Epstein-Barr virus BGLF4 kinase retards cellular S-phase progression and induces chromosomal abnormality. - Test2 - elhamkhani - TriplyDB

Epstein-Barr virus BGLF4 kinase retards cellular S-phase progression and induces chromosomal abnormality.

Epstein-Barr virus BGLF4 kinase retards cellular S-phase progression and induces chromosomal abnormality.

http://www.wikidata.org/entity/Q34328083

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Modulation of DNA damage and repair pathways by human tumour viruses Epstein-Barr virus lytic reactivation regulation and its pathogenic role in carcinogenesis The synergistic effect of chemical carcinogens enhances Epstein-Barr virus reactivation and tumor progression of nasopharyngeal carcinoma cells Epstein-Barr virus BALF3 mediates genomic instability and progressive malignancy in nasopharyngeal carcinoma.BGLF4 kinase modulates the structure and transport preference of the nuclear pore complex to facilitate nuclear import of Epstein-Barr virus lytic proteins Identification of herpesvirus proteins that contribute to G1/S arrest.DNA Damage Signaling Is Induced in the Absence of Epstein-Barr Virus (EBV) Lytic DNA Replication and in Response to Expression of ZEBRA.EBV reactivation as a target of luteolin to repress NPC tumorigenesis.Inhibition of Epstein-Barr virus reactivation by the flavonoid apigenin.Short-term inhibition of TERT induces telomere length-independent cell cycle arrest and apoptotic response in EBV-immortalized and transformed B cells.Potential of protein kinase inhibitors for treating herpesvirus-associated disease.Modes of infection and oncogenesis by the Epstein-Barr virus.Regulation of Epstein-Barr virus reactivation from latency.The long-lasting love affair between the budding yeast Saccharomyces cerevisiae and the Epstein-Barr virus.Epstein-Barr virus BZLF1 protein impairs accumulation of host DNA damage proteins at damage sites in response to DNA damage.Epstein-Barr virus: more than 50 years old and still providing surprises.The role of oxidative stress in EBV lytic reactivation, radioresistance and the potential preventive and therapeutic implications.An MCM4 mutation detected in cancer cells affects MCM4/6/7 complex formation.The Immunomodulatory Capacity of an Epstein-Barr Virus Abortive Lytic Cycle: Potential Contribution to Viral Tumorigenesis.Therapies based on targeting Epstein-Barr virus lytic replication for EBV-associated malignancies.

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P2860

Epstein-Barr virus BGLF4 kinase retards cellular S-phase progression and induces chromosomal abnormality.

http://www.wikidata.org/entity/Q34328083

description

2012 nî lūn-bûn

@nan

2012 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի հունիսին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Epstein-Barr virus BGLF4 kinas ...... duces chromosomal abnormality.

@ast

Epstein-Barr virus BGLF4 kinas ...... duces chromosomal abnormality.

@en

Epstein-Barr virus BGLF4 kinas ...... duces chromosomal abnormality.

@nl

type

label

Epstein-Barr virus BGLF4 kinas ...... duces chromosomal abnormality.

@ast

Epstein-Barr virus BGLF4 kinas ...... duces chromosomal abnormality.

@en

Epstein-Barr virus BGLF4 kinas ...... duces chromosomal abnormality.

@nl

prefLabel

Epstein-Barr virus BGLF4 kinas ...... duces chromosomal abnormality.

@ast

Epstein-Barr virus BGLF4 kinas ...... duces chromosomal abnormality.

@en

Epstein-Barr virus BGLF4 kinas ...... duces chromosomal abnormality.

@nl

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JOURNAL.PONE.0039217

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Epstein-Barr virus BGLF4 kinas ...... duces chromosomal abnormality.

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Ching-Hwa Tsai

http://www.w3.org/2001/XMLSchema#string

Chung-Pei Lee

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Jen-Yang Chen

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Jiin-Tarng Wang

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Kenzo Takada

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Mei-Ru Chen

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Mei-Tzu Su

http://www.w3.org/2001/XMLSchema#string

Min-Jei Hsieh

http://www.w3.org/2001/XMLSchema#string

Su-Fang Lin

http://www.w3.org/2001/XMLSchema#string

Yu-Hsin Chang

http://www.w3.org/2001/XMLSchema#string

P2860

Phosphorylation of pRB at Ser612 by Chk1/2 leads to a complex between pRB and E2F-1 after DNA damage

Cell cycle, CDKs and cancer: a changing paradigm

The candidate tumor suppressor BTG3 is a transcriptional target of p53 that inhibits E2F1

The consensus motif for phosphorylation by cyclin D1-Cdk4 is different from that for phosphorylation by cyclin A/E-Cdk2

Genomic instability--an evolving hallmark of cancer

Multiple levels of cyclin specificity in cell-cycle control

Cyclin-dependent kinase-like function is shared by the beta- and gamma- subset of the conserved herpesvirus protein kinases

Epstein-Barr virus: 40 years on

Requirement for cell-to-cell contact in Epstein-Barr virus infection of nasopharyngeal carcinoma cells and keratinocytes.

Premature condensation induces breaks at the interface of early and late replicating chromosome bands bearing common fragile sites.

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6

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228331656

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Epstein–Barr virus

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3387188

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