A new quantitative assay for cytochrome c release in apoptotic cells.
about
BAK multimerization for apoptosis, but not bid binding, is inhibited by negatively charged residue in the BAK hydrophobic grooveSurvivin monomer plays an essential role in apoptosis regulationDetecting apoptosis in Drosophila tissues and cellsInhibition of apoptosis prevents West Nile virus induced cell deathGranzyme M mediates a novel form of perforin-dependent cell deathThe mitochondrial protein Bak is pivotal for gliotoxin-induced apoptosis and a critical host factor of Aspergillus fumigatus virulence in miceBetulin is a potent anti-tumor agent that is enhanced by cholesterol.Growth factor erv1-like modulates Drp1 to preserve mitochondrial dynamics and function in mouse embryonic stem cellsApplication of flow cytometry to determine differential redistribution of cytochrome c and Smac/DIABLO from mitochondria during cell death signaling.Apoptosis and non-inflammatory phagocytosis can be induced by mitochondrial damage without caspases.Apoptosis induction by Bid requires unconventional ubiquitination and degradation of its N-terminal fragment.The C-terminal region of Bfl-1 sensitizes non-small cell lung cancer to gemcitabine-induced apoptosis by suppressing NF-κB activity and down-regulating Bfl-1.BMX Negatively Regulates BAK Function, Thereby Increasing Apoptotic Resistance to Chemotherapeutic DrugsDifferent apoptotic pathways activated by oxaliplatin in primary astrocytes vs. colo-rectal cancer cellsp47phox-deficient immune microenvironment signals dysregulate naive T-cell apoptosis.IL-17A Promotes Intracellular Growth of Mycobacterium by Inhibiting Apoptosis of Infected Macrophages.Apaf-1 and caspase-9 accelerate apoptosis, but do not determine whether factor-deprived or drug-treated cells dieSmall-molecule XIAP antagonist restores caspase-9 mediated apoptosis in XIAP-positive diffuse large B-cell lymphoma cellsVibrio cholerae Porin OmpU Induces Caspase-independent Programmed Cell Death upon Translocation to the Host Cell MitochondriaCaspase-cleaved arrestin-2 and BID cooperatively facilitate cytochrome C release and cell death.CD160 signaling mediates PI3K-dependent survival and growth signals in chronic lymphocytic leukemia.A functional genomics screen identifies PCAF and ADA3 as regulators of human granzyme B-mediated apoptosis and Bid cleavage.Inhibiting the system xC-/glutathione axis selectively targets cancers with mutant-p53 accumulationp73 engages A2B receptor signalling to prime cancer cells to chemotherapy-induced death.Engaging Ly-6A/Sca-1 triggers lipid raft-dependent and -independent responses in CD4(+) T-cell lines.Betulinic acid induces a novel cell death pathway that depends on cardiolipin modification.Mouse cytotoxic T cell-derived granzyme B activates the mitochondrial cell death pathway in a Bim-dependent fashion.Resistance to UV-induced apoptosis by β-HPV5 E6 involves targeting of activated BAK for proteolysis by recruitment of the HERC1 ubiquitin ligase.PARP inhibitor olaparib increases the oncolytic activity of dl922-947 in in vitro and in vivo model of anaplastic thyroid carcinomaMode of cell death induction by pharmacological vacuolar H+-ATPase (V-ATPase) inhibition.Extended survival of SH-SY5Y cells following overexpression of Lys67Glu neuroglobin is associated with stabilization of ΔψM.Antitumour and antiangiogenic effects of Aplidin in the 5TMM syngeneic models of multiple myelomaReovirus infection or ectopic expression of outer capsid protein micro1 induces apoptosis independently of the cellular proapoptotic proteins Bax and Bak.Tyrosine dephosphorylation is required for Bak activation in apoptosis.Granzyme B-induced cell death exerted by ex vivo CTL: discriminating requirements for cell death and some of its signs.Caspase inhibition blocks cell death and results in cell cycle arrest in cytokine-deprived hematopoietic cells.Loss of caspase-9 reveals its essential role for caspase-2 activation and mitochondrial membrane depolarization.Loss of caspase-9 provides genetic evidence for the type I/II concept of CD95-mediated apoptosis.Chk1 activity is required for BAK multimerization in association with PUMA during mitochondrial apoptosis.Cyclin-dependent kinase inhibitors uncouple cell cycle progression from mitochondrial apoptotic functions in DNA-damaged cancer cells.
P2860
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P2860
A new quantitative assay for cytochrome c release in apoptotic cells.
description
2003 nî lūn-bûn
@nan
2003 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
A new quantitative assay for cytochrome c release in apoptotic cells.
@ast
A new quantitative assay for cytochrome c release in apoptotic cells.
@en
A new quantitative assay for cytochrome c release in apoptotic cells.
@nl
type
label
A new quantitative assay for cytochrome c release in apoptotic cells.
@ast
A new quantitative assay for cytochrome c release in apoptotic cells.
@en
A new quantitative assay for cytochrome c release in apoptotic cells.
@nl
prefLabel
A new quantitative assay for cytochrome c release in apoptotic cells.
@ast
A new quantitative assay for cytochrome c release in apoptotic cells.
@en
A new quantitative assay for cytochrome c release in apoptotic cells.
@nl
P2860
P356
P1476
A new quantitative assay for cytochrome c release in apoptotic cells
@en
P2093
Waterhouse NJ
P2860
P2888
P304
P356
10.1038/SJ.CDD.4401263
P577
2003-07-01T00:00:00Z