Collagens: molecular biology, diseases, and potentials for therapy.
about
Molecular and structural discrimination of proline racemase and hydroxyproline-2-epimerase from nosocomial and bacterial pathogensSplicing mutations of 54-bp exons in the COL11A1 gene cause Marshall syndrome, but other mutations cause overlapping Marshall/Stickler phenotypesComplete exon-intron organization of the human gene for the alpha1 chain of type XV collagen (COL15A1) and comparison with the homologous COL18A1 genePCOLCE2 encodes a functional procollagen C-proteinase enhancer (PCPE2) that is a collagen-binding protein differing in distribution of expression and post-translational modification from the previously described PCPE1Cloning of the human prolyl 4-hydroxylase alpha subunit isoform alpha(II) and characterization of the type II enzyme tetramer. The alpha(I) and alpha(II) subunits do not form a mixed alpha(I)alpha(II)beta2 tetramerCrystal structure and mapping by site-directed mutagenesis of the collagen-binding epitope of an activated form of BM-40/SPARC/osteonectinHuman endplate acetylcholinesterase deficiency caused by mutations in the collagen-like tail subunit (ColQ) of the asymmetric enzymeIdentification and characterization of the endocytic transmembrane glycoprotein Endo180 as a novel collagen receptorCloning and characterization of a novel human lysyl hydroxylase isoform highly expressed in pancreas and muscleCollagen XVI harbors an integrin alpha1 beta1 recognition site in its C-terminal domainsDevelopmental regulation of mRNA species for types II, IX and XI collagens during mouse embryogenesisERp60 does not substitute for protein disulphide isomerase as the beta-subunit of prolyl 4-hydroxylaseVariation in the vitreous phenotype of Stickler syndrome can be caused by different amino acid substitutions in the X position of the type II collagen Gly-X-Y triple helixCongenital end-plate acetylcholinesterase deficiency caused by a nonsense mutation and an A-->G splice-donor-site mutation at position +3 of the collagenlike-tail-subunit gene (COLQ): how does G at position +3 result in aberrant splicing?The C-proteinase that processes procollagens to fibrillar collagens is identical to the protein previously identified as bone morphogenic protein-1Physiological levels of tumstatin, a fragment of collagen IV alpha3 chain, are generated by MMP-9 proteolysis and suppress angiogenesis via alphaV beta3 integrinHuman prolyl-4-hydroxylase alpha(I) transcription is mediated by upstream stimulatory factorsCandidate cell and matrix interaction domains on the collagen fibril, the predominant protein of vertebratesLocation of type XV collagen in human tissues and its accumulation in the interstitial matrix of the fibrotic kidneyClinical and Molecular genetics of Stickler syndromeEmbryonic lethality of molecular chaperone hsp47 knockout mice is associated with defects in collagen biosynthesisInflammation, fibrosis, and modulation of the process by mesenchymal stem/stromal cellsOsteogenesis imperfecta due to mutations in non-collagenous genes: lessons in the biology of bone formationPost-translational hydroxylation by 2OG/Fe(II)-dependent oxygenases as a novel regulatory mechanism in bacteriaThe adventitia: essential regulator of vascular wall structure and functionDevelopments in purification methods for obtaining and evaluation of collagen derived endogenous angioinhibitorsDevelopment and characterization of a eukaryotic expression system for human type II procollagenBowstring Stretching and Quantitative Imaging of Single Collagen Fibrils via Atomic Force MicroscopyCrystal structure of NC1 domains. Structural basis for type IV collagen assembly in basement membranesLysyl hydroxylase 3 is a multifunctional protein possessing collagen glucosyltransferase activity.Transforming growth factor-beta induces secretion of activated ADAMTS-2. A procollagen III N-proteinase.Aqueous outflow - A continuum from trabecular meshwork to episcleral veinsType XIII collagen is identified as a plasma membrane proteinProduction of gene-targeted sheep by nuclear transfer from cultured somatic cellsWidely distributed mutations in the COL2A1 gene produce achondrogenesis type II/hypochondrogenesisDiscoidin domain receptor 1 is activated independently of beta(1) integrinThe transcription factor CCAAT-binding factor CBF/NF-Y regulates the proximal promoter activity in the human alpha 1(XI) collagen gene (COL11A1)Fertilin beta and other ADAMs as integrin ligands: insights into cell adhesion and fertilizationBiosynthetic processing of the pro-alpha 1(V)2pro-alpha 2(V) collagen heterotrimer by bone morphogenetic protein-1 and furin-like proprotein convertasesType XXVI collagen, a new member of the collagen family, is specifically expressed in the testis and ovary
P2860
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P2860
Collagens: molecular biology, diseases, and potentials for therapy.
description
1995 nî lūn-bûn
@nan
1995 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի հունվարին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Collagens: molecular biology, diseases, and potentials for therapy.
@ast
Collagens: molecular biology, diseases, and potentials for therapy.
@en
Collagens: molecular biology, diseases, and potentials for therapy.
@nl
type
label
Collagens: molecular biology, diseases, and potentials for therapy.
@ast
Collagens: molecular biology, diseases, and potentials for therapy.
@en
Collagens: molecular biology, diseases, and potentials for therapy.
@nl
prefLabel
Collagens: molecular biology, diseases, and potentials for therapy.
@ast
Collagens: molecular biology, diseases, and potentials for therapy.
@en
Collagens: molecular biology, diseases, and potentials for therapy.
@nl
P1476
Collagens: molecular biology, diseases, and potentials for therapy.
@en
P2093
Kivirikko KI
Prockop DJ
P304
P356
10.1146/ANNUREV.BI.64.070195.002155
P577
1995-01-01T00:00:00Z