Mechanisms of opioid-induced pain and antinociceptive tolerance: descending facilitation and spinal dynorphin.
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Structure-activity relationships of bifunctional peptides based on overlapping pharmacophores at opioid and cholecystokinin receptorsNeuropathic pain activates the endogenous kappa opioid system in mouse spinal cord and induces opioid receptor toleranceIncreased glutamate synaptic transmission in the nucleus raphe magnus neurons from morphine-tolerant ratsPLGA-Curcumin Attenuates Opioid-Induced Hyperalgesia and Inhibits Spinal CaMKIIαSpinal NK-1 receptor expressing neurons mediate opioid-induced hyperalgesia and antinociceptive tolerance via activation of descending pathways.Sex differences in the anatomical and functional organization of the periaqueductal gray-rostral ventromedial medullary pathway in the rat: a potential circuit mediating the sexually dimorphic actions of morphine.Oxycodone plus ultra-low-dose naltrexone attenuates neuropathic pain and associated mu-opioid receptor-Gs coupling.Gabapentin improves cold-pressor pain responses in methadone-maintained patients.Spinal mediators that may contribute selectively to antinociceptive tolerance but not other effects of morphine as revealed by deletion of GluR5Lack of effect of chronic dextromethorphan on experimental pain tolerance in methadone-maintained patients.Spinal or systemic TY005, a peptidic opioid agonist/neurokinin 1 antagonist, attenuates pain with reduced tolerance.Role of NK-1 neurotransmission in opioid-induced hyperalgesiaOpioid induced hyperalgesia in anesthetic settings.Pathobiology of dynorphins in trauma and disease.Adeno-associated viral transfer of opioid receptor gene to primary sensory neurons: a strategy to increase opioid antinociception.G-protein receptor kinase 3 (GRK3) influences opioid analgesic tolerance but not opioid withdrawal.Chronic morphine increases Fos-positive neurons after concurrent cornea and tail stimulationActivation of descending pain-facilitatory pathways from the rostral ventromedial medulla by cholecystokinin elicits release of prostaglandin-E₂ in the spinal cord.Tolerance, opioid-induced allodynia and withdrawal associated allodynia in infant and young rats.Pharmacological strategies in relieving neuropathic pain.Repeated morphine treatment-mediated hyperalgesia, allodynia and spinal glial activation are blocked by co-administration of a selective cannabinoid receptor type-2 agonistOpioid-independent mechanisms supporting offset analgesia and temporal sharpening of nociceptive informationOpioids and central sensitisation: II. Induction and reversal of hyperalgesia.Pronociceptive and Antinociceptive Effects of Buprenorphine in the Spinal Cord Dorsal Horn Cover a Dose Range of Four Orders of Magnitude.TRPV1 receptor in expression of opioid-induced hyperalgesiaPregabalin suppresses spinal neuronal hyperexcitability and visceral hypersensitivity in the absence of peripheral pathophysiologyStructure-activity relationships of bifunctional cyclic disulfide peptides based on overlapping pharmacophores at opioid and cholecystokinin receptors.Intravenous ibandronate rapidly reduces pain, neurochemical indices of central sensitization, tumor burden, and skeletal destruction in a mouse model of bone cancer.Altered quantitative sensory testing outcome in subjects with opioid therapy.Building a better analgesic: multifunctional compounds that address injury-induced pathology to enhance analgesic efficacy while eliminating unwanted side effects.Peroxynitrite and opiate antinociceptive tolerance: a painful reality.Chronic morphine exposure increases the proportion of on-cells in the rostral ventromedial medulla in rats.Preclinical and early clinical investigations related to monoaminergic pain modulation.Withdrawal-associated injury site pain (WISP): a descriptive case series of an opioid cessation phenomenon.Treatment of central sensitization in patients with 'unexplained' chronic pain: what options do we have?Opioid administration following spinal cord injury: implications for pain and locomotor recovery.Transgene-mediated expression of tumor necrosis factor soluble receptor attenuates morphine tolerance in rats.Targeting Opioid-Induced Hyperalgesia in Clinical Treatment: Neurobiological Considerations.Opioid-induced hyperalgesia and tolerance: understanding opioid side effects.Intrathecal lamotrigine attenuates antinociceptive morphine tolerance and suppresses spinal glial cell activation in morphine-tolerant rats.
P2860
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P2860
Mechanisms of opioid-induced pain and antinociceptive tolerance: descending facilitation and spinal dynorphin.
description
2001 nî lūn-bûn
@nan
2001 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Mechanisms of opioid-induced p ...... litation and spinal dynorphin.
@ast
Mechanisms of opioid-induced p ...... litation and spinal dynorphin.
@en
Mechanisms of opioid-induced p ...... litation and spinal dynorphin.
@nl
type
label
Mechanisms of opioid-induced p ...... litation and spinal dynorphin.
@ast
Mechanisms of opioid-induced p ...... litation and spinal dynorphin.
@en
Mechanisms of opioid-induced p ...... litation and spinal dynorphin.
@nl
prefLabel
Mechanisms of opioid-induced p ...... litation and spinal dynorphin.
@ast
Mechanisms of opioid-induced p ...... litation and spinal dynorphin.
@en
Mechanisms of opioid-induced p ...... litation and spinal dynorphin.
@nl
P2093
P1433
P1476
Mechanisms of opioid-induced p ...... litation and spinal dynorphin.
@en
P2093
P356
10.1016/S0304-3959(01)00311-6
P407
P577
2001-05-01T00:00:00Z