Crosstalk between mitochondrial and sarcoplasmic reticulum Ca2+ cycling modulates cardiac pacemaker cell automaticity.
about
Modern concepts concerning the origin of the heartbeat.Integrating beat rate variability: from single cells to heartsThe mitochondrial Na+-Ca2+ exchanger, NCLX, regulates automaticity of HL-1 cardiomyocytes.Cardiac responses to β-adrenoceptor stimulation is partly dependent on mitochondrial calcium uniporter activityCa²+/calmodulin-dependent protein kinase II (CaMKII) activity and sinoatrial nodal pacemaker cell energetics.From beat rate variability in induced pluripotent stem cell-derived pacemaker cells to heart rate variability in human subjects.Complete atrial-specific knockout of sodium-calcium exchange eliminates sinoatrial node pacemaker activity.Modulation of intracellular calcium waves and triggered activities by mitochondrial ca flux in mouse cardiomyocytes.Spatiotemporal stability of neonatal rat cardiomyocyte monolayers spontaneous activity is dependent on the culture substrate.Modern perspectives on numerical modeling of cardiac pacemaker cellMechano-sensitivity of cardiac pacemaker function: pathophysiological relevance, experimental implications, and conceptual integration with other mechanisms of rhythmicity.Regionally diverse mitochondrial calcium signaling regulates spontaneous pacing in developing cardiomyocytes.Mechanisms that match ATP supply to demand in cardiac pacemaker cells during high ATP demand.New evidence for coupled clock regulation of the normal automaticity of sinoatrial nodal pacemaker cells: bradycardic effects of ivabradine are linked to suppression of intracellular Ca²⁺ cycling.Electrochemical Na+ and Ca2+ gradients drive coupled-clock regulation of automaticity of isolated rabbit sinoatrial nodal pacemaker cells.Numerical Modeling Calcium and CaMKII Effects in the SA Node.Cardiac physiology and clinical efficacy of dietary fish oil clarified through cellular mechanisms of omega-3 polyunsaturated fatty acids.The destiny of Ca(2+) released by mitochondria.9-Phenanthrol and flufenamic acid inhibit calcium oscillations in HL-1 mouse cardiomyocytes.Involvement of mitochondrial permeability transition pore (mPTP) in cardiac arrhythmias: Evidence from cyclophilin D knockout mice.
P2860
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P2860
Crosstalk between mitochondrial and sarcoplasmic reticulum Ca2+ cycling modulates cardiac pacemaker cell automaticity.
description
2012 nî lūn-bûn
@nan
2012 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Crosstalk between mitochondria ...... c pacemaker cell automaticity.
@ast
Crosstalk between mitochondria ...... c pacemaker cell automaticity.
@en
Crosstalk between mitochondria ...... c pacemaker cell automaticity.
@nl
type
label
Crosstalk between mitochondria ...... c pacemaker cell automaticity.
@ast
Crosstalk between mitochondria ...... c pacemaker cell automaticity.
@en
Crosstalk between mitochondria ...... c pacemaker cell automaticity.
@nl
prefLabel
Crosstalk between mitochondria ...... c pacemaker cell automaticity.
@ast
Crosstalk between mitochondria ...... c pacemaker cell automaticity.
@en
Crosstalk between mitochondria ...... c pacemaker cell automaticity.
@nl
P2093
P2860
P50
P1433
P1476
Crosstalk between mitochondria ...... c pacemaker cell automaticity.
@en
P2093
Bruce D Ziman
Dongmei Yang
Harold A Spurgeon
P2860
P304
P356
10.1371/JOURNAL.PONE.0037582
P407
P577
2012-05-29T00:00:00Z