Kruppel-like factor 6 (KLF6) is a tumor-suppressor gene frequently inactivated in colorectal cancer. - Test2 - elhamkhani - TriplyDB

Kruppel-like factor 6 (KLF6) is a tumor-suppressor gene frequently inactivated in colorectal cancer.

Kruppel-like factor 6 (KLF6) is a tumor-suppressor gene frequently inactivated in colorectal cancer.

http://www.wikidata.org/entity/Q34310233

about

Mammalian Krüppel-like factors in health and diseases Ah Receptor Pathway Intricacies; Signaling Through Diverse Protein Partners and DNA-Motifs Tumor suppressor and hepatocellular carcinoma Hepatic stellate cells: protean, multifunctional, and enigmatic cells of the liver p21 in cancer: intricate networks and multiple activities Prostate-specific Klf6 inactivation impairs anterior prostate branching morphogenesis through increased activation of the Shh pathway.Alterations in Barrett's-related adenocarcinomas: a proteomic approach.Genetic and genomic analysis modeling of germline c-MYC overexpression and cancer susceptibility KRAS and TP53 mutations in inflammatory bowel disease-associated colorectal cancer: a meta-analysis.Targeted reduction of KLF6-SV1 restores chemotherapy sensitivity in resistant lung adenocarcinoma.Tuba1a gene expression is regulated by KLF6/7 and is necessary for CNS development and regeneration in zebrafish.KLF6 depletion promotes NF-κB signaling in glioblastoma.KLF16 promotes proliferation in gastric cancer cells via regulating p21 and CDK4.Aberrant expression of krUppel-like factor 6 protein in colorectal cancers.Prognostic cancer gene signatures share common regulatory motifs TGF-beta regulates the expression of transcription factor KLF6 and its splice variants and promotes co-operative transactivation of common target genes through a Smad3-Sp1-KLF6 interaction KLF6 Gene and early melanoma development in a collagen I-rich extracellular environment.Epigenetic silencing of a proapoptotic cell adhesion molecule, the immunoglobulin superfamily member IGSF4, by promoter CpG methylation protects Hodgkin lymphoma cells from apoptosis Nucleo-cytoplasmic localization domains regulate Krüppel-like factor 6 (KLF6) protein stability and tumor suppressor function Involvement of Kruppel-like factor 6 (KLF6) mutation in the development of nonpolypoid colorectal carcinoma.Identification of miRNAs that specifically target tumor suppressive KLF6-FL rather than oncogenic KLF6-SV1 isoform.Functional role of the KLF6 tumour suppressor gene in gastric cancer.Energy restriction-mimetic agents induce apoptosis in prostate cancer cells in part through epigenetic activation of KLF6 tumor suppressor gene expression.Emerging roles of Kruppel-like factor 6 and Kruppel-like factor 6 splice variant 1 in ovarian cancer progression and treatment.KLF6-SV1 overexpression accelerates human and mouse prostate cancer progression and metastasis.Inactivation of the tumor suppressor Krüppel-like factor 6 (KLF6) by mutation or decreased expression in hepatocellular carcinomas.Evidence of linkage to chromosomes 10p15.3-p15.1, 14q24.3-q31.1 and 9q33.3-q34.3 in non-syndromic colorectal cancer families.Developmental regulation of yolk sac hematopoiesis by Kruppel-like factor 6 Transactivation of inducible nitric oxide synthase gene by Kruppel-like factor 6 regulates apoptosis during influenza A virus infection.Enhanced hepatocarcinogenesis in mouse models and human hepatocellular carcinoma by coordinate KLF6 depletion and increased messenger RNA splicing.Development and progression of colorectal neoplasia.Expression and significance of Kruppel-like factor 6 gene in osteosarcoma.KLF6-SV1 drives breast cancer metastasis and is associated with poor survival KLF8 promotes tumorigenesis, invasion and metastasis of colorectal cancer cells by transcriptional activation of FHL2.Downregulation of KLF6 is an early event in hepatocarcinogenesis, and stimulates proliferation while reducing differentiation.Transcriptional regulation of stellate cell activation.The Pathobiology of Krüppel-like Factors in Colorectal Cancer.Role of Kruppel-like factor 6 in transforming growth factor-beta1-induced epithelial-mesenchymal transition of proximal tubule cells Ras promotes growth by alternative splicing-mediated inactivation of the KLF6 tumor suppressor in hepatocellular carcinoma.Expression and Function of Kruppel Like-Factors (KLF) in Carcinogenesis.

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Kruppel-like factor 6 (KLF6) is a tumor-suppressor gene frequently inactivated in colorectal cancer.

http://www.wikidata.org/entity/Q34310233

description

2004 nî lūn-bûn

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2004 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2004 թվականի ապրիլին հրատարակված գիտական հոդված

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2004年の論文

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Kruppel-like factor 6 (KLF6) i ...... ctivated in colorectal cancer.

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J.GASTRO.2004.01.005

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Gastroenterology

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Kruppel-like factor 6 (KLF6) i ...... activated in colorectal cancer

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Amanda Katz

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Asif I Haq

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Eldad Hod

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Francis J Eng

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Helen L Reeves

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John A Martignetti

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Michael J P Arthur

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Olagunju Ogunbiyi

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Scott L Friedman

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Sharon Benzeno

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1090-1103

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scholarly article

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10.1053/J.GASTRO.2004.01.005

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4

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2004-04-01T00:00:00Z

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colorectal cancer