphological and functional a ...... degeneration in prion disease

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Ayodeji Asuni

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Carianne Pudney

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Don Joseph Mahad

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Victor Hugh Perry

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P2860

Mitochondrial defects in acute multiple sclerosis lesions

Mitochondrial DNA mutations in human disease

Synthesis of N-acetyl-L-aspartate by rat brain mitochondria and its involvement in mitochondrial/cytosolic carbon transport

Mitochondrial respiratory-chain diseases

The importance of dendritic mitochondria in the morphogenesis and plasticity of spines and synapses

Molecular pathways of neurodegeneration in Parkinson's disease

Abnormal mitochondrial morphology in sporadic Parkinson's and Alzheimer's disease cybrid cell lines.

Morphology and ATP-ase of isolated mitochondria.

MRI and MRS alterations in the preclinical phase of murine prion disease: association with neuropathological and behavioural changes.

Characterization of a histone-like protein extracted from yeast mitochondria.

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Graham R Campbell

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Prion protein family

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