NK cells are not required for spontaneous autoimmune diabetes in NOD mice. - Test2 - elhamkhani - TriplyDB

NK cells are not required for spontaneous autoimmune diabetes in NOD mice.

NK cells are not required for spontaneous autoimmune diabetes in NOD mice.

http://www.wikidata.org/entity/Q34257031

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Type 1 diabetes: translating mechanistic observations into effective clinical outcomes Tissue-Resident NK Cells Mediate Ischemic Kidney Injury and Are Not Depleted by Anti-Asialo-GM1 Antibody.Comparative genetics: synergizing human and NOD mouse studies for identifying genetic causation of type 1 diabetes Limitations of IL-2 and rapamycin in immunotherapy of type 1 diabetes Flow cytometric gating for spleen monocyte and DC subsets: differences in autoimmune NOD mice and with acute inflammation Type 1 diabetes therapy beyond T cell targeting: monocytes, B cells, and innate lymphocytes The Expanding Role of Natural Killer Cells in Type 1 Diabetes and Immunotherapy.Dietary gluten increases natural killer cell cytotoxicity and cytokine secretion.Depletion of IL-2 receptor β-positive cells protects from diabetes in non-obese diabetic mice.The Four-Way Stop Sign: Viruses, 12-Lipoxygenase, Islets, and Natural Killer Cells in Type 1 Diabetes Progression The combination of anti-NKG2D and CTLA-4 Ig therapy prolongs islet allograft survival in a murine model.

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NK cells are not required for spontaneous autoimmune diabetes in NOD mice.

http://www.wikidata.org/entity/Q34257031

description

2012 nî lūn-bûn

@nan

2012 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի ապրիլին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

NK cells are not required for spontaneous autoimmune diabetes in NOD mice.

@ast

NK cells are not required for spontaneous autoimmune diabetes in NOD mice.

@en

NK cells are not required for spontaneous autoimmune diabetes in NOD mice.

@nl

type

label

NK cells are not required for spontaneous autoimmune diabetes in NOD mice.

@ast

NK cells are not required for spontaneous autoimmune diabetes in NOD mice.

@en

NK cells are not required for spontaneous autoimmune diabetes in NOD mice.

@nl

prefLabel

NK cells are not required for spontaneous autoimmune diabetes in NOD mice.

@ast

NK cells are not required for spontaneous autoimmune diabetes in NOD mice.

@en

NK cells are not required for spontaneous autoimmune diabetes in NOD mice.

@nl

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NK cells are not required for spontaneous autoimmune diabetes in NOD mice.

@en

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Craig T Meagher

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Jeffrey A Bluestone

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Joshua N Beilke

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Karoline Hosiawa

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Lewis L Lanier

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Marine Champsaur

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P2860

Innate immunity and intestinal microbiota in the development of Type 1 diabetes

NK cell recognition

Analysis of the cellular mechanism underlying inhibition of EAE after treatment with anti-NKG2A F(ab')2

The role of CD4 and CD8 T cells in the development of autoimmune diabetes.

A defective Il15 allele underlies the deficiency in natural killer cell activity in nonobese diabetic mice.

Activation of CD1d-restricted T cells protects NOD mice from developing diabetes by regulating dendritic cell subsets.

Natural killer cells trigger osteoclastogenesis and bone destruction in arthritis.

Central nervous system (CNS)-resident natural killer cells suppress Th17 responses and CNS autoimmune pathology.

Natural killer cells distinguish innocuous and destructive forms of pancreatic islet autoimmunity.

How punctual ablation of regulatory T cells unleashes an autoimmune lesion within the pancreatic islets

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